Yanming F. Wang

1.1k citations

8 papers · 935 indexed · 1 hit paper · h-index 7

Co-authors: Stuart A. Lipton Sulpicio G. Soriano Philip E. Stieg Sidney Strickland Stella E. Tsirka Paul R. Hickey Angela Coxon Tanya N. Mayadas
Topics: Cell Adhesion Molecules Research (3 papers)S100 Proteins and Annexins (3 papers)Acute Ischemic Stroke Management (2 papers)
Cited by: Neurology Immunology and Allergy Cancer Research
Journals: Nature MedicineSHILAP Revista de lepidopterologíaStroke
Partner nations: United States

In The Last Decade

Yanming F. Wang

8 papers receiving 922 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Tissue plasminogen activator (tPA) increase neuronal damage after focal cerebral ischemia in wild-type and tPA-deficient mice

1998 541 citations Yanming F. Wang, Stella E. Tsirka et al. Nature Medicine profile →

Peers

Countries citing papers authored by Yanming F. Wang

Since Specialization

Citations

This map shows the geographic impact of Yanming F. Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yanming F. Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yanming F. Wang more than expected).

Fields of papers citing papers by Yanming F. Wang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yanming F. Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yanming F. Wang. The network helps show where Yanming F. Wang may publish in the future.

Co-authorship network of co-authors of Yanming F. Wang

This figure shows the co-authorship network connecting the top 25 collaborators of Yanming F. Wang. A scholar is included among the top collaborators of Yanming F. Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yanming F. Wang. Yanming F. Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	Cerebrospinal fluid may mediate CNS ischemic injury 2005 ·SHILAP Revista de lepidopterología ·Yanming F. Wang,Judith K. Gwathmey,Guorong Zhang,Sulpicio G. Soriano,Shunli He,Yanguang Wang	9
2	P- and E-selectin-deficient mice are susceptible to cerebral ischemia–reperfusion injury 1999 ·Brain Research ·Sulpicio G. Soriano,Yanming F. Wang,Denisa D. Wagner,Paul S. Frenette	17
3	Mice Deficient in Mac-1 (CD11b/CD18) Are Less Susceptible to Cerebral Ischemia/Reperfusion Injury 1999 ·Stroke ·Sulpicio G. Soriano,Angela Coxon,Yanming F. Wang,Matthew P. Frosch,Stuart A. Lipton,Paul R. Hickey,Tanya N. Mayadas	185
4	805 MICE DEFICIENT IN NEUROTACTIN, A MEMBRANE BOUND CHEMOKINE ARE LESS SUSCEPTIBLE TO CEREBRAL-ISCHEMIA INJURY 1999 ·Journal of Neurosurgical Anesthesiology ·Sulpicio G. Soriano,Yanming F. Wang,Yang Pan	4
5	ICAM-1 dependent pathway is not involved in the development of neuronal apoptosis after transient focal cerebral ischemia 1998 ·Brain Research ·Sulpicio G. Soriano,Yanming F. Wang,Stuart A. Lipton,Pieter Dikkes,José-Carlos Gutierrez-Ramos,Paul R. Hickey	8
6	Tissue plasminogen activator (tPA) increase neuronal damage after focal cerebral ischemia in wild-type and tPA-deficient micebreakdown → 1998 ·Nature Medicine ·Yanming F. Wang,Stella E. Tsirka,Sidney Strickland,Philip E. Stieg,Sulpicio G. Soriano,Stuart A. Lipton	541
7	Enhanced neuronal death from focal ischemia in AMPA-receptor transgenic mice 1997 ·Molecular Brain Research ·Dean A. Le,Saumya Das,Yanming F. Wang,Toshihiro Yoshizawa,Y. Sasaki,(unknown),Adriana Nemes,Monica Mendelsohn,Pieter Dikkes,Stuart A. Lipton,Nobuki Nakanishi	25
8	Intercellular adhesion molecule‐1‐deficient mice are less susceptible to cerebral ischemia‐reperfusion lnjury 1996 ·Annals of Neurology ·Sulpicio G. Soriano,Stuart A. Lipton,Yanming F. Wang,(unknown),(unknown),Jose‐Carlos Gutierrez‐Ramos,Paul R. Hickey	146

About Yanming F. Wang

Yanming F. Wang is a scholar working on Immunology and Allergy, Neurology and Developmental Neuroscience, having authored 8 papers that have together received 935 indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (3 papers), S100 Proteins and Annexins (3 papers) and Acute Ischemic Stroke Management (2 papers). The work is most often cited by research in Neurology (324 citations), Immunology and Allergy (117 citations) and Cancer Research (256 citations). Yanming F. Wang has collaborated with scholars based in United States. Frequent co-authors include Stuart A. Lipton, Sulpicio G. Soriano, Philip E. Stieg, Sidney Strickland, Stella E. Tsirka, Paul R. Hickey, Sulpicio G. Soriano, Angela Coxon, Tanya N. Mayadas and Matthew P. Frosch. Their work appears in journals such as Nature Medicine, SHILAP Revista de lepidopterología and Stroke.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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