Andrew M. Tan

2.0k total citations
44 papers, 1.5k citations indexed

About

Andrew M. Tan is a scholar working on Physiology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Andrew M. Tan has authored 44 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Physiology, 17 papers in Cellular and Molecular Neuroscience and 11 papers in Molecular Biology. Recurrent topics in Andrew M. Tan's work include Pain Mechanisms and Treatments (20 papers), Spinal Cord Injury Research (10 papers) and Nerve injury and regeneration (9 papers). Andrew M. Tan is often cited by papers focused on Pain Mechanisms and Treatments (20 papers), Spinal Cord Injury Research (10 papers) and Nerve injury and regeneration (9 papers). Andrew M. Tan collaborates with scholars based in United States, Canada and United Kingdom. Andrew M. Tan's co-authors include Stephen G. Waxman, Peng Zhao, Joel M. Levine, Bryan C. Hains, Omar Abdel Samad, Yu-Wen Chang, Sulayman D. Dib‐Hajj, Weibing Zhang, Tanya Fischer and Corinna Darian‐Smith and has published in prestigious journals such as Journal of Neuroscience, Brain and The Journal of Physiology.

In The Last Decade

Andrew M. Tan

41 papers receiving 1.5k citations

Peers

Andrew M. Tan

PHYSI

CMN

PFM

NEURO

James R. Goss United States

Christopher R. Donnelly United States

Alexander Chamessian United States

Kyle M. Baumbauer United States

Paul D. Morton United States

Joshua P. Klein United States

Sandro Dá Mesquita United States

Stephanie Shiers United States

Geza Gemes Austria

Takeshi Kaneko Japan

James R. Goss United States

Andrew M. Tan

622 ×0.9

PHYSI

998 ×1.6

CMN

778 ×2.2

104 ×0.3

PFM

436 ×2.0

NEURO

Citations per year, relative to Andrew M. Tan Andrew M. Tan (= 1×) peers James R. Goss

Countries citing papers authored by Andrew M. Tan

Since Specialization

Citations

This map shows the geographic impact of Andrew M. Tan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew M. Tan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew M. Tan more than expected).

Fields of papers citing papers by Andrew M. Tan

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andrew M. Tan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew M. Tan. The network helps show where Andrew M. Tan may publish in the future.

Co-authorship network of co-authors of Andrew M. Tan

This figure shows the co-authorship network connecting the top 25 collaborators of Andrew M. Tan. A scholar is included among the top collaborators of Andrew M. Tan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew M. Tan. Andrew M. Tan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Tan, Andrew M., et al.. (2025). Pridopidine, a Potent and Selective Therapeutic Sigma-1 Receptor (S1R) Agonist for Treating Neurodegenerative Diseases. Pharmaceuticals. 18(12). 1900–1900.

Cruz-Herranz, Andrés, Kelly Chen, Katrin Spiegel, et al.. (2025). The Safety Profile of Pridopidine, a Novel Sigma-1 Receptor Agonist for the Treatment of Huntington’s Disease. CNS Drugs. 39(5). 485–498. 3 indexed citations

Geva, Michal, Y. Paul Goldberg, Melanie Leitner, et al.. (2025). Pridopidine treatment in ALS: subgroup analyses from the HEALEY ALS Platform trial. Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration. 1–13.

Benson, Curtis A., et al.. (2024). PAK1 inhibition with Romidepsin attenuates H‐reflex hyperexcitability after spinal cord injury. The Journal of Physiology. 602(19). 5061–5081. 2 indexed citations

Benson, Curtis A., et al.. (2023). Conditional Astrocyte Rac1KO Attenuates Hyperreflexia after Spinal Cord Injury. Journal of Neuroscience. 44(1). e1670222023–e1670222023. 6 indexed citations

Benson, Curtis A., et al.. (2021). Conditional RAC1 knockout in motor neurons restores H-reflex rate-dependent depression after spinal cord injury. Scientific Reports. 11(1). 7838–7838. 11 indexed citations

Chen, Lubin, Jianying Huang, Curtis A. Benson, et al.. (2020). Sodium channel Nav1.6 in sensory neurons contributes to vincristine-induced allodynia. Brain. 143(8). 2421–2436. 30 indexed citations

Burke, Hannah, et al.. (2016). A multidisciplinary team case management approach reduces the burden of frequent asthma admissions. ERJ Open Research. 2(3). 39–2016. 31 indexed citations

Tan, Andrew M., Omar Abdel Samad, Sulayman D. Dib‐Hajj, & Stephen G. Waxman. (2015). Virus-Mediated Knockdown of Nav1.3 in Dorsal Root Ganglia of STZ-Induced Diabetic Rats Alleviates Tactile Allodynia. Molecular Medicine. 21(1). 544–552. 60 indexed citations

10.

Tan, Andrew M., et al.. (2014). The impact of input from a psychologist on a cohort of asthmatic frequent fliers. European Respiratory Journal. 44(Suppl 58). 2940–2940. 1 indexed citations

11.

Tan, Andrew M., et al.. (2014). The impact of a specialist multi-disciplinary approach on burden of frequent asthma admissions. European Respiratory Journal. 44(Suppl 58). P3009–P3009. 1 indexed citations

12.

Tan, Andrew M. & Stephen G. Waxman. (2014). Dendritic spine dysgenesis in neuropathic pain. Neuroscience Letters. 601. 54–60. 32 indexed citations

13.

Tan, Andrew M., et al.. (2013). Burn injury-induced mechanical allodynia is maintained by Rac1-regulated dendritic spine dysgenesis. Experimental Neurology. 248. 509–519. 33 indexed citations

14.

Samad, Omar Abdel, Andrew M. Tan, Xiaoyang Cheng, et al.. (2012). Virus-mediated shRNA Knockdown of Nav1.3 in Rat Dorsal Root Ganglion Attenuates Nerve Injury-induced Neuropathic Pain. Molecular Therapy. 21(1). 49–56. 91 indexed citations

15.

Tan, Andrew M., Yu-Wen Chang, Peng Zhao, Bryan C. Hains, & Stephen G. Waxman. (2011). Rac1-regulated dendritic spine remodeling contributes to neuropathic pain after peripheral nerve injury. Experimental Neurology. 232(2). 222–233. 63 indexed citations

16.

Ho, Hao Chih, et al.. (2009). Preventing Loss of Domain: A Management Strategy for Closure of the “Open Abdomen” During the Initial Hospitalization. Journal of surgical education. 66(2). 89–95. 24 indexed citations

17.

Fischer, Tanya, Andrew M. Tan, & Stephen G. Waxman. (2009). Thalamic neuron hyperexcitability and enlarged receptive fields in the STZ model of diabetic pain. Brain Research. 1268. 154–161. 56 indexed citations

18.

Tan, Andrew M., Séverine Stamboulian, Yu-Wen Chang, et al.. (2008). Neuropathic Pain Memory Is Maintained by Rac1-Regulated Dendritic Spine Remodeling after Spinal Cord Injury. Journal of Neuroscience. 28(49). 13173–13183. 102 indexed citations

19.

Tan, Andrew M., Weibing Zhang, & Joel M. Levine. (2005). NG2: a component of the glial scar that inhibits axon growth. Journal of Anatomy. 207(6). 717–725. 105 indexed citations

20.

Goller, Jane L., et al.. (2004). Long-term features of norovirus gastroenteritis in the elderly. Journal of Hospital Infection. 58(4). 286–291. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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