Lilach Moyal

3.2k total citations · 1 hit paper
18 papers, 2.3k citations indexed

About

Lilach Moyal is a scholar working on Dermatology, Molecular Biology and Oncology. According to data from OpenAlex, Lilach Moyal has authored 18 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Dermatology, 9 papers in Molecular Biology and 9 papers in Oncology. Recurrent topics in Lilach Moyal's work include Cutaneous lymphoproliferative disorders research (10 papers), DNA Repair Mechanisms (5 papers) and Cancer-related Molecular Pathways (5 papers). Lilach Moyal is often cited by papers focused on Cutaneous lymphoproliferative disorders research (10 papers), DNA Repair Mechanisms (5 papers) and Cancer-related Molecular Pathways (5 papers). Lilach Moyal collaborates with scholars based in Israel, United States and Switzerland. Lilach Moyal's co-authors include Yosef Shiloh, Yael Ziv, Nechama I. Smorodinsky, Carol Prives, Yoichi Taya, Carl W. Anderson, Yuval Reiss, Sharon Banin, L. Chessa and Sheau-Yann Shieh and has published in prestigious journals such as Science, Journal of Biological Chemistry and Genes & Development.

In The Last Decade

Lilach Moyal

18 papers receiving 2.2k citations

Hit Papers

Enhanced Phosphorylation of p53 by ATM in Response to DNA... 1998 2026 2007 2016 1998 500 1000 1.5k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lilach Moyal Israel 12 1.9k 1.2k 496 249 139 18 2.3k
Douglas Woods United States 11 2.0k 1.1× 1.3k 1.1× 383 0.8× 285 1.1× 228 1.6× 11 2.5k
Isabel García‐Cao Spain 15 1.4k 0.8× 726 0.6× 442 0.9× 153 0.6× 166 1.2× 16 1.9k
Nicholas D. Lakin United Kingdom 19 1.9k 1.0× 1.2k 1.0× 512 1.0× 277 1.1× 125 0.9× 30 2.3k
Sharon Banin United Kingdom 8 1.6k 0.8× 1.0k 0.9× 447 0.9× 398 1.6× 153 1.1× 11 2.0k
L. Chessa Italy 13 1.8k 0.9× 1.1k 0.9× 535 1.1× 241 1.0× 67 0.5× 18 2.0k
Joseph H. Taube United States 18 1.3k 0.7× 967 0.8× 656 1.3× 154 0.6× 131 0.9× 40 2.0k
Sylvia Wilder Israel 15 1.9k 1.0× 1.0k 0.9× 464 0.9× 492 2.0× 293 2.1× 21 2.7k
Sandy Giuliano France 23 1.4k 0.8× 656 0.5× 554 1.1× 280 1.1× 335 2.4× 29 2.1k
Imelda Mercado‐Uribe United States 24 1.2k 0.6× 922 0.8× 756 1.5× 421 1.7× 354 2.5× 28 2.2k
Xiangpeng Dai United States 20 1.5k 0.8× 838 0.7× 361 0.7× 325 1.3× 329 2.4× 29 2.2k

Countries citing papers authored by Lilach Moyal

Since Specialization
Citations

This map shows the geographic impact of Lilach Moyal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lilach Moyal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lilach Moyal more than expected).

Fields of papers citing papers by Lilach Moyal

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lilach Moyal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lilach Moyal. The network helps show where Lilach Moyal may publish in the future.

