Michael J. Zuscik

7.8k total citations
130 papers, 6.3k citations indexed

About

Michael J. Zuscik is a scholar working on Molecular Biology, Rheumatology and Surgery. According to data from OpenAlex, Michael J. Zuscik has authored 130 papers receiving a total of 6.3k indexed citations (citations by other indexed papers that have themselves been cited), including 75 papers in Molecular Biology, 55 papers in Rheumatology and 22 papers in Surgery. Recurrent topics in Michael J. Zuscik's work include Osteoarthritis Treatment and Mechanisms (50 papers), Bone Metabolism and Diseases (29 papers) and TGF-β signaling in diseases (26 papers). Michael J. Zuscik is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (50 papers), Bone Metabolism and Diseases (29 papers) and TGF-β signaling in diseases (26 papers). Michael J. Zuscik collaborates with scholars based in United States, China and Japan. Michael J. Zuscik's co-authors include Regis J. O’Keefe, Randy N. Rosier, Edward M. Schwarz, Hicham Drissi, Di Chen, J. Edward Puzas, Matthew J. Hilton, Robert A. Mooney, Qiuqian Wu and Hani A. Awad and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

Michael J. Zuscik

125 papers receiving 6.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael J. Zuscik United States 49 3.3k 2.5k 902 840 790 130 6.3k
Hicham Drissi United States 49 4.2k 1.3× 1.9k 0.8× 1.1k 1.2× 1.4k 1.6× 483 0.6× 177 7.0k
Daniel Lajeunesse Canada 41 2.4k 0.7× 3.8k 1.5× 890 1.0× 1.0k 1.2× 1.4k 1.8× 99 6.3k
Mohit Kapoor Canada 43 2.9k 0.9× 3.8k 1.5× 870 1.0× 720 0.9× 1.4k 1.8× 144 7.5k
Mei Wan United States 45 3.8k 1.1× 1.4k 0.5× 727 0.8× 1.4k 1.7× 420 0.5× 127 6.9k
Jang‐Soo Chun South Korea 45 2.6k 0.8× 2.3k 0.9× 461 0.5× 629 0.7× 862 1.1× 112 5.5k
Cory J. Xian Australia 50 3.1k 0.9× 882 0.4× 1.0k 1.1× 1.2k 1.4× 354 0.4× 218 7.6k
Martine Cohen‐Solal France 40 2.4k 0.7× 1.4k 0.6× 862 1.0× 1.5k 1.8× 533 0.7× 242 6.1k
Per Qvist Denmark 44 2.0k 0.6× 1.8k 0.7× 712 0.8× 1.8k 2.1× 700 0.9× 113 6.1k
Hee‐Jeong Im United States 35 1.6k 0.5× 2.1k 0.9× 916 1.0× 356 0.4× 971 1.2× 68 4.7k
Yūji Shimizu Japan 29 3.8k 1.2× 935 0.4× 617 0.7× 1.4k 1.7× 231 0.3× 238 7.0k

Countries citing papers authored by Michael J. Zuscik

Since Specialization
Citations

This map shows the geographic impact of Michael J. Zuscik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael J. Zuscik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael J. Zuscik more than expected).

Fields of papers citing papers by Michael J. Zuscik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael J. Zuscik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael J. Zuscik. The network helps show where Michael J. Zuscik may publish in the future.

