Aditya J. Desai
- Molecular Biology
- Cellular and Molecular Neuroscience top 10%
- Physiology top 5%
- Endocrine and Autonomic Systems
- Endocrinology, Diabetes and Metabolism
- Co-authors
- Laurence J. MillerMaoqing DongKaleeckal G. HarikumarPaula FernándezMichael H. PillingerJiang‐Fan ChenBruce N. CronsteinAllison B. Reiss
- Topics
- Neuropeptides and Animal Physiology (15 papers)Receptor Mechanisms and Signaling (12 papers)Diabetes Treatment and Management (3 papers)
- Partner nations
- United StatesAustraliaUnited Kingdom
In The Last Decade
Aditya J. Desai
16 papers receiving 340 citations
Peers
Comparison fields: 5 of 78
- Molecular Biology 168
- Cellular and Molecular Neuroscience 137
- Physiology 55
- Endocrine and Autonomic Systems 47
- Endocrinology, Diabetes and Metabolism 41
Countries citing papers authored by Aditya J. Desai
This map shows the geographic impact of Aditya J. Desai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aditya J. Desai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aditya J. Desai more than expected).
Fields of papers citing papers by Aditya J. Desai
This network shows the impact of papers produced by Aditya J. Desai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aditya J. Desai. The network helps show where Aditya J. Desai may publish in the future.
Co-authorship network of co-authors of Aditya J. Desai
This figure shows the co-authorship network connecting the top 25 collaborators of Aditya J. Desai. A scholar is included among the top collaborators of Aditya J. Desai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aditya J. Desai. Aditya J. Desai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 5 | |
| 3 | 7 | |
| 4 | 5 | |
| 5 | 17 | |
| 6 | 45 | |
| 7 | 22 | |
| 8 | 16 | |
| 9 | 30 | |
| 10 | 10 | |
| 11 | 11 | |
| 12 | 14 | |
| 13 | 20 | |
| 14 | 17 | |
| 15 | 19 | |
| 16 | 107 |
About Aditya J. Desai
Aditya J. Desai is a scholar working on Cellular and Molecular Neuroscience, Endocrine and Autonomic Systems and Physiology, having authored 16 papers that have together received 346 indexed citations. Recurring topics across this work include Neuropeptides and Animal Physiology (15 papers), Receptor Mechanisms and Signaling (12 papers) and Diabetes Treatment and Management (3 papers). The work is most often cited by research in Physiology (55 citations), Cellular and Molecular Neuroscience (137 citations) and Endocrine and Autonomic Systems (47 citations). Aditya J. Desai has collaborated with scholars based in United States, Australia and United Kingdom. Frequent co-authors include Laurence J. Miller, Maoqing Dong, Kaleeckal G. Harikumar, Paula Fernández, Michael H. Pillinger, Jiang‐Fan Chen, Bruce N. Cronstein, Allison B. Reiss, Marjana Tomic‐Canic and M. Carmen Montesinos. Their work appears in journals such as Journal of Biological Chemistry, PLoS ONE and American Journal of Clinical Nutrition.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.