William J. Hendry

732 total citations
31 papers, 573 citations indexed

About

William J. Hendry is a scholar working on Genetics, Health, Toxicology and Mutagenesis and Molecular Biology. According to data from OpenAlex, William J. Hendry has authored 31 papers receiving a total of 573 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Genetics, 12 papers in Health, Toxicology and Mutagenesis and 10 papers in Molecular Biology. Recurrent topics in William J. Hendry's work include Estrogen and related hormone effects (17 papers), Effects and risks of endocrine disrupting chemicals (12 papers) and Reproductive Biology and Fertility (5 papers). William J. Hendry is often cited by papers focused on Estrogen and related hormone effects (17 papers), Effects and risks of endocrine disrupting chemicals (12 papers) and Reproductive Biology and Fertility (5 papers). William J. Hendry collaborates with scholars based in United States, United Kingdom and Ireland. William J. Hendry's co-authors include Wendell W. Leavitt, Rawden W. Evans, Benjamin J. Danzo, Shafiq A. Khan, Isabel R. Hendry, Tong J. Chen, John S. Davis, Ov D. Slayden, Daniel M. Sheehan and F. Stormshak and has published in prestigious journals such as Biochemical and Biophysical Research Communications, Endocrinology and Biology of Reproduction.

In The Last Decade

William J. Hendry

31 papers receiving 549 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
William J. Hendry United States 13 281 174 134 126 122 31 573
David R. Zehr United States 11 238 0.8× 141 0.8× 150 1.1× 43 0.3× 82 0.7× 11 446
John Krisinger Canada 14 292 1.0× 97 0.6× 107 0.8× 26 0.2× 152 1.2× 20 573
Mariana M. Yates United States 8 504 1.8× 121 0.7× 187 1.4× 57 0.5× 227 1.9× 10 910
C. Wang United States 10 189 0.7× 57 0.3× 58 0.4× 96 0.8× 172 1.4× 14 796
JK Findlay Australia 11 178 0.6× 29 0.2× 97 0.7× 194 1.5× 95 0.8× 20 457
Pavine Lefèvre Canada 14 103 0.4× 139 0.8× 112 0.8× 45 0.4× 172 1.4× 21 558
Annie Benhaïm France 15 150 0.5× 22 0.1× 97 0.7× 115 0.9× 134 1.1× 37 515
M L Dufau United States 7 161 0.6× 45 0.3× 48 0.4× 67 0.5× 216 1.8× 9 654
Karina F. Rodriguez United States 20 385 1.4× 80 0.5× 221 1.6× 95 0.8× 343 2.8× 38 1.0k
Signe M. Kilen United States 16 267 1.0× 58 0.3× 108 0.8× 58 0.5× 436 3.6× 18 917

Countries citing papers authored by William J. Hendry

Since Specialization
Citations

This map shows the geographic impact of William J. Hendry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William J. Hendry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William J. Hendry more than expected).

Fields of papers citing papers by William J. Hendry

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William J. Hendry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William J. Hendry. The network helps show where William J. Hendry may publish in the future.

Co-authorship network of co-authors of William J. Hendry

This figure shows the co-authorship network connecting the top 25 collaborators of William J. Hendry. A scholar is included among the top collaborators of William J. Hendry based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William J. Hendry. William J. Hendry is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Yao, Li, Sheng Yao, William T. Daly, et al.. (2012). Non-viral gene therapy for spinal cord regeneration. Drug Discovery Today. 17(17-18). 998–1005. 15 indexed citations
4.
Alwis, Imala, Isabel R. Hendry, Shyamal K. Roy, et al.. (2011). Neonatal diethylstilbestrol exposure disrupts female reproductive tract structure/function via both direct and indirect mechanisms in the hamster. Reproductive Toxicology. 32(4). 472–483. 10 indexed citations
5.
Hendry, William J., et al.. (2006). Differential progression of neonatal diethylstilbestrol-induced disruption of the hamster testis and seminal vesicle. Reproductive Toxicology. 21(3). 225–240. 12 indexed citations
6.
Hendry, William J., William S. Branham, & Daniel M. Sheehan. (2004). Diethylstilbestrol Versus Estradiol as Neonatal Disruptors of the Hamster (Mesocricetus auratus) Cervix1. Biology of Reproduction. 70(5). 1306–1316. 9 indexed citations
7.
Hendry, William J., et al.. (2003). Gene discovery in the hamster: a comparative genomics approach for gene annotation by sequencing of hamster testis cDNAs. BMC Genomics. 4(1). 22–22. 7 indexed citations
8.
Hendry, William J., Daniel M. Sheehan, Shafiq A. Khan, & Jeffrey V. May. (2002). Developing a laboratory animal model for perinatal endocrine disruption: the hamster chronicles. Holmes Museum Of Anthropology (Wichita State University). 2 indexed citations
9.
Hendry, William J., Daniel M. Sheehan, Shafiq A. Khan, & Jeffrey V. May. (2002). Developing a Laboratory Animal Model for Perinatal Endocrine Disruption: The Hamster Chronicles. Experimental Biology and Medicine. 227(9). 709–723. 20 indexed citations
10.
Rueda, Bo R., Isabel R. Hendry, William J. Hendry, et al.. (2000). Decreased Progesterone Levels and Progesterone Receptor Antagonists Promote Apoptotic Cell Death in Bovine Luteal Cells1. Biology of Reproduction. 62(2). 269–276. 118 indexed citations
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Zheng, X. Long & William J. Hendry. (1997). Neonatal diethylstilbestrol treatment alters the estrogen-regulated expression of both cell proliferation and apoptosis-related proto-oncogenes (c-jun, c-fos, c-myc, bax, bcl-2, and bcl-x) in the hamster uterus. Holmes Museum Of Anthropology (Wichita State University). 2 indexed citations
14.
Hendry, William J., Reza Hakkak, & Robert W. Harrison. (1993). An analysis of autologous glucocorticoid receptor protein regulation in AtT-20 cells also reveals differential specificity of the BuGR2 monoclonal antibody. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1178(2). 176–188. 5 indexed citations
15.
Hendry, William J. & Wendell W. Leavitt. (1993). Altered morphogenesis of the immature hamster uterus following neonatal exposure to diethylstilbestrol. Differentiation. 52(3). 221–227. 12 indexed citations
16.
Hendry, William J., Reza Hakkak, & Lawrence E. Cornett. (1992). Selective loss of glucocorticoid-dependent responses in a variant of the DDT1MF-2 tumor cell line.. PubMed. 52(9). 2516–22. 4 indexed citations
17.
Hendry, William J., William S. Branham, & Daniel M. Sheehan. (1992). The hamster cheek pouch as a convenient ectopic site for studies of uterine morphogenesis and endocrine responsiveness. Differentiation. 51(1). 49–54. 7 indexed citations
18.
Hendry, William J., et al.. (1990). The mouse glucocorticoid receptor DNA-binding domain is not phosphorylated invivo. Biochemical and Biophysical Research Communications. 166(2). 931–936. 5 indexed citations
19.
Harrison, Robert W., et al.. (1990). Isolation of a Genomic Sublibrary Enriched for Glucocorticoid-Regulated Genes. DNA and Cell Biology. 9(2). 95–102. 4 indexed citations
20.
Leavitt, Wendell W., Rawden W. Evans, & William J. Hendry. (1982). Etiology of DES-Induced Uterine Tumors in the Syrian Hamster. Advances in experimental medicine and biology. 138. 63–86. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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