Co-authorship network of co-authors of Michael J. Zuscik

This figure shows the co-authorship network connecting the top 25 collaborators of Michael J. Zuscik. A scholar is included among the top collaborators of Michael J. Zuscik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael J. Zuscik. Michael J. Zuscik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Villani, David A., Janne Prawitt, Ann Gill, et al.. (2025). Gut and Joint Microbiomes. Rheumatic Disease Clinics of North America. 51(2). 295–324. 2 indexed citations
2.
Fang, Liang, Xiangfeng Niu, Tiandao Li, et al.. (2025). NFIA regulates articular chondrocyte fatty acid metabolism and joint homeostasis. Science Translational Medicine. 17(809). eadm9488–eadm9488.
3.
Ackert‐Bicknell, Cheryl L., et al.. (2025). Synovial Dysregulation in Ankle Osteoarthritis: Molecular Insights and Pathogenetic Pathways. Journal of Orthopaedic Research®. 43(10). 1736–1747.
4.
Holers, V. Michael, Rachel M. Frank, Michael J. Zuscik, et al.. (2024). Decay-Accelerating Factor Differentially Associates With Complement-Mediated Damage in Synovium After Meniscus Tear as Compared to Anterior Cruciate Ligament Injury. Immune Network. 24(2). e17–e17. 2 indexed citations
5.
Thomas, Stacey M., Cheryl L. Ackert‐Bicknell, Michael J. Zuscik, & Karin A. Payne. (2022). Understanding the Transcriptomic Landscape to Drive New Innovations in Musculoskeletal Regenerative Medicine. Current Osteoporosis Reports. 20(2). 141–152. 2 indexed citations
6.
Gupta, Ranjan, et al.. (2019). Pharmacological Attenuation of Electrical Effects in a Model of Compression Neuropathy. Journal of Bone and Joint Surgery. 101(6). 523–530. 4 indexed citations
7.
Schott, Eric M., David A. Villani, Alex Grier, et al.. (2018). Oral hydrolyzed type 2 collagen protects against the OA of obesity and mitigates obese gut microbiome dysbiosis. Osteoarthritis and Cartilage. 26. S173–S174. 5 indexed citations
8.
Schott, Eric M., Jacquelyn Lillis, Christopher W Farnsworth, et al.. (2018). Early synovial B-cell infiltration as a candidate mechanism of pathogenesis in the OA of obesity. Osteoarthritis and Cartilage. 26. S14–S14. 1 indexed citations
9.
Schott, Eric M., Christopher W Farnsworth, Alex Grier, et al.. (2018). Targeting the gut microbiome to treat the osteoarthritis of obesity. JCI Insight. 3(8). 203 indexed citations
10.
Schott, Eric M., Christopher W Farnsworth, Alex Grier, et al.. (2017). Prebiotic manipulation of the gut microbiome with oligofructose confers protection against the osteoarthritis of obesity. Osteoarthritis and Cartilage. 25. S11–S12. 1 indexed citations
11.
Mirando, Anthony J., et al.. (2015). A dual role for notch signaling in joint cartilage maintenance and osteoarthritis. Osteoarthritis and Cartilage. 23. A64–A64. 3 indexed citations
12.
Beier, Eric E., Tzong‐Jen Sheu, Kiminori Yukata, et al.. (2014). Inhibition of beta‐catenin signaling by Pb leads to incomplete fracture healing. Journal of Orthopaedic Research®. 32(11). 1397–1405. 29 indexed citations
13.
Jacobson, Justin A., David G. Reynolds, Tulin Dadali, et al.. (2010). Teriparatide Therapy and Beta-Tricalcium Phosphate Enhance Scaffold Reconstruction of Mouse Femoral Defects. Tissue Engineering Part A. 17(3-4). 389–398. 28 indexed citations
14.
Dong, Yufeng, Anat Kohn, Tasuku Honjo, et al.. (2010). RBPjκ-dependent Notch signaling regulates mesenchymal progenitor cell proliferation and differentiation during skeletal development. Development. 137(9). 1461–1471. 149 indexed citations
15.
Zhu, Mei, Dezhi Tang, Qiuqian Wu, et al.. (2008). Activation of β-Catenin Signaling in Articular Chondrocytes Leads to Osteoarthritis-Like Phenotype in Adult β-Catenin Conditional Activation Mice. Journal of Bone and Mineral Research. 24(1). 12–21. 390 indexed citations
16.
Wu, Qiuqian, Erik R. Sampson, Di Chen, et al.. (2008). Induction of an osteoarthritis‐like phenotype and degradation of phosphorylated Smad3 by Smurf2 in transgenic mice. Arthritis & Rheumatism. 58(10). 3132–3144. 102 indexed citations
17.
Eliseev, Roman A., Erik R. Sampson, Michael J. Zuscik, et al.. (2008). Runx2-mediated activation of the Bax gene increases osteosarcoma cell sensitivity to apoptosis. Oncogene. 27(25). 3605–3614. 58 indexed citations
18.
Holz, Jonathan D., et al.. (2007). Environmental agents affect skeletal growth and development. Birth Defects Research Part C Embryo Today Reviews. 81(1). 41–50. 22 indexed citations
19.
Drissi, Hicham, Michael J. Zuscik, Randy N. Rosier, & Regis J. O’Keefe. (2005). Transcriptional regulation of chondrocyte maturation: Potential involvement of transcription factors in OA pathogenesis. Molecular Aspects of Medicine. 26(3). 169–179. 107 indexed citations
20.
Zuscik, Michael J., Scott A. Sands, Sean Ross, et al.. (2000). Overexpression of the α1B-adrenergic receptor causes apoptotic neurodegeneration: Multiple system atrophy. Nature Medicine. 6(12). 1388–1394. 104 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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