Alexandra Sobeck

1.8k total citations
24 papers, 1.4k citations indexed

About

Alexandra Sobeck is a scholar working on Molecular Biology, Cancer Research and Cell Biology. According to data from OpenAlex, Alexandra Sobeck has authored 24 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 12 papers in Cancer Research and 6 papers in Cell Biology. Recurrent topics in Alexandra Sobeck's work include DNA Repair Mechanisms (24 papers), Carcinogens and Genotoxicity Assessment (12 papers) and Genomics and Chromatin Dynamics (10 papers). Alexandra Sobeck is often cited by papers focused on DNA Repair Mechanisms (24 papers), Carcinogens and Genotoxicity Assessment (12 papers) and Genomics and Chromatin Dynamics (10 papers). Alexandra Sobeck collaborates with scholars based in United States, Germany and United Kingdom. Alexandra Sobeck's co-authors include Maureen E. Hoatlin, Indrajit Chaudhury, Weidong Wang, Archana Sareen, Eric A. Hendrickson, Stacie Stone, Jinhu Yin, Lei Li, Amom Ruhikanta Meetei and Chang Xu and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Genes & Development.

In The Last Decade

Alexandra Sobeck

24 papers receiving 1.3k citations

Peers

Alexandra Sobeck

ONCOL

GENET

Alan D. D’Andrea United States

Ken‐ichi Morishima Japan

Michelle Le Beau United States

Simone Sabbioneda Italy

Kelly M. Trujillo United States

David Ciccone United States

Wojciech Niedźwiedź United Kingdom

Mitsuo Wakasugi Japan

Jacqueline H. Barlow United States

Satoshi Tateishi Japan

Alan D. D’Andrea United States

Alexandra Sobeck

1.0k ×0.8

290 ×0.7

344 ×1.2

ONCOL

174 ×0.9

181 ×1.0

GENET

Citations per year, relative to Alexandra Sobeck Alexandra Sobeck (= 1×) peers Alan D. D’Andrea

Countries citing papers authored by Alexandra Sobeck

Since Specialization

Citations

This map shows the geographic impact of Alexandra Sobeck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexandra Sobeck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexandra Sobeck more than expected).

Fields of papers citing papers by Alexandra Sobeck

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alexandra Sobeck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexandra Sobeck. The network helps show where Alexandra Sobeck may publish in the future.

Co-authorship network of co-authors of Alexandra Sobeck

This figure shows the co-authorship network connecting the top 25 collaborators of Alexandra Sobeck. A scholar is included among the top collaborators of Alexandra Sobeck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexandra Sobeck. Alexandra Sobeck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Leung, Wendy, Ryan M. Baxley, Liangjun Wang, et al.. (2023). FANCD2-dependent mitotic DNA synthesis relies on PCNA K164 ubiquitination. Cell Reports. 42(12). 113523–113523. 8 indexed citations

Lin, Kevin, et al.. (2023). Fanconi anemia-associated chromosomal radial formation is dependent on POLθ-mediated alternative end joining. Cell Reports. 42(5). 112428–112428. 5 indexed citations

Shameem, Mohammad, et al.. (2023). Mitotic DNA Synthesis in Untransformed Human Cells Preserves Common Fragile Site Stability via a FANCD2-Driven Mechanism That Requires HELQ. Journal of Molecular Biology. 435(22). 168294–168294. 5 indexed citations

Okamoto, Yusuke, Masako Abe, Anfeng Mu, et al.. (2020). SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells. Blood. 137(3). 336–348. 25 indexed citations

Baxley, Ryan M., Fredy Kurniawan, Rawle Francis, et al.. (2019). The anti-parasitic agent suramin and several of its analogues are inhibitors of the DNA binding protein Mcm10. Open Biology. 9(8). 190117–190117. 11 indexed citations

