Peter Schmezer

8.7k total citations · 2 hit papers
138 papers, 5.8k citations indexed

About

Peter Schmezer is a scholar working on Molecular Biology, Cancer Research and Health, Toxicology and Mutagenesis. According to data from OpenAlex, Peter Schmezer has authored 138 papers receiving a total of 5.8k indexed citations (citations by other indexed papers that have themselves been cited), including 86 papers in Molecular Biology, 63 papers in Cancer Research and 24 papers in Health, Toxicology and Mutagenesis. Recurrent topics in Peter Schmezer's work include Carcinogens and Genotoxicity Assessment (54 papers), DNA Repair Mechanisms (39 papers) and Epigenetics and DNA Methylation (15 papers). Peter Schmezer is often cited by papers focused on Carcinogens and Genotoxicity Assessment (54 papers), DNA Repair Mechanisms (39 papers) and Epigenetics and DNA Methylation (15 papers). Peter Schmezer collaborates with scholars based in Germany, United States and United Kingdom. Peter Schmezer's co-authors include Odilia Popanda, Diana Anderson, B.L. Pool-Zobel, Barbara Phillips, Tao Yu, Valerie J. McKelvey‐Martin, M.H.L. Green, Andrew Collins, M.P. De Méo and Helmut Bartsch and has published in prestigious journals such as Nucleic Acids Research, Nature Communications and Hepatology.

In The Last Decade

Peter Schmezer

138 papers receiving 5.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The single cell gel electrophoresis assay (comet assay): A European review

1993 777 citations Peter Schmezer, B.L. Pool-Zobel et al. Mutation research. Fundamental and molecular mechanisms of mutagenesis profile →
The effect of various antioxidants and other modifying agents on oxygen-radical-generated DNA damage in human lymphocytes in the COMET assay

1994 564 citations Peter Schmezer et al. Mutation research. Fundamental and molecular mechanisms of mutagenesis profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Peter Schmezer Germany	42	2.7k	2.3k	969	839	735	138	5.8k
Alberto Izzotti Italy	53	4.6k 1.7×	2.7k 1.2×	1.2k 1.2×	590 0.7×	738 1.0×	258	9.2k
Richard J. Albertini United States	42	2.7k 1.0×	2.1k 0.9×	971 1.0×	622 0.7×	420 0.6×	137	5.6k
Hideki Wanibuchi Japan	40	2.7k 1.0×	1.3k 0.6×	1.0k 1.1×	946 1.1×	240 0.3×	305	6.2k
Paul T. Strickland United States	45	1.9k 0.7×	1.8k 0.8×	2.0k 2.0×	1.0k 1.2×	153 0.2×	181	6.1k
Eliezer Huberman United States	55	5.9k 2.2×	2.5k 1.1×	690 0.7×	1.6k 1.9×	332 0.5×	153	9.3k
David H. Sherr United States	48	3.7k 1.4×	1.9k 0.8×	1.4k 1.5×	1.5k 1.7×	244 0.3×	142	9.0k
F. Darroudi Netherlands	41	2.4k 0.9×	2.3k 1.0×	749 0.8×	291 0.3×	544 0.7×	118	4.6k
Eun Hee Kim South Korea	45	3.6k 1.3×	1.1k 0.5×	853 0.9×	888 1.1×	141 0.2×	155	6.5k
Ryszard Oliński Poland	44	3.7k 1.4×	1.4k 0.6×	519 0.5×	575 0.7×	156 0.2×	164	5.8k
Pavel Vodička Czechia	44	4.0k 1.5×	3.3k 1.5×	777 0.8×	1.6k 1.9×	145 0.2×	223	7.3k

Countries citing papers authored by Peter Schmezer

Since Specialization

Citations

This map shows the geographic impact of Peter Schmezer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter Schmezer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter Schmezer more than expected).

Fields of papers citing papers by Peter Schmezer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter Schmezer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter Schmezer. The network helps show where Peter Schmezer may publish in the future.

Co-authorship network of co-authors of Peter Schmezer

This figure shows the co-authorship network connecting the top 25 collaborators of Peter Schmezer. A scholar is included among the top collaborators of Peter Schmezer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter Schmezer. Peter Schmezer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Corte, G, Mei-Ju May Chen, Yu‐Yu Lin, et al.. (2024). ARID1A regulates DNA repair through chromatin organization and its deficiency triggers DNA damage-mediated anti-tumor immune response. Nucleic Acids Research. 52(10). 5698–5719. 23 indexed citations

Liu, Chunshan, Réka Tóth, Ashish Goyal, et al.. (2021). Epigenetic Modulation of Radiation-Induced Diacylglycerol Kinase Alpha Expression Prevents Pro-Fibrotic Fibroblast Response. Cancers. 13(10). 2455–2455. 8 indexed citations

