Wei Bi

2.2k total citations · 1 hit paper
51 papers, 1.7k citations indexed

About

Wei Bi is a scholar working on Neurology, Molecular Biology and Immunology. According to data from OpenAlex, Wei Bi has authored 51 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Neurology, 13 papers in Molecular Biology and 10 papers in Immunology. Recurrent topics in Wei Bi's work include Neuroinflammation and Neurodegeneration Mechanisms (16 papers), Immune Response and Inflammation (8 papers) and Neurological Disease Mechanisms and Treatments (6 papers). Wei Bi is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (16 papers), Immune Response and Inflammation (8 papers) and Neurological Disease Mechanisms and Treatments (6 papers). Wei Bi collaborates with scholars based in China, United States and Hong Kong. Wei Bi's co-authors include Li Zhu, Jiawei Zhang, Daxiang Lu, Shu Xiao, Jiayi Zhao, Xiaofeng Cheng, Xin Lan, Yanping Wang, Hongmei Li and Wei Wei and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Advanced Functional Materials.

In The Last Decade

Wei Bi

50 papers receiving 1.7k citations

Hit Papers

Neuroinflammation induced by lipopolysaccharide causes co... 2019 2026 2021 2023 2019 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei Bi China 22 640 593 300 244 181 51 1.7k
Maheedhar Kodali United States 27 861 1.3× 433 0.7× 319 1.1× 114 0.5× 115 0.6× 49 2.1k
Govindhasamy Pushpavathi Selvakumar United States 26 642 1.0× 550 0.9× 418 1.4× 246 1.0× 285 1.6× 45 2.0k
Sangho Choi South Korea 21 650 1.0× 549 0.9× 329 1.1× 177 0.7× 268 1.5× 90 1.9k
Marco Ávila-Rodriguez Colombia 27 640 1.0× 352 0.6× 483 1.6× 171 0.7× 99 0.5× 60 1.8k
Elisa Navarro Spain 17 944 1.5× 686 1.2× 343 1.1× 324 1.3× 196 1.1× 33 2.1k
Weiming Luo United States 21 631 1.0× 585 1.0× 367 1.2× 179 0.7× 172 1.0× 49 1.8k
Xiu‐Qi Bao China 25 909 1.4× 345 0.6× 302 1.0× 129 0.5× 89 0.5× 92 2.0k
Yong Tang China 30 1.2k 1.9× 352 0.6× 444 1.5× 171 0.7× 157 0.9× 94 2.6k
Yajin Liao China 20 790 1.2× 430 0.7× 252 0.8× 245 1.0× 287 1.6× 28 1.5k

Countries citing papers authored by Wei Bi

Since Specialization
Citations

This map shows the geographic impact of Wei Bi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Bi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Bi more than expected).

Fields of papers citing papers by Wei Bi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Bi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Bi. The network helps show where Wei Bi may publish in the future.

