Ann‐Karin Olsen
- Molecular Biology
- Cancer Research top 10%
- Health, Toxicology and Mutagenesis top 5%
- Public Health, Environmental and Occupational Health
- Reproductive Medicine top 5%
- Co-authors
- Gunnar BrunborgNur DualeBirgitte LindemanRichard WigerErik J. SøderlundChristine InstanesAnne GraupnerDag M. Eide
- Topics
- Carcinogens and Genotoxicity Assessment (15 papers)DNA Repair Mechanisms (9 papers)Effects and risks of endocrine disrupting chemicals (6 papers)
- Partner nations
- NorwayNetherlandsUnited States
In The Last Decade
Ann‐Karin Olsen
40 papers receiving 917 citations
Peers
Comparison fields: 5 of 105
- Molecular Biology 377
- Cancer Research 249
- Health, Toxicology and Mutagenesis 212
- Public Health, Environmental and Occupational Health 124
- Reproductive Medicine 121
Countries citing papers authored by Ann‐Karin Olsen
This map shows the geographic impact of Ann‐Karin Olsen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ann‐Karin Olsen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ann‐Karin Olsen more than expected).
Fields of papers citing papers by Ann‐Karin Olsen
This network shows the impact of papers produced by Ann‐Karin Olsen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ann‐Karin Olsen. The network helps show where Ann‐Karin Olsen may publish in the future.
Co-authorship network of co-authors of Ann‐Karin Olsen
This figure shows the co-authorship network connecting the top 25 collaborators of Ann‐Karin Olsen. A scholar is included among the top collaborators of Ann‐Karin Olsen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ann‐Karin Olsen. Ann‐Karin Olsen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 3 | |
| 4 | 2 | |
| 5 | 21 | |
| 6 | 20 | |
| 7 | 7 | |
| 8 | 8 | |
| 9 | 18 | |
| 10 | 35 | |
| 11 | 38 | |
| 12 | 12 | |
| 13 | 18 | |
| 14 | 16 | |
| 15 | 10 | |
| 16 | 33 | |
| 17 | 36 | |
| 18 | 39 | |
| 19 | 41 | |
| 20 | 5 |
About Ann‐Karin Olsen
Ann‐Karin Olsen is a scholar working on Cancer Research, Health, Toxicology and Mutagenesis and Aging, having authored 43 papers that have together received 941 indexed citations. Recurring topics across this work include Carcinogens and Genotoxicity Assessment (15 papers), DNA Repair Mechanisms (9 papers) and Effects and risks of endocrine disrupting chemicals (6 papers). The work is most often cited by research in Cancer Research (249 citations), Health, Toxicology and Mutagenesis (212 citations) and Reproductive Medicine (121 citations). Ann‐Karin Olsen has collaborated with scholars based in Norway, Netherlands and United States. Frequent co-authors include Gunnar Brunborg, Nur Duale, Birgitte Lindeman, Richard Wiger, Erik J. Søderlund, Christine Instanes, Anne Graupner, Dag M. Eide, Kristine B. Gützkow and Dag Anders Brede. Their work appears in journals such as Nucleic Acids Research, PLoS ONE and The Science of The Total Environment.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.