Takamitsu Maruyama

1.0k total citations
20 papers, 780 citations indexed

About

Takamitsu Maruyama is a scholar working on Molecular Biology, Genetics and Genetics. According to data from OpenAlex, Takamitsu Maruyama has authored 20 papers receiving a total of 780 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 10 papers in Genetics and 6 papers in Genetics. Recurrent topics in Takamitsu Maruyama's work include dental development and anomalies (6 papers), Craniofacial Disorders and Treatments (6 papers) and Connective tissue disorders research (4 papers). Takamitsu Maruyama is often cited by papers focused on dental development and anomalies (6 papers), Craniofacial Disorders and Treatments (6 papers) and Connective tissue disorders research (4 papers). Takamitsu Maruyama collaborates with scholars based in United States, Japan and Switzerland. Takamitsu Maruyama's co-authors include Wei Hsu, Anthony J. Mirando, Tzong‐Jen Sheu, Hsiao‐Man Ivy Yu, Chu‐Xia Deng, Marit H. Aure, Jiang Fu, Catherine E. Ovitt, Ming Jiang and Katsuhiko Nishimori and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Science Advances.

In The Last Decade

Takamitsu Maruyama

19 papers receiving 770 citations

Peers

Takamitsu Maruyama

GENET

SURGE

ONCOL

Yongxing Gao United States

Cynthia A. Luppen United States

Charles H. Rundle United States

Anaïs Julien France

Zhijia Tan China

Shawon Debnath United States

Shiro Baba Japan

Kimberly A. Jacobsen United States

Benjamin P. Sinder United States

S. Leah Etheridge United Kingdom

Yongxing Gao United States

Takamitsu Maruyama

742 ×1.6

133 ×0.5

GENET

217 ×1.6

GENET

140 ×1.0

SURGE

102 ×1.1

ONCOL

Citations per year, relative to Takamitsu Maruyama Takamitsu Maruyama (= 1×) peers Yongxing Gao

Countries citing papers authored by Takamitsu Maruyama

Since Specialization

Citations

This map shows the geographic impact of Takamitsu Maruyama's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takamitsu Maruyama with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takamitsu Maruyama more than expected).

Fields of papers citing papers by Takamitsu Maruyama

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takamitsu Maruyama. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takamitsu Maruyama. The network helps show where Takamitsu Maruyama may publish in the future.

Co-authorship network of co-authors of Takamitsu Maruyama

This figure shows the co-authorship network connecting the top 25 collaborators of Takamitsu Maruyama. A scholar is included among the top collaborators of Takamitsu Maruyama based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takamitsu Maruyama. Takamitsu Maruyama is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hsu, Wei & Takamitsu Maruyama. (2023). Analysis of skeletal stem cells by renal capsule transplantation and ex vivo culture systems. Frontiers in Physiology. 14. 1143344–1143344. 1 indexed citations

Wang, Shumin, et al.. (2023). miRNA-27a is essential for bone remodeling by modulating p62-mediated osteoclast signaling. eLife. 12. 6 indexed citations

Uchida, Hitoshi, Takamitsu Maruyama, & Wei Hsu. (2023). Determining Bone-forming Ability and Frequency of Skeletal Stem Cells by Kidney Capsule Transplantation and Limiting Dilution Assay. BIO-PROTOCOL. 13(6).

Shilagardi, Khurts, Takamitsu Maruyama, Wei Wu, et al.. (2022). Abolishing the prelamin A ZMPSTE24 cleavage site leads to progeroid phenotypes with near-normal longevity in mice. Proceedings of the National Academy of Sciences. 119(9). 12 indexed citations

Maruyama, Takamitsu, Hsiao‐Man Ivy Yu, & Wei Hsu. (2022). Skeletal Stem Cell Isolation from Cranial Suture Mesenchyme and Maintenance of Stemness in Culture. BIO-PROTOCOL. 12(3). e4339–e4339. 4 indexed citations

