Akihiro Tomida

4.4k citations
99 papers · 3.6k · h-index 35

Impact in

Papers in

    • Cancer therapeutics and mechanisms 22
    • Ubiquitin and proteasome pathways 9
    • RNA regulation and disease 7
    • Drug Transport and Resistance Mechanisms 11
    • Cancer Treatment and Pharmacology 10

Akihiro Tomida

97 papers receiving 3.6k citations

Peers

Akihiro Tomida
Comparison fields: 5 of 124
  • Cancer Research 686
  • Cell Biology 676
  • Toxicology 122
  • Oncology 929
  • Molecular Biology 2.3k
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Citations per field
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Citations per year

Countries citing papers authored by Akihiro Tomida

Since Specialization
Citations

This map shows the geographic impact of Akihiro Tomida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Akihiro Tomida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Akihiro Tomida more than expected).

Fields of papers citing papers by Akihiro Tomida

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Akihiro Tomida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Akihiro Tomida. The network helps show where Akihiro Tomida may publish in the future.

Co-authors

The 25 scholars most cited alongside Akihiro Tomida, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Akihiro Tomida Line = papers co-authored together Akihiro Tomida links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 99 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2003434
2 2001183
3 2004180
4 2004178
5 2009137
6
Drug resistance mediated by cellular stress response to the microenvironment of solid tumors.
1999121
7
Proteasome inhibition circumvents solid tumor resistance to topoisomerase II-directed drugs.
2000103
8 199977
9
DT-diaphorase as a critical determinant of sensitivity to mitomycin C in human colon and gastric carcinoma cell lines.
199672
10 200765
11 200965
12 201062
13
Glucose-regulated stresses confer resistance to VP-16 in human cancer cells through a decreased expression of DNA topoisomerase II.
199561
14 201060
15 200760
16 200458
17 199354
18
Increased expression of thioredoxin/adult T-cell leukemia-derived factor in cisplatin-resistant human cancer cell lines.
199654
19 201352
20 200248

About Akihiro Tomida

Akihiro Tomida is a scholar working on Molecular Biology, Oncology, Cell Biology, Cancer Research and Organic Chemistry, having authored 99 papers that have together received 3.6k indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (24 papers), Cancer therapeutics and mechanisms (22 papers), Drug Transport and Resistance Mechanisms (11 papers), Cancer Treatment and Pharmacology (10 papers), Cancer, Hypoxia, and Metabolism (10 papers), Ubiquitin and proteasome pathways (9 papers), Autophagy in Disease and Therapy (8 papers) and RNA regulation and disease (7 papers). The work is most often cited by research in Cancer Research (686 citations), Cell Biology (676 citations), Toxicology (122 citations), Oncology (929 citations) and Molecular Biology (2.3k citations). Akihiro Tomida has collaborated with scholars based in Japan, South Korea and United States. Frequent co-authors include Takashi Tsuruo, T Tsuruo, Mikihiko Naito, Naoya Fujita, Naomi Haga, Tetsuo Mashima, Hiroyuki Suzuki, Hiroshi Sakamoto, Yasunari Ogiso and Kazuo Shin‐ya. Their work appears in journals such as Biochemical and Biophysical Research Communications, Journal of Natural Products, Cancer Science, Journal of Cellular Physiology and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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