Yi‐Hsin Weng

1.5k total citations
33 papers, 1.0k citations indexed

About

Yi‐Hsin Weng is a scholar working on Neurology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Yi‐Hsin Weng has authored 33 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Neurology, 15 papers in Cellular and Molecular Neuroscience and 11 papers in Neurology. Recurrent topics in Yi‐Hsin Weng's work include Parkinson's Disease Mechanisms and Treatments (21 papers), Neurological diseases and metabolism (9 papers) and Genetic Neurodegenerative Diseases (7 papers). Yi‐Hsin Weng is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (21 papers), Neurological diseases and metabolism (9 papers) and Genetic Neurodegenerative Diseases (7 papers). Yi‐Hsin Weng collaborates with scholars based in Taiwan, Netherlands and United Kingdom. Yi‐Hsin Weng's co-authors include Tu‐Hsueh Yeh, Chin-Song Lu, Hung‐Li Wang, An-Hsun Chou, Rou-Shayn Chen, Yah‐Huei Wu‐Chou, Hsiu‐Chen Chang, Szu‐Chia Lai, Vincenzo Bonifati and Ying‐Ling Chen and has published in prestigious journals such as The Journal of Physiology, International Journal of Molecular Sciences and Biochemical Pharmacology.

In The Last Decade

Yi‐Hsin Weng

32 papers receiving 987 citations

Peers

Yi‐Hsin Weng
Szu‐Chia Lai Taiwan
Ruth E. Musgrove Germany
Amanda Singleton United States
Rou-Shayn Chen Taiwan
Kenichi Yasui Japan
Christina Ulane United States
Yaping Yan China
Eva Schaeffer Germany
G M Szpak Poland
Pavanni Ratnagopal Singapore
Szu‐Chia Lai Taiwan
Yi‐Hsin Weng
Citations per year, relative to Yi‐Hsin Weng Yi‐Hsin Weng (= 1×) peers Szu‐Chia Lai

Countries citing papers authored by Yi‐Hsin Weng

Since Specialization
Citations

This map shows the geographic impact of Yi‐Hsin Weng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yi‐Hsin Weng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yi‐Hsin Weng more than expected).

Fields of papers citing papers by Yi‐Hsin Weng

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yi‐Hsin Weng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yi‐Hsin Weng. The network helps show where Yi‐Hsin Weng may publish in the future.

