Issei Imoto

14.9k citations

251 papers · 10.7k indexed · 1 hit paper · h-index 56

Impact in

Cancer Research top 0.2%
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
Molecular Biology top 0.5%
- Epigenetics and DNA Methylation
- RNA modifications and cancer
- Cancer-related gene regulation
- Circular RNAs in diseases

Papers in

Genetics 95
- Genomic variations and chromosomal abnormalities 51
- Genetics and Neurodevelopmental Disorders 15
Cancer Research 51
- Cancer Genomics and Diagnostics 26

Co-authors: Johji Inazawa Hitoshi Tsuda Ken‐ichi Kozaki Ken Omura Yutaka Shimada Mayuko Furuta Setsuo Hirohashi Atiphan Pimkhaokham
Journals: Cancer Science (21 papers)Oncogene (13 papers)Cancer Research (10 papers)Carcinogenesis (7 papers)Oncotarget (6 papers)
Partner nations: Japan United States United Kingdom

In The Last Decade

Issei Imoto

246 papers receiving 10.6k citations

Hit Papers

align trajectories log scale

Exploration of Tumor-Suppressive MicroRNAs Silenced by DNA Hypermethylation in Oral Cancer 2008 · 512 citations

Peers

Countries citing papers authored by Issei Imoto

Since Specialization

Citations

This map shows the geographic impact of Issei Imoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Issei Imoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Issei Imoto more than expected).

Fields of papers citing papers by Issei Imoto

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Issei Imoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Issei Imoto. The network helps show where Issei Imoto may publish in the future.

