Zhigao Wang
- Molecular Biology top 1%
- Immunology top 2%
- Cancer Research top 2%
- Epidemiology top 5%
- Cell Biology top 2%
- Topics
- Cell death mechanisms and regulation (10 papers)Mitochondrial Function and Pathology (4 papers)Signaling Pathways in Disease (4 papers)
- Partner nations
- United StatesChinaHong Kong
In The Last Decade
Zhigao Wang
26 papers receiving 5.7k citations
Hit Papers
Peers
Comparison fields: 5 of 115
- Molecular Biology 4.6k
- Immunology 1.3k
- Cancer Research 774
- Epidemiology 721
- Cell Biology 551
Countries citing papers authored by Zhigao Wang
This map shows the geographic impact of Zhigao Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Zhigao Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Zhigao Wang more than expected).
Fields of papers citing papers by Zhigao Wang
This network shows the impact of papers produced by Zhigao Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Zhigao Wang. The network helps show where Zhigao Wang may publish in the future.
Co-authorship network of co-authors of Zhigao Wang
This figure shows the co-authorship network connecting the top 25 collaborators of Zhigao Wang. A scholar is included among the top collaborators of Zhigao Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Zhigao Wang. Zhigao Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 2 | |
| 4 | 40 | |
| 5 | 21 | |
| 6 | Mitochondrial Phosphatase PGAM5 modulates cellular senescence by regulatingmitochondrial dynamics | 5 |
| 7 | 174 | |
| 8 | 37 | |
| 9 | 46 | |
| 10 | 4 | |
| 11 | 2 | |
| 12 | 36 | |
| 13 | 0 | |
| 14 | The Mitochondrial Phosphatase PGAM5 Functions at the Convergence Point of Multiple Necrotic Death Pathwaysbreakdown → | 776 |
| 15 | Mixed Lineage Kinase Domain-like Protein Mediates Necrosis Signaling Downstream of RIP3 Kinasebreakdown → | 2094 |
| 16 | 163 | |
| 17 | 44 | |
| 18 | 467 | |
| 19 | 395 | |
| 20 | Activation of Cardiac Gene Expression by Myocardin, a Transcriptional Cofactor for Serum Response Factorbreakdown → | 726 |
About Zhigao Wang
Zhigao Wang is a scholar working on Aging, Cell Biology and Molecular Biology, having authored 28 papers that have together received 5.7k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (10 papers), Mitochondrial Function and Pathology (4 papers) and Signaling Pathways in Disease (4 papers). The work is most often cited by research in Molecular Biology (4.6k citations), Immunology (1.3k citations) and Cancer Research (774 citations). Zhigao Wang has collaborated with scholars based in United States, China and Hong Kong. Frequent co-authors include Eric N. Olson, She Chen, Xiaodong Wang, Da‐Zhi Wang, Huayi Wang, Liming Sun, Sudan He, Jiacong Yan, Weilong Liu and Xiaoguang Lei. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.