Wonjin Park

703 total citations
17 papers, 592 citations indexed

About

Wonjin Park is a scholar working on Cancer Research, Molecular Biology and Immunology. According to data from OpenAlex, Wonjin Park has authored 17 papers receiving a total of 592 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Cancer Research, 7 papers in Molecular Biology and 4 papers in Immunology. Recurrent topics in Wonjin Park's work include MicroRNA in disease regulation (5 papers), Angiogenesis and VEGF in Cancer (5 papers) and Pregnancy and preeclampsia studies (3 papers). Wonjin Park is often cited by papers focused on MicroRNA in disease regulation (5 papers), Angiogenesis and VEGF in Cancer (5 papers) and Pregnancy and preeclampsia studies (3 papers). Wonjin Park collaborates with scholars based in South Korea, Japan and China. Wonjin Park's co-authors include Kwon‐Soo Ha, Joo‐Hwan Kim, Seunghwan Choi, Young‐Guen Kwon, Young‐Myeong Kim, Minsik Park, Moo‐Ho Won, Dong-Keon Lee, Jong Yun Hwang and Sungwoo Ryoo and has published in prestigious journals such as Journal of Biological Chemistry, The FASEB Journal and Free Radical Biology and Medicine.

In The Last Decade

Wonjin Park

17 papers receiving 584 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wonjin Park South Korea 13 199 164 140 125 112 17 592
Xin Yan China 11 139 0.7× 85 0.5× 51 0.4× 71 0.6× 7 0.1× 37 450
Lihua Chen China 11 142 0.7× 84 0.5× 20 0.1× 31 0.2× 42 0.4× 32 378
Yingying Gong China 15 309 1.6× 164 1.0× 32 0.2× 39 0.3× 19 0.2× 32 522
Chengzhan Zhu China 14 332 1.7× 163 1.0× 22 0.2× 35 0.3× 22 0.2× 44 766
Xiaodong Jin China 20 520 2.6× 314 1.9× 384 2.7× 37 0.3× 30 0.3× 59 1.3k
Weilin Peng China 15 341 1.7× 73 0.4× 107 0.8× 24 0.2× 6 0.1× 43 558
Nicole Bitsch Netherlands 10 324 1.6× 128 0.8× 13 0.1× 103 0.8× 16 0.1× 16 662
Minho Kang South Korea 13 385 1.9× 252 1.5× 37 0.3× 163 1.3× 7 0.1× 21 810
Tianzhao Xu China 12 516 2.6× 315 1.9× 31 0.2× 56 0.4× 7 0.1× 22 849
Yiqiao Zhao China 11 236 1.2× 146 0.9× 19 0.1× 52 0.4× 17 0.2× 29 448

Countries citing papers authored by Wonjin Park

Since Specialization
Citations

This map shows the geographic impact of Wonjin Park's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wonjin Park with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wonjin Park more than expected).

Fields of papers citing papers by Wonjin Park

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wonjin Park. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wonjin Park. The network helps show where Wonjin Park may publish in the future.