Co-authorship network of co-authors of Wei Bi

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Bi. A scholar is included among the top collaborators of Wei Bi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Bi. Wei Bi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xie, Yimeng, Zhengwei Cai, Jian Sun, et al.. (2025). The injectable cationic double-crosslinked hydrogel facilitates bone regeneration and repair through sustained antibacterial activity. Materials Today Bio. 35. 102461–102461. 1 indexed citations
2.
Li, Yafu, et al.. (2024). Spotting AI’s Touch: Identifying LLM-Paraphrased Spans in Text. 7088–7107. 1 indexed citations
3.
Zhou, Liming, Huiling Liu, Borui Zhang, et al.. (2024). A Novel 3D‐Printed Bi‐Layer Cranial‐Brain Patch Promotes Brain Injury Repair and Bone Tissue Regeneration. Advanced Functional Materials. 34(18). 23 indexed citations
4.
Fan, Yu, Jincai Wang, Jingyi Jian, et al.. (2024). High-throughput discovery of highly selective reversible hMAO-B inhibitors based on at-line nanofractionation. Acta Pharmaceutica Sinica B. 14(4). 1772–1786. 10 indexed citations
5.
Bi, Wei, Xinqi Zhou, Yihui Deng, et al.. (2023). PSMC5 regulates microglial polarization and activation in LPS-induced cognitive deficits and motor impairments by interacting with TLR4. Journal of Neuroinflammation. 20(1). 277–277. 21 indexed citations
6.
Bi, Wei, et al.. (2023). Exploring the research landscape of COVID-19-induced olfactory dysfunction: A bibliometric study. Frontiers in Neuroscience. 17. 1164901–1164901. 8 indexed citations
7.
Zhou, Ruiyi, Jiawei Zhang, Rui Guo, et al.. (2022). Targeted brain delivery of RVG29‐modified rifampicin‐loaded nanoparticles for Alzheimer's disease treatment and diagnosis. Bioengineering & Translational Medicine. 7(3). e10395–e10395. 24 indexed citations
8.
Jiang, Yingnan, Dan-Hua Lu, Ming‐Hai Pan, et al.. (2020). Tianma Gouteng granules decreases the susceptibility of Parkinson's disease by inhibiting ALOX15-mediated lipid peroxidation. Journal of Ethnopharmacology. 256. 112824–112824. 25 indexed citations
9.
Zhao, Jiayi, Wei Bi, Jiawei Zhang, et al.. (2020). USP8 protects against lipopolysaccharide-induced cognitive and motor deficits by modulating microglia phenotypes through TLR4/MyD88/NF-κB signaling pathway in mice. Brain Behavior and Immunity. 88. 582–596. 40 indexed citations
10.
Zhao, Jiayi, Wei Bi, Shu Xiao, et al.. (2019). Neuroinflammation induced by lipopolysaccharide causes cognitive impairment in mice. Scientific Reports. 9(1). 5790–5790. 645 indexed citations breakdown →
11.
Liang, Yanran, Xiuna Jing, Wei Bi, et al.. (2015). Rifampicin attenuates rotenone-induced inflammation via suppressing NLRP3 inflammasome activation in microglia. Brain Research. 1622. 43–50. 35 indexed citations
12.
Zhu, Li, Wei Bi, Dan Lu, et al.. (2014). Regulation of ubiquitin-specific processing protease 8 suppresses neuroinflammation. Molecular and Cellular Neuroscience. 64. 74–83. 16 indexed citations
13.
Bi, Wei, Li Zhu, Xiuna Jing, et al.. (2014). Investigations into the Role of 26S Proteasome Non-ATPase Regulatory Subunit 13 in Neuroinflammation. NeuroImmunoModulation. 21(6). 331–337. 13 indexed citations
14.
Jing, Xiuna, Qiaoyun Shi, Wei Bi, et al.. (2014). Rifampicin Protects PC12 Cells from Rotenone-Induced Cytotoxicity by Activating GRP78 via PERK-eIF2α-ATF4 Pathway. PLoS ONE. 9(3). e92110–e92110. 30 indexed citations
16.
Bi, Wei, Xiuna Jing, Li Zhu, et al.. (2012). Inhibition of 26S Protease Regulatory Subunit 7 (MSS1) Suppresses Neuroinflammation. PLoS ONE. 7(5). e36142–e36142. 11 indexed citations
17.
Zhu, Li, Wei Bi, Renbin Qi, Huadong Wang, & Daxiang Lu. (2011). Luteolin Inhibits Microglial Inflammation and Improves Neuron Survival Against Inflammation. International Journal of Neuroscience. 121(6). 329–336. 58 indexed citations
18.
Zhu, Li, Wei Bi, Renbin Qi, et al.. (2011). Luteolin reduces primary hippocampal neurons death induced by neuroinflammation. Neurological Research. 33(9). 927–934. 25 indexed citations
19.
Bi, Wei, Li Zhu, Yanran Liang, et al.. (2011). Rifampicin inhibits microglial inflammation and improves neuron survival against inflammation. Brain Research. 1395. 12–20. 75 indexed citations
20.
Wang, Jinglan, Bing Yang, Lina Song, et al.. (2008). Phosphoproteome analysis of the human Chang liver cells using SCX and a complementary mass spectrometric strategy. PROTEOMICS. 8(10). 2024–2034. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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