Yuta Kochi

10.1k total citations
67 papers, 2.6k citations indexed

About

Yuta Kochi is a scholar working on Immunology, Rheumatology and Molecular Biology. According to data from OpenAlex, Yuta Kochi has authored 67 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Immunology, 32 papers in Rheumatology and 18 papers in Molecular Biology. Recurrent topics in Yuta Kochi's work include T-cell and B-cell Immunology (23 papers), Systemic Lupus Erythematosus Research (22 papers) and Rheumatoid Arthritis Research and Therapies (20 papers). Yuta Kochi is often cited by papers focused on T-cell and B-cell Immunology (23 papers), Systemic Lupus Erythematosus Research (22 papers) and Rheumatoid Arthritis Research and Therapies (20 papers). Yuta Kochi collaborates with scholars based in Japan, United States and Italy. Yuta Kochi's co-authors include Kazuhiko Yamamoto, Akari Suzuki, Ryo Yamada, Yukinori Okada, Tetsuji Sawada, Keishi Fujio, Tatsuhiko Tsunoda, Xiaotian Chang, Atsushi Takahashi and Yusuke Nakamura and has published in prestigious journals such as Journal of Clinical Investigation, Nature Communications and Nature Genetics.

In The Last Decade

Yuta Kochi

64 papers receiving 2.6k citations

Peers

Yuta Kochi
Marieke J. H. Coenen Netherlands
Elizabeth C. Jury United Kingdom
Linda K. Myers United States
N. Miyasaka Japan
Robert W. McMurray United States
Swapan K. Nath United States
Alla Skapenko Germany
Jan Piet van Hamburg Netherlands
Tong Zhou United States
Kendall M. Mohler United States
Marieke J. H. Coenen Netherlands
Yuta Kochi
Citations per year, relative to Yuta Kochi Yuta Kochi (= 1×) peers Marieke J. H. Coenen

Countries citing papers authored by Yuta Kochi

Since Specialization
Citations

This map shows the geographic impact of Yuta Kochi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuta Kochi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuta Kochi more than expected).

Fields of papers citing papers by Yuta Kochi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuta Kochi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuta Kochi. The network helps show where Yuta Kochi may publish in the future.

