Alexander Schulz
- Neurology top 5%
- Cellular and Molecular Neuroscience top 10%
- Molecular Biology
- Epidemiology
- Cell Biology top 10%
- Co-authors
- Helen MorrisonStephan L. BaaderReinhard BauerThomas MindosDario Lucas HelbingVictor‐Felix MautnerAnsgar ZochChristian Hagel
- Topics
- Neurofibromatosis and Schwannoma Cases (13 papers)Meningioma and schwannoma management (6 papers)Hippo pathway signaling and YAP/TAZ (6 papers)
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
Alexander Schulz
27 papers receiving 667 citations
Peers
Comparison fields: 5 of 77
- Neurology 301
- Cellular and Molecular Neuroscience 244
- Molecular Biology 215
- Epidemiology 127
- Cell Biology 111
Countries citing papers authored by Alexander Schulz
This map shows the geographic impact of Alexander Schulz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexander Schulz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexander Schulz more than expected).
Fields of papers citing papers by Alexander Schulz
This network shows the impact of papers produced by Alexander Schulz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexander Schulz. The network helps show where Alexander Schulz may publish in the future.
Co-authorship network of co-authors of Alexander Schulz
This figure shows the co-authorship network connecting the top 25 collaborators of Alexander Schulz. A scholar is included among the top collaborators of Alexander Schulz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexander Schulz. Alexander Schulz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 12 | |
| 4 | 56 | |
| 5 | 34 | |
| 6 | 110 | |
| 7 | 84 | |
| 8 | 8 | |
| 9 | 20 | |
| 10 | The importance of nerve microenvironment for schwannoma development | 1 |
| 11 | 4 | |
| 12 | 8 | |
| 13 | 53 | |
| 14 | 13 | |
| 15 | 41 | |
| 16 | 23 | |
| 17 | 43 | |
| 18 | 4 | |
| 19 | 22 | |
| 20 | 9 |
About Alexander Schulz
Alexander Schulz is a scholar working on Neurology, Cellular and Molecular Neuroscience and Aging, having authored 28 papers that have together received 673 indexed citations. Recurring topics across this work include Neurofibromatosis and Schwannoma Cases (13 papers), Meningioma and schwannoma management (6 papers) and Hippo pathway signaling and YAP/TAZ (6 papers). The work is most often cited by research in Neurology (301 citations), Developmental Neuroscience (64 citations) and Cellular and Molecular Neuroscience (244 citations). Alexander Schulz has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Helen Morrison, Stephan L. Baader, Reinhard Bauer, Thomas Mindos, Dario Lucas Helbing, Victor‐Felix Mautner, Ansgar Zoch, Christian Hagel, Lars Björn Riecken and David B. Parkinson. Their work appears in journals such as Journal of Neuroscience, The Journal of Cell Biology and Nature Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.