Thompson, Elizabeth L., Yinan Kan, Constanze Wiek, et al.. (2017). FANCI and FANCD2 have common as well as independent functions during the cellular replication stress response. Nucleic Acids Research. 45(20). 11837–11857. 27 indexed citations

Castellà, María, Céline Jacquemont, Elizabeth L. Thompson, et al.. (2015). FANCI Regulates Recruitment of the FA Core Complex at Sites of DNA Damage Independently of FANCD2. PLoS Genetics. 11(10). e1005563–e1005563. 62 indexed citations

Budzowska, Magda, Thomas G.W. Graham, Alexandra Sobeck, S Waga, & Johannes C. Walter. (2015). Regulation of the Rev1–pol ζ complex during bypass of a DNA interstrand cross‐link. The EMBO Journal. 34(14). 1971–1985. 99 indexed citations

Lee, Eu Han, et al.. (2014). CtIP mediates replication fork recovery in a FANCD2-regulated manner. Human Molecular Genetics. 23(14). 3695–3705. 67 indexed citations

10.

Chaudhury, Indrajit, et al.. (2013). FANCD2 regulates BLM complex functions independently of FANCI to promote replication fork recovery. Nucleic Acids Research. 41(13). 6444–6459. 86 indexed citations

11.

Sareen, Archana, et al.. (2012). Fanconi anemia proteins FANCD2 and FANCI exhibit different DNA damage responses during S-phase. Nucleic Acids Research. 40(17). 8425–8439. 46 indexed citations

12.

Waltes, Regina, Reinhard Kalb, Magtouf Gatei, et al.. (2009). Human RAD50 Deficiency in a Nijmegen Breakage Syndrome-like Disorder. The American Journal of Human Genetics. 84(5). 605–616. 189 indexed citations

13.

Sobeck, Alexandra, Stacie Stone, Igor Landais, Bendert de Graaf, & Maureen E. Hoatlin. (2009). The Fanconi Anemia Protein FANCM Is Controlled by FANCD2 and the ATR/ATM Pathways. Journal of Biological Chemistry. 284(38). 25560–25568. 36 indexed citations

14.

Xu, Dongyi, Rong Guo, Alexandra Sobeck, et al.. (2008). RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability. Genes & Development. 22(20). 2843–2855. 165 indexed citations

15.

Stone, Stacie, Alexandra Sobeck, Margriet van Kogelenberg, et al.. (2007). Identification, developmental expression and regulation of the Xenopus ortholog of human FANCG/XRCC9. Genes to Cells. 12(7). 841–851. 6 indexed citations

16.

Sobeck, Alexandra, Stacie Stone, & Maureen E. Hoatlin. (2007). DNA Structure-Induced Recruitment and Activation of the Fanconi Anemia Pathway Protein FANCD2. Molecular and Cellular Biology. 27(12). 4283–4292. 29 indexed citations

17.

Yin, Jinhu, Alexandra Sobeck, Chang Xu, et al.. (2005). BLAP75, an essential component of Bloom's syndrome protein complexes that maintain genome integrity. The EMBO Journal. 24(7). 1465–1476. 151 indexed citations

18.

Sobeck, Alexandra, Stacie Stone, Vincenzo Costanzo, et al.. (2005). Fanconi Anemia Proteins Are Required To Prevent Accumulation of Replication-Associated DNA Double-Strand Breaks. Molecular and Cellular Biology. 26(2). 425–437. 77 indexed citations

19.

Léveillé, France, Eric Blom, Annette L. Medhurst, et al.. (2004). The Fanconi Anemia Gene Product FANCF Is a Flexible Adaptor Protein. Journal of Biological Chemistry. 279(38). 39421–39430. 49 indexed citations

20.

Oppitz, Ulrich, Detlev Schindler, Alexandra Sobeck, et al.. (1999). Sequence analysis of the ATM gene in 20 patients with RTOG grade 3 or 4 acute and/or late tissue radiation side effects. International Journal of Radiation Oncology*Biology*Physics. 44(5). 981–988. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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