Ann E. Rougvie

8.4k citations

33 papers · 6.4k indexed · 2 hit papers · h-index 25

Impact in

Aging top 0.05%
- Genetics, Aging, and Longevity in Model Organisms
Cancer Research top 0.2%
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research

Papers in

Aging 25
- Genetics, Aging, and Longevity in Model Organisms 25
Endocrine and Autonomic Systems 10
- Circadian rhythm and melatonin 10

Co-authors: Jill C. Bettinger Brenda J. Reinhart H. Robert Horvitz Amy E. Pasquinelli Michael Basson Gary Ruvkun Frank J. Slack John T. Lis
Journals: Development (5 papers)Current Biology (2 papers)Nature (2 papers)Developmental Biology (2 papers)Genome Research (2 papers)
Partner nations: United States Japan Canada

In The Last Decade

Ann E. Rougvie

33 papers receiving 6.3k citations

Hit Papers

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The 21-nucleotide let-7 RNA regulates developmental timing in Caenorhabditis elegans 2000 · 3.7k citations

Peers

Countries citing papers authored by Ann E. Rougvie

Since Specialization

Citations

This map shows the geographic impact of Ann E. Rougvie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ann E. Rougvie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ann E. Rougvie more than expected).

Fields of papers citing papers by Ann E. Rougvie

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ann E. Rougvie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ann E. Rougvie. The network helps show where Ann E. Rougvie may publish in the future.

