William M. Schmidt

892 total citations
46 papers, 624 citations indexed

About

William M. Schmidt is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and General Health Professions. According to data from OpenAlex, William M. Schmidt has authored 46 papers receiving a total of 624 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 10 papers in Cardiology and Cardiovascular Medicine and 6 papers in General Health Professions. Recurrent topics in William M. Schmidt's work include Cardiomyopathy and Myosin Studies (10 papers), Muscle Physiology and Disorders (9 papers) and Cardiovascular Effects of Exercise (7 papers). William M. Schmidt is often cited by papers focused on Cardiomyopathy and Myosin Studies (10 papers), Muscle Physiology and Disorders (9 papers) and Cardiovascular Effects of Exercise (7 papers). William M. Schmidt collaborates with scholars based in United States, Germany and United Kingdom. William M. Schmidt's co-authors include Lisa Burkhart, Robert B. Reed, Thomas F. Pugh, Anthony Cammarato, Jeffrey R. Moore, William Lehman, Meera Viswanathan, Elizabeth P. Rice, D. Brian Foster and João Serôdio and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and JAMA.

In The Last Decade

William M. Schmidt

40 papers receiving 559 citations

Peers

William M. Schmidt
James A.J. Ferris United Kingdom
Tbl Kirkwood United Kingdom
H. Sobel United States
John D. Hardy United States
Robbee Wedow United States
William M. Schmidt
Citations per year, relative to William M. Schmidt William M. Schmidt (= 1×) peers Pei-Ying Lin

Countries citing papers authored by William M. Schmidt

Since Specialization
Citations

This map shows the geographic impact of William M. Schmidt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William M. Schmidt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William M. Schmidt more than expected).

Fields of papers citing papers by William M. Schmidt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William M. Schmidt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William M. Schmidt. The network helps show where William M. Schmidt may publish in the future.

Co-authorship network of co-authors of William M. Schmidt

This figure shows the co-authorship network connecting the top 25 collaborators of William M. Schmidt. A scholar is included among the top collaborators of William M. Schmidt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William M. Schmidt. William M. Schmidt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schmidt, William M., C. Parker, & Benjamin J. McCormick. (2025). 69-Year-Old Man With Acute Kidney Injury. Mayo Clinic Proceedings. 100(6). 1064–1069.
2.
Viswanathan, Meera, William M. Schmidt, Peter Franz, et al.. (2020). A role for actin flexibility in thin filament-mediated contractile regulation and myopathy. Nature Communications. 11(1). 2417–2417. 19 indexed citations
3.
Viswanathan, Meera, Kathleen C. Woulfe, William M. Schmidt, et al.. (2020). TNNT2 mutations in the tropomyosin binding region of TNT1 disrupt its role in contractile inhibition and stimulate cardiac dysfunction. Proceedings of the National Academy of Sciences. 117(31). 18822–18831. 24 indexed citations
4.
Lin, Ying, William M. Schmidt, Kristofer S. Fritz, et al.. (2020). Site-specific acetyl-mimetic modification of cardiac troponin I modulates myofilament relaxation and calcium sensitivity. Journal of Molecular and Cellular Cardiology. 139. 135–147. 23 indexed citations
5.
Schmidt, William M., et al.. (2020). Lysine acetylation of F-actin decreases tropomyosin-based inhibition of actomyosin activity. Journal of Biological Chemistry. 295(46). 15527–15539. 10 indexed citations
6.
Viswanathan, Meera, William M. Schmidt, Michael J. Rynkiewicz, et al.. (2017). Distortion of the Actin A-Triad Results in Contractile Disinhibition and Cardiomyopathy. Cell Reports. 20(11). 2612–2625. 25 indexed citations
7.
Serôdio, João, William M. Schmidt, & Silja Frankenbach. (2017). A chlorophyll fluorescence-based method for the integrated characterization of the photophysiological response to light stress. Journal of Experimental Botany. 68(5). erw492–erw492. 18 indexed citations
8.
Kooij, Viola, Meera Viswanathan, Dong I. Lee, et al.. (2016). Profilin modulates sarcomeric organization and mediates cardiomyocyte hypertrophy. Cardiovascular Research. 110(2). 238–248. 31 indexed citations
9.
Viswanathan, Meera, et al.. (2015). Pseudo-acetylation of K326 and K328 of actin disrupts Drosophila melanogaster indirect flight muscle structure and performance. Frontiers in Physiology. 6. 116–116. 34 indexed citations
10.
Schmidt, William M., et al.. (2015). Pseudo-Acetylation of Actin Residues K326 and K328 Disrupts Drosophila Flight Performance and Muscle Structure. Biophysical Journal. 108(2). 421a–422a. 1 indexed citations
11.
Muthu, Priya, Jingsheng Liang, William M. Schmidt, Jeffrey R. Moore, & Danuta Szczesna‐Cordary. (2013). In vitro rescue study of a malignant familial hypertrophic cardiomyopathy phenotype by pseudo-phosphorylation of myosin regulatory light chain. Archives of Biochemistry and Biophysics. 552-553. 29–39. 21 indexed citations
12.
Burkhart, Lisa & William M. Schmidt. (2012). Measuring Effectiveness of a Spiritual Care Pedagogy in Nursing Education. Journal of Professional Nursing. 28(5). 315–321. 58 indexed citations
13.
Huang, Jingsheng, et al.. (2006). CTLA-4·FasL inhibits allogeneic responses in vivo. Cellular Immunology. 239(2). 129–135. 7 indexed citations
14.
Schmidt, William M., et al.. (2003). CTLA-4 · FasL Induces Alloantigen-Specific Hyporesponsiveness. The Journal of Immunology. 170(12). 5842–5850. 24 indexed citations
15.
Schmidt, William M.. (1973). The development of health services for mothers and children in the United States.. American Journal of Public Health. 63(5). 419–427. 16 indexed citations
16.
Teele, James E., et al.. (1967). Factors related to social work services for mothers of babies born out of wedlock.. American Journal of Public Health and the Nations Health. 57(8). 1300–1307. 6 indexed citations
17.
Rice, Elizabeth P., et al.. (1965). Social Work in a Child Health Clinic: A Report of a Demonstration. American Journal of Public Health and the Nations Health. 55(6). 821–831. 4 indexed citations
18.
Pugh, Thomas F., et al.. (1963). Rates of Mental Disease Related to Childbearing. New England Journal of Medicine. 268(22). 1224–1228. 103 indexed citations
19.
Schmidt, William M., et al.. (1962). Variations in prenatal care and well-child supervision in a New England city. The Journal of Pediatrics. 61(3). 430–437. 8 indexed citations
20.
Schmidt, William M.. (1962). Health Services for Urban Youth. American Journal of Public Health and the Nations Health. 52(9). 1407–1412. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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