Co-authorship network of co-authors of Yi‐Hsin Weng

This figure shows the co-authorship network connecting the top 25 collaborators of Yi‐Hsin Weng. A scholar is included among the top collaborators of Yi‐Hsin Weng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yi‐Hsin Weng. Yi‐Hsin Weng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Hung‐Li, Ying‐Zu Huang, Yi‐Hsin Weng, et al.. (2020). Alda-1, an activator of ALDH2, ameliorates Achilles tendinopathy in cellular and mouse models. Biochemical Pharmacology. 175. 113919–113919. 23 indexed citations
2.
Chiu, Ching‐Chi, Hung‐Li Wang, Yi‐Hsin Weng, et al.. (2019). Generation of induced pluripotent stem cells from a young-onset Parkinson's disease patient carrying the compound heterozygous PLA2G6 p.D331Y/p.M358IfsX mutations. Stem Cell Research. 40. 101552–101552. 3 indexed citations
3.
4.
Weng, Yi‐Hsin, Chuyu Chen, Ying‐Ling Chen, et al.. (2015). (R1441C) LRRK2 induces the degeneration of SN dopaminergic neurons and alters the expression of genes regulating neuronal survival in a transgenic mouse model. Experimental Neurology. 275. 104–115. 26 indexed citations
5.
Chen, Chuyu, Ying‐Ling Chen, Yi‐Hsin Weng, et al.. (2014). (G2019S) LRRK2 causes early-phase dysfunction of SNpc dopaminergic neurons and impairment of corticostriatal long-term depression in the PD transgenic mouse. Neurobiology of Disease. 68. 190–199. 48 indexed citations
6.
Wang, Hung‐Li, et al.. (2013). XLID CUL4B mutants are defective in promoting TSC2 degradation and positively regulating mTOR signaling in neocortical neurons. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(4). 585–593. 15 indexed citations
7.
Lin, Kun‐Ju, et al.. (2013). Brain fluorodeoxyglucose positron emission tomography (18FDG PET) in patients with acute thallium intoxication. Clinical Toxicology. 51(3). 167–173. 2 indexed citations
8.
9.
Weng, Yi‐Hsin, Ing‐Tsung Hsiao, Chia‐Ju Hsieh, et al.. (2012). Quantitative analysis of binding sites for 9‐fluoropropyl‐(+)‐dihydrotetrabenazine ([18F]AV‐133) in a MPTP‐lesioned PD mouse model. Synapse. 66(9). 823–831. 11 indexed citations
10.
Wu‐Chou, Yah‐Huei, Ying‐Ting Chen, Tu‐Hsueh Yeh, et al.. (2012). Genetic variants of SNCA and LRRK2 genes are associated with sporadic PD susceptibility: A replication study in a Taiwanese cohort. Parkinsonism & Related Disorders. 19(2). 251–255. 34 indexed citations
11.
Wang, Hung‐Li, et al.. (2011). PARK6 PINK1 mutants are defective in maintaining mitochondrial membrane potential and inhibiting ROS formation of substantia nigra dopaminergic neurons. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1812(6). 674–684. 103 indexed citations
12.
Lu, Chin‐Song, Szu‐Chia Lai, Ruey‐Meei Wu, et al.. (2011). PLA2G6 mutations in PARK14‐linked young‐onset parkinsonism and sporadic Parkinson's disease. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 159B(2). 183–191. 62 indexed citations
13.
Wu‐Chou, Yah‐Huei, Tu‐Hsueh Yeh, Chuanyu Wang, et al.. (2010). High frequency of multiexonic deletion of the GCH1 gene in a Taiwanese cohort of dopa‐response dystonia. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 153B(4). 903–908. 15 indexed citations
14.
Weng, Yi‐Hsin, et al.. (2010). Impact of a nationwide outreach program on the diffusion of evidence-based practice in Taiwan. International Journal for Quality in Health Care. 22(5). 430–436. 19 indexed citations
15.
Chou, An-Hsun, et al.. (2010). HDAC inhibitor sodium butyrate reverses transcriptional downregulation and ameliorates ataxic symptoms in a transgenic mouse model of SCA3. Neurobiology of Disease. 41(2). 481–488. 83 indexed citations
16.
Tan, Eng‐King, Chin-Song Lu, Rong Peng, et al.. (2009). Analysis of the UCHL1 genetic variant in Parkinson's disease among Chinese. Neurobiology of Aging. 31(12). 2194–2196. 14 indexed citations
17.
Lu, Chin-Song, Yah‐Huei Wu‐Chou, Marina van Doeselaar, et al.. (2008). The LRRK2 Arg1628Pro variant is a risk factor for Parkinson’s disease in the Chinese population. Neurogenetics. 9(4). 271–276. 50 indexed citations
18.
Lu, Chin‐Song, et al.. (2008). Analysis of the LRRK2 Gly2385Arg variant in primary dystonia and multiple system atrophy in Taiwan. Parkinsonism & Related Disorders. 14(5). 393–396. 8 indexed citations
19.
Fonzo, Alessio Di, Yah‐Huei Wu‐Chou, Chin-Song Lu, et al.. (2006). A common missense variant in the LRRK2 gene, Gly2385Arg, associated with Parkinson’s disease risk in Taiwan. Neurogenetics. 7(3). 133–138. 180 indexed citations
20.
Tsai, Chon‐Haw, Fang‐Chia Chang, Chin‐Song Lu, et al.. (2004). Pallidotomy effect on the cortical excitability in patients with severe Parkinson's disease. Movement Disorders. 20(4). 463–470. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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