Yanran Liang

909 total citations
30 papers, 695 citations indexed

About

Yanran Liang is a scholar working on Molecular Biology, Neurology and Epidemiology. According to data from OpenAlex, Yanran Liang has authored 30 papers receiving a total of 695 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 11 papers in Neurology and 9 papers in Epidemiology. Recurrent topics in Yanran Liang's work include Parkinson's Disease Mechanisms and Treatments (11 papers), Neuroinflammation and Neurodegeneration Mechanisms (8 papers) and Autophagy in Disease and Therapy (7 papers). Yanran Liang is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (11 papers), Neuroinflammation and Neurodegeneration Mechanisms (8 papers) and Autophagy in Disease and Therapy (7 papers). Yanran Liang collaborates with scholars based in China, United States and Hong Kong. Yanran Liang's co-authors include Enxiang Tao, Xiuna Jing, Wei Bi, Danyu Lin, Li Zhu, Tianen Zhou, Qiaoyun Shi, Lianhong Yang, Ying Chen and Songhua Xiao and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Hepatology.

In The Last Decade

Yanran Liang

29 papers receiving 691 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yanran Liang China 17 311 184 167 139 106 30 695
Xiuna Jing China 15 281 0.9× 155 0.8× 144 0.9× 185 1.3× 80 0.8× 24 693
Hanuma Kumar Karnati United States 11 374 1.2× 144 0.8× 228 1.4× 111 0.8× 83 0.8× 11 778
Ting Sun China 11 351 1.1× 156 0.8× 89 0.5× 73 0.5× 126 1.2× 31 635
Jenna L Leclerc United States 14 294 0.9× 163 0.9× 290 1.7× 74 0.5× 83 0.8× 22 731
Yinyi Sun China 10 228 0.7× 215 1.2× 106 0.6× 86 0.6× 300 2.8× 12 638
Weijin Liu China 11 339 1.1× 56 0.3× 206 1.2× 116 0.8× 191 1.8× 21 684
Ana Lukić United States 17 326 1.0× 114 0.6× 121 0.7× 263 1.9× 45 0.4× 27 762
Fengyuan Che China 13 188 0.6× 174 0.9× 243 1.5× 84 0.6× 98 0.9× 49 685
Cheng‐Liang Luo China 13 562 1.8× 102 0.6× 219 1.3× 89 0.6× 255 2.4× 20 900
Yifeng Du China 15 212 0.7× 78 0.4× 149 0.9× 72 0.5× 61 0.6× 36 653

Countries citing papers authored by Yanran Liang

Since Specialization
Citations

This map shows the geographic impact of Yanran Liang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yanran Liang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yanran Liang more than expected).

Fields of papers citing papers by Yanran Liang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yanran Liang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yanran Liang. The network helps show where Yanran Liang may publish in the future.