Maruyama, Takamitsu, Daigaku Hasegawa, Tomáš Valenta, et al.. (2022). GATA3 mediates nonclassical β-catenin signaling in skeletal cell fate determination and ectopic chondrogenesis. Science Advances. 8(48). eadd6172–eadd6172. 5 indexed citations

Maruyama, Takamitsu, et al.. (2021). BMPR1A maintains skeletal stem cell properties in craniofacial development and craniosynostosis. Science Translational Medicine. 13(583). 49 indexed citations

Maruyama, Takamitsu. (2019). Stem cells of the suture mesenchyme in craniofacial bone development, repair and regeneration. The Keio Journal of Medicine. 68(2). 42–42. 12 indexed citations

Aure, Marit H., et al.. (2018). Limited Regeneration of Adult Salivary Glands after Severe Injury Involves Cellular Plasticity. Cell Reports. 24(6). 1464–1470.e3. 95 indexed citations

10.

Maruyama, Takamitsu, Ming Jiang, Qirong Huang, et al.. (2017). Rap1b Is an Effector of Axin2 Regulating Crosstalk of Signaling Pathways During Skeletal Development. Journal of Bone and Mineral Research. 32(9). 1816–1828. 26 indexed citations

11.

Maruyama, Takamitsu, et al.. (2016). Stem cells of the suture mesenchyme in craniofacial bone development, repair and regeneration. Nature Communications. 7(1). 10526–10526. 183 indexed citations

12.

Yukata, Kiminori, Guoqiang Yin, Tommy Sheu, et al.. (2014). BMP-2 induces ATF4 phosphorylation in chondrocytes through a COX-2/PGE2 dependent signaling pathway. Osteoarthritis and Cartilage. 22(3). 481–489. 44 indexed citations

13.

Maruyama, Takamitsu, Ming Jiang, & Wei Hsu. (2012). Gpr177, a novel locus for bone mineral density and osteoporosis, regulates osteogenesis and chondrogenesis in skeletal development. Journal of Bone and Mineral Research. 28(5). 1150–1159. 36 indexed citations

14.

Fu, Jiang, Hsiao‐Man Ivy Yu, Takamitsu Maruyama, Anthony J. Mirando, & Wei Hsu. (2011). Gpr177/mouse Wntless is essential for Wnt‐mediated craniofacial and brain development. Developmental Dynamics. 240(2). 365–371. 85 indexed citations

15.

Lin, Congxing, Alexander Fisher, Yan Yin, et al.. (2011). The inductive role of Wnt-β-Catenin signaling in the formation of oral apparatus. Developmental Biology. 356(1). 40–50. 49 indexed citations

16.

Mirando, Anthony J., Takamitsu Maruyama, Jiang Fu, Hsiao‐Man Ivy Yu, & Wei Hsu. (2010). β-catenin/cyclin D1 mediated development of suture mesenchyme in calvarial morphogenesis. BMC Developmental Biology. 10(1). 116–116. 33 indexed citations

17.

Maruyama, Takamitsu, Anthony J. Mirando, Chu‐Xia Deng, & Wei Hsu. (2010). The Balance of WNT and FGF Signaling Influences Mesenchymal Stem Cell Fate During Skeletal Development. Science Signaling. 3(123). ra40–ra40. 100 indexed citations

18.

Hidema, Shizu, Takamitsu Maruyama, Shigekí Kato, & Katsuhiko Nishimori. (2007). Highly Induced DNA Recombination Mediated by Membrane Permeabilized Recombinant Cre Protein in Mouse Primary Cells. Bioscience Biotechnology and Biochemistry. 71(3). 817–820. 4 indexed citations

19.

Maruyama, Takamitsu, et al.. (2007). Human Smooth Muscle α-Actin Promoter Drives Cre Recombinase Expression in the Cranial Suture in Addition to Smooth Muscle Cell. Bioscience Biotechnology and Biochemistry. 71(4). 1103–1106. 4 indexed citations

20.

Murakami, Yasushi, et al.. (1982). Esophageal Reconstruction With a Skin-Grafted Pectoralis Major Muscle Flap. Archives of Otolaryngology - Head and Neck Surgery. 108(11). 719–722. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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