Annette Schenck

9.2k total citations
64 papers, 3.5k citations indexed

About

Annette Schenck is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Annette Schenck has authored 64 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Molecular Biology, 28 papers in Genetics and 19 papers in Cellular and Molecular Neuroscience. Recurrent topics in Annette Schenck's work include Genetics and Neurodevelopmental Disorders (27 papers), Neurobiology and Insect Physiology Research (15 papers) and Mitochondrial Function and Pathology (9 papers). Annette Schenck is often cited by papers focused on Genetics and Neurodevelopmental Disorders (27 papers), Neurobiology and Insect Physiology Research (15 papers) and Mitochondrial Function and Pathology (9 papers). Annette Schenck collaborates with scholars based in Netherlands, United States and Germany. Annette Schenck's co-authors include Barbara Bardoni, Jean‐Louis Mandel, Jamie M. Kramer, Angela Giangrande, Merel A.W. Oortveld, Claudia Bagni, Christiane Zweier, Hans van Bokhoven, Nicholas Harden and Monique van der Voet and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Annette Schenck

63 papers receiving 3.5k citations

Peers

Annette Schenck

GENET

CMN

Dietrich Stephan United States

Nael Nadif Kasri Netherlands

Sean McBride United States

Laurence Colleaux France

Fred A. Pereira United States

Elizabeth J. Hong United States

Gerhard Schratt Germany

Wei Xie China

Zilong Qiu China

Lawrence T. Reiter United States

Dietrich Stephan United States

Annette Schenck

2.3k ×1.0

812 ×0.6

GENET

334 ×0.5

520 ×0.9

CMN

371 ×0.8

Citations per year, relative to Annette Schenck Annette Schenck (= 1×) peers Dietrich Stephan

Countries citing papers authored by Annette Schenck

Since Specialization

Citations

This map shows the geographic impact of Annette Schenck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Annette Schenck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Annette Schenck more than expected).

Fields of papers citing papers by Annette Schenck

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Annette Schenck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Annette Schenck. The network helps show where Annette Schenck may publish in the future.

Co-authorship network of co-authors of Annette Schenck

This figure shows the co-authorship network connecting the top 25 collaborators of Annette Schenck. A scholar is included among the top collaborators of Annette Schenck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Annette Schenck. Annette Schenck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Fencková, Michaela, Villő Muha, Daniel Mariappa, et al.. (2022). Intellectual disability-associated disruption of O-GlcNAc cycling impairs habituation learning in Drosophila. PLoS Genetics. 18(5). e1010159–e1010159. 11 indexed citations

Klein, Marieke, Ditte Demontis, Anders D. Børglum, et al.. (2020). Contribution of Intellectual Disability–Related Genes to ADHD Risk and to Locomotor Activity in Drosophila. American Journal of Psychiatry. 177(6). 526–536. 17 indexed citations

Castells‐Nobau, Anna, Ilse Eidhof, Michaela Fencková, et al.. (2019). Conserved regulation of neurodevelopmental processes and behavior by FoxP in Drosophila. PLoS ONE. 14(2). e0211652–e0211652. 24 indexed citations

Merkling, Sarah H., Pavel M. Itskov, Tjitske Kleefstra, et al.. (2019). The histone methyltransferase G9a regulates tolerance to oxidative stress–induced energy consumption. PLoS Biology. 17(3). e2006146–e2006146. 28 indexed citations

Eidhof, Ilse, Bart P.C. van de Warrenburg, & Annette Schenck. (2018). Integrative network and brain expression analysis reveals mechanistic modules in ataxia. Journal of Medical Genetics. 56(5). 283–292. 9 indexed citations

Zweier, Christiane, Bonnie Nijhof, Michaela Fencková, et al.. (2016). Systematic Phenomics Analysis Deconvolutes Genes Mutated in Intellectual Disability into Biologically Coherent Modules. The American Journal of Human Genetics. 98(1). 149–164. 189 indexed citations

Gupta, Varun, Lisa Scheunemann, Tobias Eisenberg, et al.. (2013). Restoring polyamines protects from age-induced memory impairment in an autophagy-dependent manner. Nature Neuroscience. 16(10). 1453–1460. 281 indexed citations

Galy, Anne, et al.. (2011). CYFIP dependent Actin Remodeling controls specific aspects of Drosophila eye morphogenesis. Developmental Biology. 359(1). 37–46. 11 indexed citations

Kramer, Jamie M., Merel A.W. Oortveld, Hendrik Marks, et al.. (2011). Epigenetic Regulation of Learning and Memory by Drosophila EHMT/G9a. PLoS Biology. 9(1). e1000569–e1000569. 141 indexed citations

10.

Zweier, Christiane, Eiko K. de Jong, Markus Zweier, et al.. (2009). CNTNAP2 and NRXN1 Are Mutated in Autosomal-Recessive Pitt-Hopkins-like Mental Retardation and Determine the Level of a Common Synaptic Protein in Drosophila. The American Journal of Human Genetics. 85(5). 655–666. 244 indexed citations

11.

Kramer, Jamie M., Khalid Hussain, Joris H. Robben, et al.. (2009). SLC29A3 gene is mutated in pigmented hypertrichosis with insulin-dependent diabetes mellitus syndrome and interacts with the insulin signaling pathway. Human Molecular Genetics. 18(12). 2257–2265. 85 indexed citations

12.

Qurashi, Abrar, et al.. (2007). HSPC300 and its role in neuronal connectivity. Neural Development. 2(1). 18–18. 27 indexed citations

13.

Kim, Yong, Jee Young Sung, Ilaria Ceglia, et al.. (2006). Phosphorylation of WAVE1 regulates actin polymerization and dendritic spine morphology. Nature. 442(7104). 814–817. 251 indexed citations

14.

Schenck, Annette, Abrar Qurashi, Pilar Carrera, et al.. (2004). WAVE/SCAR, a multifunctional complex coordinating different aspects of neuronal connectivity. Developmental Biology. 274(2). 260–270. 59 indexed citations

15.

Mayne, Michael, Hong Kong, Paul J. McLaren, et al.. (2004). CYFIP2 is highly abundant in CD4⁺ cells from multiple sclerosis patients and is involved in T cell adhesion. European Journal of Immunology. 34(4). 1217–1227. 32 indexed citations

16.

Schenck, Annette, et al.. (2003). CYFIP/Sra-1 Controls Neuronal Connectivity in Drosophila and Links the Rac1 GTPase Pathway to the Fragile X Protein. Neuron. 38(6). 887–898. 255 indexed citations

17.

Ragone, Gianluca, Martial Kammerer, Anne Galy, et al.. (2003). Transcriptional regulation of glial cell specification. Developmental Biology. 255(1). 138–150. 26 indexed citations

18.

Bardoni, Barbara, Annette Schenck, & Jean‐Louis Mandel. (2001). The Fragile X mental retardation protein. Brain Research Bulletin. 56(3-4). 375–382. 66 indexed citations

19.

Schenck, Annette, et al.. (2001). A highly conserved protein family interacting with the fragile X mental retardation protein (FMRP) and displaying selective interactions with FMRP-related proteins FXR1P and FXR2P. Proceedings of the National Academy of Sciences. 98(15). 8844–8849. 291 indexed citations

20.

Bardoni, Barbara, Annette Schenck, & Jean‐Louis Mandel. (1999). A Novel RNA-binding Nuclear Protein That Interacts With the Fragile X Mental Retardation (FMR1) Protein. Human Molecular Genetics. 8(13). 2557–2566. 107 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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