Co-authorship network of co-authors of Wonjin Park

This figure shows the co-authorship network connecting the top 25 collaborators of Wonjin Park. A scholar is included among the top collaborators of Wonjin Park based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wonjin Park. Wonjin Park is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Park, Minsik, Joo‐Hwan Kim, Taesam Kim, et al.. (2021). REDD1 is a determinant of low-dose metronomic doxorubicin-elicited endothelial cell dysfunction through downregulation of VEGFR-2/3 expression. Experimental & Molecular Medicine. 53(10). 1612–1622. 12 indexed citations
2.
Park, Wonjin, Joo‐Hwan Kim, Seunghwan Choi, et al.. (2020). Human plasminogen-derived N-acetyl-Arg-Leu-Tyr-Glu antagonizes VEGFR-2 to prevent blood-retinal barrier breakdown in diabetic mice. Biomedicine & Pharmacotherapy. 134. 111110–111110. 6 indexed citations
3.
Kim, Jihee, Ji-Yoon Kim, Minsik Park, et al.. (2020). NF-κB-dependent miR-31/155 biogenesis is essential for TNF-α-induced impairment of endothelial progenitor cell function. Experimental & Molecular Medicine. 52(8). 1298–1309. 14 indexed citations
5.
Park, Minsik, Seunghwan Choi, Suji Kim, et al.. (2019). NF-κB-responsive miR-155 induces functional impairment of vascular smooth muscle cells by downregulating soluble guanylyl cyclase. Experimental & Molecular Medicine. 51(2). 1–12. 36 indexed citations
6.
Kim, Chan Ul, Jae Choul Yu, Eui Dae Jung, et al.. (2019). Optimization of device design for low cost and high efficiency planar monolithic perovskite/silicon tandem solar cells. Nano Energy. 60. 213–221. 98 indexed citations
7.
Park, Wonjin, Joo‐Hwan Kim, Dong Hyun Jo, et al.. (2019). Arg-Leu-Tyr-Glu Suppresses Retinal Endothelial Permeability and Choroidal Neovascularization by Inhibiting the VEGF Receptor 2 Signaling Pathway. Biomolecules & Therapeutics. 27(5). 474–483. 8 indexed citations
8.
Kim, Chan Ul, et al.. (2019). Two-terminal mechanical perovskite/silicon tandem solar cells with transparent conductive adhesives. Nano Energy. 65. 104044–104044. 48 indexed citations
9.
Choi, Seunghwan, Minsik Park, Joo‐Hwan Kim, et al.. (2018). TNF-α elicits phenotypic and functional alterations of vascular smooth muscle cells by miR-155-5p–dependent down-regulation of cGMP-dependent kinase 1. Journal of Biological Chemistry. 293(38). 14812–14822. 32 indexed citations
10.
Kim, Suji, Kyu‐Sun Lee, Seunghwan Choi, et al.. (2018). NF-κB–responsive miRNA-31-5p elicits endothelial dysfunction associated with preeclampsia via down-regulation of endothelial nitric-oxide synthase. Journal of Biological Chemistry. 293(49). 18989–19000. 67 indexed citations
11.
Kim, Joo‐Hwan, Seunghwan Choi, Min‐Sik Park, et al.. (2018). REDD‐1 aggravates endotoxin‐induced inflammation VIA atypical NF‐κB activation. The FASEB Journal. 32(8). 4585–4599. 33 indexed citations
12.
Choi, Seunghwan, Joo‐Hwan Kim, Dong-Keon Lee, et al.. (2017). Carbon monoxide prevents TNF-α-induced eNOS downregulation by inhibiting NF-κB-responsive miR-155-5p biogenesis. Experimental & Molecular Medicine. 49(11). e403–e403. 48 indexed citations
13.
Kim, Joo‐Hwan, Kyu‐Sun Lee, Ji‐Hee Kim, et al.. (2017). Aspirin prevents TNF-α-induced endothelial cell dysfunction by regulating the NF-κB-dependent miR-155/eNOS pathway: Role of a miR-155/eNOS axis in preeclampsia. Free Radical Biology and Medicine. 104. 185–198. 109 indexed citations
14.
Kim, Ji‐Hee, Dong-Keon Lee, Joo‐Hwan Kim, et al.. (2017). A miRNA-101-3p/Bim axis as a determinant of serum deprivation-induced endothelial cell apoptosis. Cell Death and Disease. 8(5). e2808–e2808. 28 indexed citations
15.
Lee, Dong-Keon, Joo‐Hwan Kim, Jihee Kim, et al.. (2016). Arg-Leu-Tyr-Glu tetrapeptide inhibits tumor progression by suppressing angiogenesis and vascular permeability via VEGF receptor-2 antagonism. Oncotarget. 8(7). 11763–11777. 14 indexed citations
16.
Nayab, Saira, et al.. (2012). Synthesis and characterization of novel tungsten complexes and their activity in the ROMP of cyclic olefins. Polyhedron. 42(1). 102–109. 7 indexed citations
17.
Inoue, Katsuro, Yoshiki Higo, Norihiro Yoshida, et al.. (2012). Experience of finding inconsistently-changed bugs in code clones of mobile software. 94–95. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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