Co-authorship network of co-authors of Yanran Liang

This figure shows the co-authorship network connecting the top 25 collaborators of Yanran Liang. A scholar is included among the top collaborators of Yanran Liang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yanran Liang. Yanran Liang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Li, Jinlian, et al.. (2024). CACIMAR: cross-species analysis of cell identities, markers, regulations, and interactions using single-cell RNA sequencing data. Briefings in Bioinformatics. 25(4). 1 indexed citations
2.
3.
Liang, Yanran, Jiongliang Wang, Guangqi Zhou, et al.. (2024). Lhx2 specifically expressed in HSCs promotes liver regeneration and inhibits liver fibrosis. Hepatology. 82(3). 683–701. 5 indexed citations
4.
Liang, H., Danyu Lin, Ying Chen, et al.. (2023). Dl-3-n-butylphthalide activates Nrf2, inhibits ferritinophagy, and protects MES23.5 dopaminergic neurons from ferroptosis. Chemico-Biological Interactions. 382. 110604–110604. 16 indexed citations
5.
Zhou, Tianen, Meng Zhang, Yingyu Xie, et al.. (2021). Effects of miRNAs in exosomes derived from α-synuclein overexpressing SH-SY5Y cells on autophagy and inflammation of microglia. Cellular Signalling. 89. 110179–110179. 4 indexed citations
6.
Bu, Lu-Lu, Yingyu Xie, Danyu Lin, et al.. (2020). LncRNA-T199678 Mitigates α-Synuclein-Induced Dopaminergic Neuron Injury via miR-101-3p. Frontiers in Aging Neuroscience. 12. 599246–599246. 30 indexed citations
7.
Zhu, Shuzhen, Hualing Li, Bin Deng, et al.. (2020). Various Diseases and Clinical Heterogeneity Are Associated With “Hot Cross Bun”. Frontiers in Aging Neuroscience. 12. 592212–592212. 20 indexed citations
8.
Liang, Yanran, Danyu Lin, Xiuna Jing, et al.. (2019). Rifampicin attenuates rotenone-treated microglia inflammation via improving lysosomal function. Toxicology in Vitro. 63. 104690–104690. 26 indexed citations
9.
Lin, Danyu, Yanran Liang, Xiuna Jing, et al.. (2019). α-Synuclein induced mitochondrial dysfunction via cytochrome c oxidase subunit 2 in SH-SY5Y cells. Experimental Cell Research. 378(1). 57–65. 18 indexed citations
10.
Liang, Yanran, et al.. (2018). Chronic back pain cured by low-dose levodopa: is it a variant of restless legs syndrome?. Journal of Pain Research. Volume 11. 277–279. 6 indexed citations
11.
Wu, Xia, Yanran Liang, Xiuna Jing, et al.. (2018). Rifampicin Prevents SH-SY5Y Cells from Rotenone-Induced Apoptosis via the PI3K/Akt/GSK-3β/CREB Signaling Pathway. Neurochemical Research. 43(4). 886–893. 44 indexed citations
12.
Liang, Yanran, Tianen Zhou, Ying Chen, et al.. (2017). Rifampicin inhibits rotenone-induced microglial inflammation via enhancement of autophagy. NeuroToxicology. 63. 137–145. 47 indexed citations
13.
14.
Liang, Yanran, Xiuna Jing, Wei Bi, et al.. (2015). Rifampicin attenuates rotenone-induced inflammation via suppressing NLRP3 inflammasome activation in microglia. Brain Research. 1622. 43–50. 35 indexed citations
15.
Bi, Wei, Li Zhu, Xiuna Jing, et al.. (2014). Investigations into the Role of 26S Proteasome Non-ATPase Regulatory Subunit 13 in Neuroinflammation. NeuroImmunoModulation. 21(6). 331–337. 13 indexed citations
16.
Yang, Lianhong, et al.. (2014). Evaluating team-based, lecture-based, and hybrid learning methods for neurology clerkship in China: a method-comparison study. BMC Medical Education. 14(1). 98–98. 36 indexed citations
17.
Jing, Xiuna, Qiaoyun Shi, Wei Bi, et al.. (2014). Rifampicin Protects PC12 Cells from Rotenone-Induced Cytotoxicity by Activating GRP78 via PERK-eIF2α-ATF4 Pathway. PLoS ONE. 9(3). e92110–e92110. 30 indexed citations
18.
Liu, Yunyun, Zhonglin Liu, Yong Wang, et al.. (2012). Investigation of the performance of PEG–PEI/ROCK-II-siRNA complexes for Alzheimer's disease in vitro. Brain Research. 1490. 43–51. 40 indexed citations
19.
Bi, Wei, Xiuna Jing, Li Zhu, et al.. (2012). Inhibition of 26S Protease Regulatory Subunit 7 (MSS1) Suppresses Neuroinflammation. PLoS ONE. 7(5). e36142–e36142. 11 indexed citations
20.
Bi, Wei, Li Zhu, Yanran Liang, et al.. (2011). Rifampicin inhibits microglial inflammation and improves neuron survival against inflammation. Brain Research. 1395. 12–20. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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