Ad J. Dekker

471 citations
13 papers · 405 indexed · h-index 12
Topics
Nerve injury and regeneration (12 papers)Neurogenesis and neuroplasticity mechanisms (5 papers)Neuropeptides and Animal Physiology (4 papers)
Partner nations
United StatesNetherlands

In The Last Decade

Ad J. Dekker

13 papers receiving 382 citations

Peers

Ad J. Dekker
Comparison fields: 5 of 53
  • Cellular and Molecular Neuroscience 309
  • Molecular Biology 139
  • Developmental Neuroscience 134
  • Pharmacology 77
  • Physiology 66
Replace Shwun‐De Wang with:
Shwun‐De Wang Taiwan
F. Javier Milán Italy
Sharon M. Edmunds United States
Michael D. Applegate United States
Humberto Martínez United States
Marco Ledri Sweden
Mikael Ängehagen Sweden
Elliot J. Mufson United States
Brigitte Raynaud France
H. Böttcher Germany
Ad J. Dekker relative to Shwun‐De Wang Taiwan Shwun‐De Wang's profile →
Citations per field
00.5×1.5×2.2×
Shwun‐De Wang · 1×
Citations per year

Countries citing papers authored by Ad J. Dekker

Since Specialization
Citations

This map shows the geographic impact of Ad J. Dekker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ad J. Dekker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ad J. Dekker more than expected).

Fields of papers citing papers by Ad J. Dekker

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ad J. Dekker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ad J. Dekker. The network helps show where Ad J. Dekker may publish in the future.

Co-authorship network of co-authors of Ad J. Dekker

This figure shows the co-authorship network connecting the top 25 collaborators of Ad J. Dekker. A scholar is included among the top collaborators of Ad J. Dekker based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ad J. Dekker. Ad J. Dekker is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
#WorkIndexed citations
1 20
2 46
3 4
4 43
5 17
6 67
7 38
8 43
9 42
10 11
11 14
12 25
13 35

About Ad J. Dekker

Ad J. Dekker is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Neurology, having authored 13 papers that have together received 405 indexed citations. Recurring topics across this work include Nerve injury and regeneration (12 papers), Neurogenesis and neuroplasticity mechanisms (5 papers) and Neuropeptides and Animal Physiology (4 papers). The work is most often cited by research in Developmental Neuroscience (134 citations), Cellular and Molecular Neuroscience (309 citations) and Pharmacology (77 citations). Ad J. Dekker has collaborated with scholars based in United States and Netherlands. Frequent co-authors include Fred H. Gage, Leon J. Thal, L. J. Thal, Jürgen Winkler, J Ray, Willem Hendrik Gispen, D. de Wied, Paul M. Salvaterra, C.L.E. Broekkamp and Leo G. J. de Leede. Their work appears in journals such as Brain Research, Neuroscience and Journal of Neurochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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