Xiong Li

531 total citations
22 papers, 316 citations indexed

About

Xiong Li is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Xiong Li has authored 22 papers receiving a total of 316 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 7 papers in Cancer Research and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Xiong Li's work include Ubiquitin and proteasome pathways (8 papers), RNA modifications and cancer (4 papers) and Epigenetics and DNA Methylation (3 papers). Xiong Li is often cited by papers focused on Ubiquitin and proteasome pathways (8 papers), RNA modifications and cancer (4 papers) and Epigenetics and DNA Methylation (3 papers). Xiong Li collaborates with scholars based in China and United States. Xiong Li's co-authors include Youhong Liu, Silong Wang, Jin‐Ming Lin, Jin Xu, Jitao Lv, Lixia Zhao, Janet M. Hock, Con Sullivan, Zhicheng Gong and Lunquan Sun and has published in prestigious journals such as The Journal of Cell Biology, PLoS ONE and Oncogene.

In The Last Decade

Xiong Li

20 papers receiving 314 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Xiong Li China 10 217 64 45 42 34 22 316
Qing Xia China 11 182 0.8× 70 1.1× 52 1.2× 16 0.4× 42 1.2× 18 315
Juanjuan Xie China 9 275 1.3× 101 1.6× 61 1.4× 16 0.4× 24 0.7× 19 385
Jun‐Dong Li China 5 250 1.2× 69 1.1× 53 1.2× 55 1.3× 45 1.3× 8 393
Fengli Zhang China 11 205 0.9× 92 1.4× 40 0.9× 102 2.4× 35 1.0× 30 416
Tiantian Lei China 11 199 0.9× 64 1.0× 36 0.8× 27 0.6× 26 0.8× 16 391
S Wang China 7 158 0.7× 54 0.8× 100 2.2× 30 0.7× 25 0.7× 13 362
Glowi Alasiri Saudi Arabia 9 156 0.7× 35 0.5× 38 0.8× 56 1.3× 16 0.5× 43 274
Yinghua Li China 8 197 0.9× 32 0.5× 58 1.3× 16 0.4× 15 0.4× 17 338
Yali Yu China 9 203 0.9× 40 0.6× 26 0.6× 23 0.5× 28 0.8× 26 289

Countries citing papers authored by Xiong Li

Since Specialization
Citations

This map shows the geographic impact of Xiong Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xiong Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xiong Li more than expected).

Fields of papers citing papers by Xiong Li

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xiong Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xiong Li. The network helps show where Xiong Li may publish in the future.

Co-authorship network of co-authors of Xiong Li

This figure shows the co-authorship network connecting the top 25 collaborators of Xiong Li. A scholar is included among the top collaborators of Xiong Li based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xiong Li. Xiong Li is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Peng, Yuchong, Jingjing Huang, Youhong Liu, et al.. (2024). SKP2 inhibition activates tumor cell-intrinsic immunity by inducing DNA replication stress and genomic instability. British Journal of Cancer. 132(1). 81–92. 5 indexed citations
3.
Gu, Lili, et al.. (2024). Roles of post-translational modifications of UHRF1 in cancer. Epigenetics & Chromatin. 17(1). 15–15. 9 indexed citations
4.
Peng, Yuchong, Youhong Liu, Yubing Ye, et al.. (2023). PLK1 maintains DNA methylation and cell viability by regulating phosphorylation-dependent UHRF1 protein stability. Cell Death Discovery. 9(1). 367–367. 6 indexed citations
5.
Zhao, Lixia, Jin Xu, Xiong Li, et al.. (2023). Recent development of chemiluminescence for bioanalysis. TrAC Trends in Analytical Chemistry. 166. 117213–117213. 67 indexed citations
6.
Li, Xiong, Jinsong Tan, Yan‐Qun Liu, et al.. (2023). LINC01305 recruits basonuclin 1 to act on G‐protein pathway suppressor 1 to promote esophageal squamous cell carcinoma. Cancer Science. 114(11). 4314–4328. 4 indexed citations
7.
Yin, Linglong, et al.. (2023). UHRF1 promotes spindle assembly and chromosome congression by catalyzing EG5 polyubiquitination. The Journal of Cell Biology. 222(11). 9 indexed citations
8.
Cao, Tuoyu, Yubing Ye, Youhong Liu, et al.. (2023). AKT1 regulates UHRF1 protein stability and promotes the resistance to abiraterone in prostate cancer. Oncogenesis. 12(1). 1–1. 15 indexed citations
9.
Peng, Yuchong, Rong Tang, Youhong Liu, et al.. (2023). Diosgenin inhibits prostate cancer progression by inducing UHRF1 protein degradation. European Journal of Pharmacology. 942. 175522–175522. 14 indexed citations
10.
Yin, Linglong, Yubing Ye, Ling Zou, et al.. (2023). AR antagonists develop drug resistance through TOMM20 autophagic degradation-promoted transformation to neuroendocrine prostate cancer. Journal of Experimental & Clinical Cancer Research. 42(1). 204–204. 8 indexed citations
11.
Deng, Tanggang, Qianling Zhu, Lin Xie, et al.. (2022). Norcantharidin promotes cancer radiosensitization through Cullin1 neddylation‐mediated CDC6 protein degradation. Molecular Carcinogenesis. 61(8). 812–824. 2 indexed citations
12.
Kong, Weijie, Changtao Wang, Mingbo Pu, et al.. (2021). Waveguide evanescent waves based structured illumination microscopy with compact structure and flexible design. Journal of Physics D Applied Physics. 54(21). 215101–215101. 2 indexed citations
13.
Liu, Xi, Xuyang Hou, Yan Zhou, et al.. (2019). <p>Downregulation of the Helicase Lymphoid-Specific (HELLS) Gene Impairs Cell Proliferation and Induces Cell Cycle Arrest in Colorectal Cancer Cells</p>. OncoTargets and Therapy. Volume 12. 10153–10163. 24 indexed citations
14.
Han, Chaofei, et al.. (2019). Knockdown of lncRNA‑UCA1 inhibits the proliferation and migration of melanoma cells through modulating the miR‑28‑5p/HOXB3 axis. Experimental and Therapeutic Medicine. 17(5). 4294–4302. 21 indexed citations
15.
Chang, Cheng, et al.. (2014). [The inhibitory effect of matrine on the growth of human colorectal cancer HT29 cells: an experimental observation].. PubMed. 34(1). 62–5. 2 indexed citations
16.
Liu, Youhong, Zhicheng Gong, Lunquan Sun, & Xiong Li. (2014). FOXM1 and androgen receptor co-regulate CDC6 gene transcription and DNA replication in prostate cancer cells. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1839(4). 297–305. 33 indexed citations
17.
Sullivan, Con, et al.. (2012). Novel Interactions between FOXM1 and CDC25A Regulate the Cell Cycle. PLoS ONE. 7(12). e51277–e51277. 29 indexed citations
18.
Liu, Youhong, Janet M. Hock, Con Sullivan, et al.. (2010). Activation of the p38 MAPK/Akt/ERK1/2 signal pathways is required for the protein stabilization of CDC6 and cyclin D1 in low‐dose arsenite‐induced cell proliferation. Journal of Cellular Biochemistry. 111(6). 1546–1555. 42 indexed citations
19.
Xie, Jin, Gennadiy Poda, Yiding Hu, et al.. (2010). Aminopyridinecarboxamide-based inhaled IKK-2 inhibitors for asthma and COPD: Structure–activity relationship. Bioorganic & Medicinal Chemistry. 19(3). 1242–1255. 14 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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