Co-authorship network of co-authors of Yuta Kochi

This figure shows the co-authorship network connecting the top 25 collaborators of Yuta Kochi. A scholar is included among the top collaborators of Yuta Kochi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuta Kochi. Yuta Kochi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Xiaoxi, Hiroyuki Suetsugu, Takeshi Iwasaki, et al.. (2024). Attenuation of HOIL-1L ligase activity promotes systemic autoimmune disorders by augmenting linear ubiquitin signaling. JCI Insight. 9(3).
2.
Inamo, Jun, Akari Suzuki, Mahoko Takahashi Ueda, et al.. (2024). Long-read sequencing for 29 immune cell subsets reveals disease-linked isoforms. Nature Communications. 15(1). 4285–4285. 10 indexed citations
3.
4.
Torii, Satoru, et al.. (2023). Uncovering the Localization and Function of a Novel Read-Through Transcript ‘TOMM40-APOE’. Cells. 13(1). 69–69. 2 indexed citations
5.
6.
Takeshima, Yusuke, Yukiko Iwasaki, M Nakano, et al.. (2022). Immune cell multiomics analysis reveals contribution of oxidative phosphorylation to B-cell functions and organ damage of lupus. Annals of the Rheumatic Diseases. 81(6). 845–853. 33 indexed citations
7.
Tsuji, Hideaki, Koichiro Ohmura, Hui Jin, et al.. (2021). Anti–Double‐Stranded DNA Antibodies Recognize DNA Presented on HLA Class II Molecules of Systemic Lupus Erythematosus Risk Alleles. Arthritis & Rheumatology. 74(1). 105–111. 7 indexed citations
8.
Singh, Bhupinder, Guru Prasad Maiti, Shu‐Feng Zhou, et al.. (2021). Lupus Susceptibility Region Containing CDKN1B rs34330 Mechanistically Influences Expression and Function of Multiple Target Genes, Also Linked to Proliferation and Apoptosis. Arthritis & Rheumatology. 73(12). 2303–2313. 16 indexed citations
9.
Kochi, Yuta, et al.. (2019). Identification of rare coding variants in TYK2 protective for rheumatoid arthritis in the Japanese population and their effects on cytokine signalling. Annals of the Rheumatic Diseases. 78(8). 1062–1069. 17 indexed citations
10.
Akizuki, Shuji, Kazuyoshi Ishigaki, Yuta Kochi, et al.. (2019). PLD4 is a genetic determinant to systemic lupus erythematosus and involved in murine autoimmune phenotypes. Annals of the Rheumatic Diseases. 78(4). 509–518. 35 indexed citations
11.
Sumitomo, Shuji, Yasuo Nagafuchi, Yumi Tsuchida, et al.. (2018). Transcriptome analysis of peripheral blood from patients with rheumatoid arthritis: a systematic review. Inflammation and Regeneration. 38(1). 21–21. 27 indexed citations
12.
Nishino, Jo, Hidenori Ochi, Yuta Kochi, Tatsuhiko Tsunoda, & Shigeyuki Matsui. (2018). Sample Size for Successful Genome-Wide Association Study of Major Depressive Disorder. Frontiers in Genetics. 9. 227–227. 27 indexed citations
13.
Ishigaki, Kazuyoshi, Yuta Kochi, & Kazuhiko Yamamoto. (2017). Genetics of human autoimmunity: From genetic information to functional insights. Clinical Immunology. 186. 9–13. 6 indexed citations
14.
Shimane, Kenichi, Yuta Kochi, Akari Suzuki, et al.. (2013). An association analysis of HLA-DRB1 with systemic lupus erythematosus and rheumatoid arthritis in a Japanese population: effects of *09:01 allele on disease phenotypes. Lara D. Veeken. 52(7). 1172–1182. 56 indexed citations
15.
Myouzen, Keiko, Yuta Kochi, Yukinori Okada, et al.. (2012). Functional Variants in NFKBIE and RTKN2 Involved in Activation of the NF-κB Pathway Are Associated with Rheumatoid Arthritis in Japanese. PLoS Genetics. 8(9). e1002949–e1002949. 45 indexed citations
16.
Kochi, Yuta, Mohamed M. Thabet, Akari Suzuki, et al.. (2010). PADI4 polymorphism predisposes male smokers to rheumatoid arthritis. Annals of the Rheumatic Diseases. 70(3). 512–515. 46 indexed citations
17.
Kochi, Yuta, Keiko Myouzen, Ryo Yamada, et al.. (2009). FCRL3, an Autoimmune Susceptibility Gene, Has Inhibitory Potential on B-Cell Receptor-Mediated Signaling. The Journal of Immunology. 183(9). 5502–5510. 76 indexed citations
18.
Oishi, Tetsuya, Aritoshi Iida, Shigeru Otsubo, et al.. (2008). A functional SNP in the NKX2.5-binding site of ITPR3 promoter is associated with susceptibility to systemic lupus erythematosus in Japanese population. Journal of Human Genetics. 53(2). 151–162. 25 indexed citations
19.
Shimane, Kenichi, Yuta Kochi, Ryo Yamada, et al.. (2008). A single nucleotide polymorphism in the IRF5 promoter region is associated with susceptibility to rheumatoid arthritis in the Japanese population. Annals of the Rheumatic Diseases. 68(3). 377–383. 36 indexed citations
20.
Shoda, Hirofumi, Keishi Fujio, Yumi Yamaguchi, et al.. (2006). Interactions between IL-32 and tumor necrosis factor alpha contribute to the exacerbation of immune-inflammatory diseases. Arthritis Research & Therapy. 8(6). R166–R166. 117 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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