Co-authors

The 25 scholars most cited alongside Ann E. Rougvie, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ann E. Rougvie Line = papers co-authored together Ann E. Rougvie links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	CGC1, a new reference genome for Caenorhabditis elegans Genome Research ·Kazuki Ichikawa, Massa J. Shoura, Karen L. Artiles, Dae‐Eun Jeong, Chie Owa, Haruka Kobayashi, Yoshihiko Suzuki, Manami Kanamori, Yu Toyoshima, Yuichi Iino, Ann E. Rougvie, Lamia Wahba, Andrew Fire, Erich M. Schwarz, Shinichi Morishita	2025	1
2	From nematode to Nobel: How community-shared resources fueled the rise of Caenorhabditis elegans as a research organism Proceedings of the National Academy of Sciences ·Victor Ambros, Martin Chalfie, Aric L. Daul, Andrew Fire, David H. Hall, H. Robert Horvitz, Craig C. Mello, Gary Ruvkun, Nathan E. Schroeder, Paul W. Sternberg, Ann E. Rougvie	2025	1
3	Recompleting the Caenorhabditis elegans genome Genome Research ·Jun Yoshimura, Kazuki Ichikawa, Massa J. Shoura, Karen L. Artiles, Idan Gabdank, Lamia Wahba, Cheryl L. Smith, Mark L. Edgley, Ann E. Rougvie, Andrew Fire, Shinichi Morishita, Erich M. Schwarz	2019	75
4	The C. elegans Hypodermis Couples Progenitor Cell Quiescence to the Dietary State Current Biology ·Masamitsu Fukuyama, Kenji Kontani, Toshiaki Katada, Ann E. Rougvie	2015	37
5	Developmental Transitions in C. elegans Larval Stages Current topics in developmental biology ·Ann E. Rougvie, Eric G. Moss	2013	86
6	TheC. elegansdevelopmental timing protein LIN-42 regulates diapause in response to environmental cues Development ·Jason M. Tennessen, Karla J. Opperman, Ann E. Rougvie	2010	27
7	miRNAs give worms the time of their lives: Small RNAs and temporal control in Caenorhabditis elegans Developmental Dynamics ·Tamar D. Resnick, Katherine A McCulloch, Ann E. Rougvie	2010	46
8	C. elegans DAF-18/PTEN Mediates Nutrient-Dependent Arrest of Cell Cycle and Growth in the Germline Current Biology ·Masamitsu Fukuyama, Ann E. Rougvie, Joel H. Rothman	2006	157
9	Regulatory Mutations of mir-48, a C. elegans let-7 Family MicroRNA, Cause Developmental Timing Defects Developmental Cell ·Ming Li, Matthew W. Jones-Rhoades, Nelson C. Lau, David P. Bartel, Ann E. Rougvie	2005	79
10	Novel heterochronic functions of the Caenorhabditis elegans period-related protein LIN-42 Developmental Biology ·Jason M. Tennessen, Heather F. Gardner, Mandy L. Volk, Ann E. Rougvie	2005	56
11	The Caenorhabditis elegans hunchback-like Gene lin-57/hbl-1 Controls Developmental Time and Is Regulated by MicroRNAs Developmental Cell ·Juan E. Abrahante, Aric L. Daul, Ming Li, Mandy L. Volk, Jason M. Tennessen, Eric A. Miller, Ann E. Rougvie	2003	299
12	Chromosome cohesion is regulated by a clock gene paralogue TIM-1 Nature ·Raymond C. Chan, Annette Chan, Mili Jeon, Tammy F. Wu, Danielle Pasqualone, Ann E. Rougvie, Barbara J Meyer	2003	139
13	Control of developmental timing in animals Nature Reviews Genetics ·Ann E. Rougvie	2001	105
14	The 21-nucleotide let-7 RNA regulates developmental timing in Caenorhabditis elegans Nature ·Brenda J. Reinhart, Frank J. Slack, Michael Basson, Amy E. Pasquinelli, Jill C. Bettinger, Ann E. Rougvie, H. Robert Horvitz, Gary Ruvkun Hit paper breakdown →	2000	3710
15	The LIN-29 Transcription Factor Is Required for Proper Morphogenesis of theCaenorhabditis elegansMale Tail Developmental Biology ·Susan Euling, Jill C. Bettinger, Ann E. Rougvie	1999	21
16	Stage-specific accumulation of the terminal differentiation factor LIN-29 during Caenorhabditis elegans development Development ·Jill C. Bettinger, Kimberly Lee, Ann E. Rougvie	1996	98
17	Chapter 14 Protein—DNA Cross-Linking as a Means to Determine the Distribution of Proteins on DNA in Vivo Methods in cell biology ·David S. Gilmour, Ann E. Rougvie, John T. Lis	1991	27
18	Molecular cloning oflin-29, a heterochronic gene required for the differentiation of hypodermal cells and the cessation of molting inC.elegans Nucleic Acids Research ·A C Papp, Ann E. Rougvie, Victor Ambros	1991	17
19	Postinitiation Transcriptional Control in Drosophila melanogaster Molecular and Cellular Biology ·Ann E. Rougvie, John T. Lis	1990	164
20	The RNA polymerase II molecule at the 5′ end of the uninduced hsp70 gene of D. melanogaster is transcriptionally engaged Cell ·Ann E. Rougvie, John T. Lis Hit paper breakdown →	1988	562

About Ann E. Rougvie

Ann E. Rougvie is a scholar working on Aging, Endocrine and Autonomic Systems, Cancer Research, Molecular Biology and Genetics, having authored 33 papers that have together received 6.4k indexed citations. Recurring topics across this work include Genetics, Aging, and Longevity in Model Organisms (25 papers), Circadian rhythm and melatonin (10 papers), CRISPR and Genetic Engineering (8 papers), RNA Research and Splicing (5 papers), MicroRNA in disease regulation (4 papers), Reproductive Biology and Fertility (4 papers), Spaceflight effects on biology (3 papers) and Pluripotent Stem Cells Research (3 papers). The work is most often cited by research in Aging (1.6k citations), Cancer Research (3.1k citations), Endocrine and Autonomic Systems (543 citations), Molecular Biology (4.8k citations) and Plant Science (613 citations). Ann E. Rougvie has collaborated with scholars based in United States, Japan and Canada. Frequent co-authors include Jill C. Bettinger, Brenda J. Reinhart, H. Robert Horvitz, Amy E. Pasquinelli, Michael Basson, Gary Ruvkun, Frank J. Slack, John T. Lis, John T. Lis and Eric A. Miller. Their work appears in journals such as Development, Current Biology, Nature, Developmental Biology and Genome Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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