Co-authorship network of co-authors of Lilach Moyal

This figure shows the co-authorship network connecting the top 25 collaborators of Lilach Moyal. A scholar is included among the top collaborators of Lilach Moyal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lilach Moyal. Lilach Moyal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Wu, Xiwei, Hanjun Qin, Yate‐Ching Yuan, et al.. (2023). Reprogramming of PD-1+ M2-like tumor-associated macrophages with anti–PD-L1 and lenalidomide in cutaneous T cell lymphoma. JCI Insight. 8(13). 12 indexed citations
2.
Hodak, Emmilia, Avishay Edri, Rachel Eren, et al.. (2023). mAb14, a Monoclonal Antibody against Cell Surface PCNA: A Potential Tool for Sezary Syndrome Diagnosis and Targeted Immunotherapy. Cancers. 15(17). 4421–4421. 4 indexed citations
3.
Moyal, Lilach, Christiane Querfeld, Steven T. Rosen, et al.. (2021). Mycosis fungoides‐derived exosomes promote cell motility and are enriched with microRNA‐155 and microRNA‐1246, and their plasma‐cell‐free expression may serve as a potential biomarker for disease burden. British Journal of Dermatology. 185(5). 999–1012. 9 indexed citations
4.
Moyal, Lilach, et al.. (2021). CXCR4/CXCL12 axis as a potential therapeutic target in mycosis fungoides: an in-vitro study. European Journal of Cancer. 156. S12–S12. 1 indexed citations
5.
Aronovich, Anna, Lilach Moyal, Batia Gorovitz, et al.. (2020). Cancer-Associated Fibroblasts in Mycosis Fungoides Promote Tumor Cell Migration and Drug Resistance through CXCL12/CXCR4. Journal of Investigative Dermatology. 141(3). 619–627.e2. 47 indexed citations
6.
Mazuz, Moran, Lilach Moyal, Emmilia Hodak, et al.. (2020). Synergistic cytotoxic activity of cannabinoids fromcannabis sativaagainst cutaneous T-cell lymphoma (CTCL)in-vitroandex-vivo. Oncotarget. 11(13). 1141–1156. 27 indexed citations
7.
Moyal, Lilach, Shiran Yehezkel, Jasmin Jacob–Hirsch, et al.. (2018). Unilesional mycosis fungoides is associated with increased expression of micro RNA ‐17~92 and T helper 1 skewing. British Journal of Dermatology. 180(5). 1123–1134. 5 indexed citations
8.
Moyal, Lilach, Batia Gorovitz, Ada Rephaeli, et al.. (2017). AN-7, a butyric acid prodrug, sensitizes cutaneous T-cell lymphoma cell lines to doxorubicin via inhibition of DNA double strand breaks repair. Investigational New Drugs. 36(1). 1–9. 15 indexed citations
9.
Moyal, Lilach, Shiran Yehezkel, Aviad Keren, et al.. (2017). Oncogenic role of microRNA‐155 in mycosis fungoides: an in vitro and xenograft mouse model study. British Journal of Dermatology. 177(3). 791–800. 23 indexed citations
10.
Sherman, Shany, Esther Rabizadeh, Lilach Moyal, et al.. (2016). Fibrinogen-like Protein 2 Activity as a Potential Biomarker for Diagnosis of Early Mycosis Fungoides. Acta Dermato Venereologica. 97(3). 370–372. 2 indexed citations
11.
Moyal, Lilach, Ada Rephaeli, Abraham Nudelman, et al.. (2016). The Therapeutic Potential of AN-7, a Novel Histone Deacetylase Inhibitor, for Treatment of Mycosis Fungoides/Sezary Syndrome Alone or with Doxorubicin. PLoS ONE. 11(1). e0146115–e0146115. 11 indexed citations
12.
Shiloh, Yosef, Efrat Shema, Lilach Moyal, & Moshe Oren. (2011). RNF20–RNF40: A ubiquitin‐driven link between gene expression and the DNA damage response. FEBS Letters. 585(18). 2795–2802. 62 indexed citations
13.
Shema, Efrat, Itay Tirosh, Yael Aylon, et al.. (2008). The histone H2B-specific ubiquitin ligase RNF20/hBRE1 acts as a putative tumor suppressor through selective regulation of gene expression. Genes & Development. 22(19). 2664–2676. 224 indexed citations
14.
Fattal‐Valevski, Aviva, Lilach Moyal, Thomas O. Crawford, et al.. (2007). Ataxia‐telangiectasia: Mild neurological presentation despite null ATM mutation and severe cellular phenotype. American Journal of Medical Genetics Part A. 143A(16). 1827–1834. 39 indexed citations
15.
Bar‐Shira, Anat, et al.. (2002). ATM-dependent activation of the gene encoding MAP kinase phosphatase 5 by radiomimetic DNA damage. Oncogene. 21(5). 849–855. 26 indexed citations
16.
Moyal, Lilach, et al.. (2001). Nuclear Retention of ATM at Sites of DNA Double Strand Breaks. Journal of Biological Chemistry. 276(41). 38224–38230. 216 indexed citations
17.
Banin, Sharon, Lilach Moyal, Sheau-Yann Shieh, et al.. (1998). Enhanced Phosphorylation of p53 by ATM in Response to DNA Damage. Science. 281(5383). 1674–1677. 1542 indexed citations breakdown →
18.
Moyal, Lilach, Yoichi Taya, Luchino Chessa, et al.. (1998). Enhanced phosphorylation of p53 by ATN in response to DNA damage. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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