Co-authors

The 25 scholars most cited alongside Issei Imoto, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Issei Imoto Line = papers co-authored together Issei Imoto links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	Array comparative genomic hybridization analysis discloses chromosome copy number alterations as indicators of patient outcome in lymph node-negative breast cancer BMC Cancer ·Ryoko Kikuchi-Koike, Kazunori Nagasaka, Hitoshi Tsuda, Yasuyuki Ishii, Masaru Sakamoto, Yoshihiro Kikuchi, Shiho Fukui, Yuko Miyagawa, Haruko Hiraike, Takayuki Kobayashi, Takayuki Kinoshita, Yae Kanai, Tatsuhiro Shibata, Issei Imoto, Johji Inazawa, Osamu Matsubara, Takuya Ayabe	2019	9
2	Genome-wide association study identifies ERBB4 on 2q34 as a novel locus associated with sperm motility in Japanese men Journal of Medical Genetics ·Youichi Sato, Atsushi Tajima, Takehiro Sato, Shiari Nozawa, Miki Yoshiike, Issei Imoto, Aiko Yamauchi, Teruaki Iwamoto	2018	9
3	Three patients with Schaaf–Yang syndrome exhibiting arthrogryposis and endocrinological abnormalities American Journal of Medical Genetics Part A ·Takuji Enya, Nobuhiko Okamoto, Yoshinori Iba, Tomoki Miyazawa, Mitsuru Okada, Shinobu Ida, Takuya Naruto, Issei Imoto, Atsushi Fujita, Noriko Miyake, Naomichi Matsumoto, Keisuke Sugimoto, Tsukasa Takemura	2018	18
4	Monitoring the HER2 copy number status in circulating tumor DNA by droplet digital PCR in patients with gastric cancer Gastric Cancer ·Katsutoshi Shoda, Daisuke Ichikawa, Yuji Fujita, Kiyoshi Masuda, Hidekazu Hiramoto, Junichi Hamada, Tomohiro Arita, Hirotaka Konishi, Shuhei Komatsu, Atsushi Shiozaki, Naoki Kakihara, Kazuma Okamoto, Hiroki Taniguchi, Issei Imoto, Eigo Otsuji	2016	108
5	Aberrant DNA Methylation of Blood in Schizophrenia by Adjusting for Estimated Cellular Proportions NeuroMolecular Medicine ·Makoto Kinoshita, Shusuke Numata, Atsushi Tajima, Kazutaka Ohi, Ryota Hashimoto, Shinji Shimodera, Issei Imoto, Masatoshi Takeda, Tetsuro Ohmori	2014	29
6	Integrative Array-Based Approach Identifies MZB1 as a Frequently Methylated Putative Tumor Suppressor in Hepatocellular Carcinoma Clinical Cancer Research ·Satoshi Matsumura, Issei Imoto, Ken‐ichi Kozaki, Takeshi Matsui, Tomoki Muramatsu, Mayuko Furuta, Shinji Tanaka, Michiie Sakamoto, Shigeki Arii, Johji Inazawa	2012	25
7	Deletion at chromosome 10p11.23-p12.1 defines characteristic phenotypes with marked midface retrusion Journal of Human Genetics ·Nana Okamoto, Shin Hayashi, Ayako Masui, Rika Kosaki, Izumi Oguri, Tomoko Hasegawa, Issei Imoto, Yoshio Makita, Akira Hata, Keiji Moriyama, Johji Inazawa	2012	14
8	The Tumor Suppressive MicroRNA miR-218 Targets the mTOR Component Rictor and Inhibits AKT Phosphorylation in Oral Cancer Cancer Research ·Atsushi Uesugi, Ken‐ichi Kozaki, Tomohiko Tsuruta, Mayuko Furuta, K. Morita, Issei Imoto, Ken Omura, Johji Inazawa	2011	216
9	miR-152 Is a Tumor Suppressor microRNA That Is Silenced by DNA Hypermethylation in Endometrial Cancer Cancer Research ·Tomohiko Tsuruta, Ken‐ichi Kozaki, Atsushi Uesugi, Mayuko Furuta, Akira Hirasawa, Issei Imoto, Nobuyuki Susumu, Daisuke Aoki, Johji Inazawa	2011	185
10	Gene amplification of Myc and its coamplification with ERBB2 and EGFR in gallbladder adenocarcinoma. PubMed ·Akishi Ooi, Shioto Suzuki, Kumiko Nakazawa, Jun Itakura, Issei Imoto, Hiroyuki Nakamura, Yoh Dobashi	2009	26
11	miR-124 and miR-203 are epigenetically silenced tumor-suppressive microRNAs in hepatocellular carcinoma Carcinogenesis ·Mayuko Furuta, Ken-ich Kozaki, Shinji Tanaka, Shigeki Arii, Issei Imoto, Johji Inazawa	2009	490
12	Expression and Gene Amplification of Actinin-4 in Invasive Ductal Carcinoma of the Pancreas Clinical Cancer Research ·Satoru Kikuchi, Kazufumi Honda, Hitoshi Tsuda, Nobuyoshi Hiraoka, Issei Imoto, Tomoo Kosuge, Tomoko Umaki, Kaoru Onozato, Miki Shitashige, Umio Yamaguchi, Masaya Ono, Akihiko Tsuchida, Tatsuya Aoki, Johji Inazawa, Setsuo Hirohashi, Tesshi Yamada	2008	96
13	Exploration of Tumor-Suppressive MicroRNAs Silenced by DNA Hypermethylation in Oral Cancer Cancer Research ·Ken‐ichi Kozaki, Issei Imoto, Seiki Mogi, Ken Omura, Johji Inazawa Hit paper breakdown →	2008	512
14	Frequent Inactivation of a Putative Tumor Suppressor, Angiopoietin-Like Protein 2, in Ovarian Cancer Cancer Research ·Ryoko Kikuchi, Hitoshi Tsuda, Ken‐ichi Kozaki, Yae Kanai, Takahiro Kasamatsu, Kazuo Sengoku, Setsuo Hirohashi, Johji Inazawa, Issei Imoto	2008	48
15	Identification of SMURF1 as a possible target for 7q21.3‐22.1 amplification detected in a pancreatic cancer cell line by in‐house array‐based comparative genomic hybridization Cancer Science ·Ayako Suzuki, Tatsuhiro Shibata, Yutaka Shimada, Yoshinori Murakami, Akira Horii, Keiko Shiratori, Setsuo Hirohashi, Johji Inazawa, Issei Imoto	2008	28
16	Promoter Hypermethylation Contributes to Frequent Inactivation of a Putative Conditional Tumor Suppressor Gene Connective Tissue Growth Factor in Ovarian Cancer Cancer Research ·Ryoko Kikuchi, Hitoshi Tsuda, Yae Kanai, Takahiro Kasamatsu, Kazuo Sengoku, Setsuo Hirohashi, Johji Inazawa, Issei Imoto	2007	68
17	Multiplex PCR/Liquid Chromatography Assay for Screening of Subtelomeric Rearrangements Genetic Testing ·Toru Udaka, Issei Imoto, Yoshinori Aizu, Chiharu Torii, Kosuke Izumi, Rika Kosaki, Takao Takahashi, Shin Hayashi, Johji Inazawa, Kenjiro Kosaki	2007	3
18	Methylation-Associated Silencing of the Nuclear Receptor 1I2 Gene in Advanced-Type Neuroblastomas, Identified by Bacterial Artificial Chromosome Array-Based Methylated CpG Island Amplification Cancer Research ·Akiko Misawa, Jun Inoue, Yuriko Sugino, Hajime Hosoi, Tohru Sugimoto, Fumie Hosoda, Misao Ohki, Issei Imoto, Johji Inazawa	2005	54
19	Frequent silencing of DBC1 is by genetic or epigenetic mechanisms in non-small cell lung cancers Human Molecular Genetics ·Hiroyuki Izumi, Jun Inoue, Sana Yokoi, Hiroshi Hosoda, Tatsuhiro Shibata, M Sunamori, Setsuo Hirohashi, Johji Inazawa, Issei Imoto	2005	52
20	Overexpression of PDZK1 within the 1q12-q22 Amplicon Is Likely To Be Associated with Drug-Resistance Phenotype in Multiple Myeloma American Journal Of Pathology ·Jun Inoue, Takemi Otsuki, Akira Hirasawa, Issei Imoto, Yoshinobu Matsuo, Shiroh Shimizu, Masafumi Taniwaki, Johji Inazawa	2004	78

About Issei Imoto

Issei Imoto is a scholar working on Genetics, Cancer Research, Molecular Biology, Reproductive Medicine and Oncology, having authored 251 papers that have together received 10.7k indexed citations. Recurring topics across this work include Genomic variations and chromosomal abnormalities (51 papers), Epigenetics and DNA Methylation (40 papers), Cancer-related gene regulation (30 papers), RNA modifications and cancer (28 papers), Cancer Genomics and Diagnostics (26 papers), Chromosomal and Genetic Variations (15 papers), Genetics and Neurodevelopmental Disorders (15 papers) and Genetic factors in colorectal cancer (12 papers). The work is most often cited by research in Cancer Research (3.2k citations), Molecular Biology (7.3k citations), Oncology (2.1k citations), Genetics (1.6k citations) and Cell Biology (912 citations). Issei Imoto has collaborated with scholars based in Japan, United States and United Kingdom. Frequent co-authors include Johji Inazawa, Hitoshi Tsuda, Ken‐ichi Kozaki, Ken Omura, Yutaka Shimada, Mayuko Furuta, Setsuo Hirohashi, Atiphan Pimkhaokham, Shigeki Arii and Seiki Mogi. Their work appears in journals such as Cancer Science, Oncogene, Cancer Research, Carcinogenesis and Oncotarget.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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