Yiting Jia
Impact in
- Immunology and Allergy top 10%
- Cell Adhesion Molecules Research
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- Protease and Inhibitor Mechanisms
- Cancer-related molecular mechanisms research
Papers in
-
- RNA modifications and cancer 5
- Cancer-related gene regulation 2
-
- Protease and Inhibitor Mechanisms 3
- Cancer, Hypoxia, and Metabolism 2
- Co-authors
- Wei Kong (13 shared papers)Chenfeng Mao (7 shared papers)Zihan Ma (6 shared papers)Yi Fu (10 shared papers)Yi Fu (2 shared papers)Qingbo Xu (4 shared papers)Yuan Zhou (4 shared papers)Qinghua Cui (2 shared papers)
- Journals
- Circulation Research (4 papers)Circulation (2 papers)Journal of the American Society of Nephrology (1 paper)Cell Death and Disease (1 paper)Cardiovascular Research (1 paper)
- Partner nations
- ChinaSouth KoreaUnited Kingdom
In The Last Decade
Yiting Jia
14 papers receiving 433 citations
Peers
Comparison fields: 5 of 69
- Immunology and Allergy 46
- Cancer Research 109
- Immunology 76
- Nephrology 25
- Molecular Biology 216
Countries citing papers authored by Yiting Jia
This map shows the geographic impact of Yiting Jia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yiting Jia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yiting Jia more than expected).
Fields of papers citing papers by Yiting Jia
This network shows the impact of papers produced by Yiting Jia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yiting Jia. The network helps show where Yiting Jia may publish in the future.
Co-authors
The 25 scholars most cited alongside Yiting Jia, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 84 | |
| 2 | 2021 | 64 | |
| 3 | 2022 | 60 | |
| 4 | 2016 | 54 | |
| 5 | 2022 | 42 | |
| 6 | 2018 | 27 | |
| 7 | 2022 | 27 | |
| 8 | 2022 | 26 | |
| 9 | 2016 | 23 | |
| 10 | 2023 | 14 | |
| 11 | 2021 | 8 | |
| 12 | 2023 | 3 | |
| 13 | 2025 | 2 | |
| 14 | 2025 | 1 |
About Yiting Jia
Yiting Jia is a scholar working on Molecular Biology, Cancer Research, Immunology and Allergy, Nephrology and Genetics, having authored 14 papers that have together received 435 indexed citations. Recurring topics across this work include RNA modifications and cancer (5 papers), Cell Adhesion Molecules Research (4 papers), Protease and Inhibitor Mechanisms (3 papers), Cancer, Hypoxia, and Metabolism (2 papers), Cancer-related gene regulation (2 papers), Peptidase Inhibition and Analysis (2 papers), Connective tissue disorders research (1 paper) and Hemoglobinopathies and Related Disorders (1 paper). The work is most often cited by research in Immunology and Allergy (46 citations), Cancer Research (109 citations), Immunology (76 citations), Nephrology (25 citations) and Molecular Biology (216 citations). Yiting Jia has collaborated with scholars based in China, South Korea and United Kingdom. Frequent co-authors include Wei Kong, Chenfeng Mao, Zihan Ma, Yi Fu, Yi Fu, Qingbo Xu, Yuan Zhou, Qinghua Cui, Meili Wang and Jin‐Peng Sun. Their work appears in journals such as Circulation Research, Circulation, Journal of the American Society of Nephrology, Cell Death and Disease and Cardiovascular Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.