Alfredo Lorenzo
- Physiology top 0.5%
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 2%
- Pharmacology top 1%
- Neurology top 1%
- Co-authors
- Bruce A. YanknerJorge BusciglioBronwyn L. RazzaboniGordon C. WeirJohn YehMenglan YuanChuang-Kuo WuDaniel Saroff
- Topics
- Alzheimer's disease research and treatments (19 papers)Prion Diseases and Protein Misfolding (10 papers)Neuroscience and Neuropharmacology Research (9 papers)
- Partner nations
- ArgentinaUnited StatesGermany
In The Last Decade
Alfredo Lorenzo
34 papers receiving 4.5k citations
Hit Papers
Peers
Comparison fields: 5 of 110
- Physiology 3.1k
- Molecular Biology 2.1k
- Cellular and Molecular Neuroscience 874
- Pharmacology 776
- Neurology 582
Countries citing papers authored by Alfredo Lorenzo
This map shows the geographic impact of Alfredo Lorenzo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alfredo Lorenzo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alfredo Lorenzo more than expected).
Fields of papers citing papers by Alfredo Lorenzo
This network shows the impact of papers produced by Alfredo Lorenzo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alfredo Lorenzo. The network helps show where Alfredo Lorenzo may publish in the future.
Co-authorship network of co-authors of Alfredo Lorenzo
This figure shows the co-authorship network connecting the top 25 collaborators of Alfredo Lorenzo. A scholar is included among the top collaborators of Alfredo Lorenzo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alfredo Lorenzo. Alfredo Lorenzo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 10 | |
| 2 | 6 | |
| 3 | 14 | |
| 4 | 10 | |
| 5 | 10 | |
| 6 | 34 | |
| 7 | 75 | |
| 8 | 8 | |
| 9 | 16 | |
| 10 | 13 | |
| 11 | 33 | |
| 12 | 63 | |
| 13 | 21 | |
| 14 | 155 | |
| 15 | 441 | |
| 16 | 135 | |
| 17 | 202 | |
| 18 | β-Amyloid fibrils induce tau phosphorylation and loss of microtubule bindingbreakdown → | 508 |
| 19 | 79 | |
| 20 | 85 |
About Alfredo Lorenzo
Alfredo Lorenzo is a scholar working on Behavioral Neuroscience, Neurology and Physiology, having authored 35 papers that have together received 4.6k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (19 papers), Prion Diseases and Protein Misfolding (10 papers) and Neuroscience and Neuropharmacology Research (9 papers). The work is most often cited by research in Physiology (3.1k citations), Neurology (582 citations) and Biological Psychiatry (147 citations). Alfredo Lorenzo has collaborated with scholars based in Argentina, United States and Germany. Frequent co-authors include Bruce A. Yankner, Jorge Busciglio, Bronwyn L. Razzaboni, Gordon C. Weir, John Yeh, Menglan Yuan, Chuang-Kuo Wu, Daniel Saroff, Alfredo Cáceres and Bin Shi. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.