Y. Shiloh

885 total citations
9 papers, 716 citations indexed

About

Y. Shiloh is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Y. Shiloh has authored 9 papers receiving a total of 716 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 4 papers in Cancer Research and 2 papers in Oncology. Recurrent topics in Y. Shiloh's work include DNA Repair Mechanisms (6 papers), Carcinogens and Genotoxicity Assessment (4 papers) and Cancer-related Molecular Pathways (2 papers). Y. Shiloh is often cited by papers focused on DNA Repair Mechanisms (6 papers), Carcinogens and Genotoxicity Assessment (4 papers) and Cancer-related Molecular Pathways (2 papers). Y. Shiloh collaborates with scholars based in Israel, Australia and United States. Y. Shiloh's co-authors include Galit Rotman, Eynat Tabor, Yael Ziv, Tsafi Danieli, Aliza Amiel, N.G.J. Jaspers, L. Trakhtenbrot, Kum Kum Khanna, Dianne Watters and Jun Yan and has published in prestigious journals such as Journal of Biological Chemistry, Annual Review of Genetics and European Journal of Human Genetics.

In The Last Decade

Y. Shiloh

8 papers receiving 701 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Y. Shiloh Israel	7	655	341	272	64	51	9	716
Yosef Shiloh Israel	7	570 0.9×	299 0.9×	178 0.7×	62 1.0×	20 0.4×	9	666
Hungjiun Liaw Taiwan	15	756 1.2×	174 0.5×	192 0.7×	108 1.7×	61 1.2×	27	868
Marcin M. Gorski Italy	11	887 1.4×	177 0.5×	219 0.8×	56 0.9×	84 1.6×	15	1.1k
Thomas Schleker Switzerland	9	879 1.3×	306 0.9×	120 0.4×	183 2.9×	71 1.4×	10	962
Alan D. D’Andrea United States	15	1.0k 1.6×	344 1.0×	290 1.1×	174 2.7×	86 1.7×	17	1.1k
Lukas Rycak Germany	9	804 1.2×	256 0.8×	259 1.0×	118 1.8×	54 1.1×	9	986
Nobuko Matsushita Japan	11	719 1.1×	124 0.4×	293 1.1×	104 1.6×	89 1.7×	17	811
Florence Larminat France	18	686 1.0×	180 0.5×	122 0.4×	101 1.6×	73 1.4×	27	829
Velibor Savic United Kingdom	9	1.2k 1.8×	363 1.1×	155 0.6×	203 3.2×	56 1.1×	15	1.2k
Nadia Barboule France	15	670 1.0×	438 1.3×	90 0.3×	123 1.9×	30 0.6×	18	809

Countries citing papers authored by Y. Shiloh

Since Specialization

Citations

This map shows the geographic impact of Y. Shiloh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Y. Shiloh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Y. Shiloh more than expected).

Fields of papers citing papers by Y. Shiloh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Y. Shiloh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Y. Shiloh. The network helps show where Y. Shiloh may publish in the future.

Co-authorship network of co-authors of Y. Shiloh

This figure shows the co-authorship network connecting the top 25 collaborators of Y. Shiloh. A scholar is included among the top collaborators of Y. Shiloh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Y. Shiloh. Y. Shiloh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

9 of 9 papers shown

1.

Moyal, Lilach, Yoichi Taya, Luchino Chessa, et al.. (1998). Enhanced phosphorylation of p53 by ATN in response to DNA damage. 5 indexed citations

2.

Khanna, Kum Kum, Jun Yan, Dianne Watters, et al.. (1997). Defective Signaling through the B Cell Antigen Receptor in Epstein-Barr Virus-transformed Ataxia-Telangiectasia Cells. Journal of Biological Chemistry. 272(14). 9489–9495. 37 indexed citations

3.

Shiloh, Y.. (1997). ATAXIA-TELANGIECTASIA AND THE NIJMEGEN BREAKAGE SYNDROME: Related Disorders But Genes Apart. Annual Review of Genetics. 31(1). 635–662. 402 indexed citations

4.

Rotman, Galit & Y. Shiloh. (1997). The ATM gene and protein: possible roles in genome surveillance, checkpoint controls and cellular defence against oxidative stress.. PubMed. 29. 285–304. 67 indexed citations

5.

Shiloh, Y.. (1995). Ataxia-Telangiectasia:Closer to Unraveling the Mystery. European Journal of Human Genetics. 3(2). 116–138. 125 indexed citations

6.

Arush, M. Weyl Ben, et al.. (1995). Ataxia Telangiectasia and Lymphoma: An Indication for Individualized Chemotherapy Dosing—Report of Treatment in a Highly Inbred Arab Family. Pediatric Hematology and Oncology. 12(2). 163–169. 20 indexed citations

7.

Shomrat, Ruth, et al.. (1994). RELATIVELY LOW PROPORATION OF DYSTROPHIN GENE DELETIONS IN ISRAEL DUCHENNE AND BECKER MUSCULAR DYSTROPHY PATIENTS. 49. 379–383. 1 indexed citations

8.

Ziv, Yael, N.G.J. Jaspers, Tsafi Danieli, et al.. (1989). Cellular and molecular characteristics of an immortalized ataxia-telangiectasia (group AB) cell line.. PubMed. 49(9). 2495–501. 42 indexed citations

9.

Shiloh, Y., et al.. (1985). In vitro phenotype of ataxia-telangiectasia (AT) fibroblast strains: clues to the nature of the "AT DNA lesion" and the molecular defect in AT.. PubMed. 19. 111–21. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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