Popanda, Odilia, Katja Butterbach, Dominic Edelmann, et al.. (2019). DNA Methylation Profiling to Explore Colorectal Tumor Differences According to Menopausal Hormone Therapy Use in Women. Epigenomics. 11(16). 1765–1778. 3 indexed citations

Perez, Ramon Lopez, et al.. (2019). DNA damage response of clinical carbon ion versus photon radiation in human glioblastoma cells. Radiotherapy and Oncology. 133. 77–86. 37 indexed citations

Henrich, Kai-Oliver, Sebastian Bender, Maral Saadati, et al.. (2016). Integrative Genome-Scale Analysis Identifies Epigenetic Mechanisms of Transcriptional Deregulation in Unfavorable Neuroblastomas. Cancer Research. 76(18). 5523–5537. 73 indexed citations

Kantor, Elizabeth D., Cornelia M. Ulrich, Robert W. Owen, et al.. (2013). Specialty Supplement Use and Biologic Measures of Oxidative Stress and DNA Damage. Cancer Epidemiology Biomarkers & Prevention. 22(12). 2312–2322. 12 indexed citations

Lehle, Simon, Dominic G. Hildebrand, Michael Becker, et al.. (2013). LORD-Q: a long-run real-time PCR-based DNA-damage quantification method for nuclear and mitochondrial genome analysis. Nucleic Acids Research. 42(6). e41–e41. 45 indexed citations

Abbasi, Rashda, Thomas Efferth, Till Opatz, et al.. (2012). The endoperoxide ascaridol shows strong differential cytotoxicity in nucleotide excision repair-deficient cells. Toxicology and Applied Pharmacology. 259(3). 302–310. 19 indexed citations

Greve, Burkhard, Stefan Könemann, Eberhard Fritz, et al.. (2009). Multicentric investigation of ionising radiation-induced cell death as a predictive parameter of individual radiosensitivity. APOPTOSIS. 14(2). 226–235. 18 indexed citations

10.

Popp, Susanne, Sharareh Moshir, Karin Scharffetter‐Kochanek, et al.. (2008). UVA radiation causes DNA strand breaks, chromosomal aberrations and tumorigenic transformation in HaCaT skin keratinocytes. Oncogene. 27(31). 4269–4280. 78 indexed citations

11.

Schmezer, Peter & Christoph Plass. (2008). Epigenetische Aspekte bei Karzinomen der Kopf-Hals-Region. HNO. 56(6). 594–602. 4 indexed citations

12.

Boukamp, Petra, et al.. (2007). Improved alkaline comet assay protocol for adherent HaCaT keratinocytes to study UVA-induced DNA damage. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 630(1-2). 122–128. 11 indexed citations

13.

Kropp, Silke, Irmgard Helmbold, Peter Schmezer, et al.. (2007). Genetic predictors of long‐term toxicities after radiation therapy for breast cancer. International Journal of Cancer. 122(6). 1333–1339. 42 indexed citations

14.

Schmezer, Peter, et al.. (2001). MUTAGEN SENSITIVITY AND DNA REPAIR CAPACITY （DRC） AS RISK FACTORS FOR NONSMALL CELL LUNG CANCER. 13(4). 207–208. 1 indexed citations

15.

Schmezer, Peter, Tobias A. Rupprecht, M. Tisch, H. Maier, & Helmut Bartsch. (2000). Laryngeal mucosa of head and neck cancer patients shows increased DNA damage as detected by single cell microgel electrophoresis. Toxicology. 144(1-3). 149–154. 3 indexed citations

16.

Wikman, Harriet, Steffen Thiel, Peter Schmezer, et al.. (1999). Genetic polymorphisms in GSTs and individual susceptibility to lung cancer. Lung Cancer. 25. S14–S15. 1 indexed citations

17.

Engelhardt, G., et al.. (1997). P XIV B.24 Genotoxic aldehydes produce specific comet assay images. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 379(1). S133–S133. 1 indexed citations

18.

Pool-Zobel, B.L., et al.. (1992). Systemic genotoxic effects of tobacco-related nitrosamines following oral and inhalational administration to Sprague-Dawley rats. Journal of Molecular Medicine. 70-70(3-4). 83 indexed citations

19.

Pool, B.L., et al.. (1990). Employment of adult mammalian primary cells in toxicology: In vivo and in vitro genotoxic effects of environmentally significant N‐nitrosodialkylamines in cells of the liver, lung, and kidney. Environmental and Molecular Mutagenesis. 15(1). 24–35. 28 indexed citations

20.

Pool, B.L., Gerhard Eisenbrand, R. Preußmann, et al.. (1986). Detection of mutations in bacteria and of DNA damage and amplified DNA sequences in mammalian cells as a systematic test strategy for elucidating biological activities of chemical carcinogens. Food and Chemical Toxicology. 24(6-7). 685–691. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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