Benjamin P. Weaver

800 total citations
16 papers, 574 citations indexed

About

Benjamin P. Weaver is a scholar working on Molecular Biology, Nutrition and Dietetics and Aging. According to data from OpenAlex, Benjamin P. Weaver has authored 16 papers receiving a total of 574 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Nutrition and Dietetics and 5 papers in Aging. Recurrent topics in Benjamin P. Weaver's work include Trace Elements in Health (6 papers), Genetics, Aging, and Longevity in Model Organisms (5 papers) and RNA regulation and disease (3 papers). Benjamin P. Weaver is often cited by papers focused on Trace Elements in Health (6 papers), Genetics, Aging, and Longevity in Model Organisms (5 papers) and RNA regulation and disease (3 papers). Benjamin P. Weaver collaborates with scholars based in United States, Japan and France. Benjamin P. Weaver's co-authors include Glen K. Andrews, Taiho Kambe, Jodi Dufner‐Beattie, Yi M. Weaver, Min Han, Mehmet Bilgen, Melissa A. Larson, Jim Geiser, Wenhao Xu and Shohei Mitani and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and PLoS ONE.

In The Last Decade

Benjamin P. Weaver

14 papers receiving 569 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Benjamin P. Weaver United States	10	372	232	196	132	105	16	574
Marouan Abouhamed Germany	10	212 0.6×	230 1.0×	188 1.0×	77 0.6×	30 0.3×	11	541
Katherine E. Vest United States	12	207 0.6×	88 0.4×	327 1.7×	38 0.3×	26 0.2×	18	556
Natalia VanDuyn United States	8	83 0.2×	82 0.4×	302 1.5×	34 0.3×	40 0.4×	10	518
Lydia K. Nyasae United States	10	367 1.0×	220 0.9×	163 0.8×	82 0.6×	31 0.3×	11	561
Kaori Ishihara Japan	9	402 1.1×	211 0.9×	200 1.0×	178 1.3×	105 1.0×	10	601
Mikaela Granvik Belgium	10	121 0.3×	42 0.2×	398 2.0×	35 0.3×	30 0.3×	11	820
Bryan Hall United States	5	470 1.3×	217 0.9×	246 1.3×	131 1.0×	97 0.9×	9	669
Dadin Fu United States	10	130 0.3×	44 0.2×	192 1.0×	27 0.2×	76 0.7×	10	456
Mainak Sengupta India	13	188 0.5×	96 0.4×	247 1.3×	47 0.4×	33 0.3×	55	539
Sı́lvia Canudas Spain	15	62 0.2×	20 0.1×	521 2.7×	15 0.1×	118 1.1×	25	893

Countries citing papers authored by Benjamin P. Weaver

Since Specialization

Citations

This map shows the geographic impact of Benjamin P. Weaver's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin P. Weaver with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin P. Weaver more than expected).

Fields of papers citing papers by Benjamin P. Weaver

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin P. Weaver. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin P. Weaver. The network helps show where Benjamin P. Weaver may publish in the future.

Co-authorship network of co-authors of Benjamin P. Weaver

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin P. Weaver. A scholar is included among the top collaborators of Benjamin P. Weaver based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin P. Weaver. Benjamin P. Weaver is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Weaver, Yi M., Chendong Yang, Guoli Hu, et al.. (2024). Proteolytic activation of fatty acid synthase signals pan-stress resolution. Nature Metabolism. 6(1). 113–126. 6 indexed citations

Weaver, Yi M., et al.. (2024). Xeroderma pigmentosum protein XPD controls caspase-mediated stress responses. Nature Communications. 15(1). 9344–9344.

Moreno, J. F., et al.. (2024). UBR-5 and UBE2D mediate timely exit from stem fate via destabilization of poly(A)-binding protein PABP-2 in cell state transition. Proceedings of the National Academy of Sciences. 121(43). e2407561121–e2407561121.

Wang, Yuan, Yi M. Weaver, Svetlana Earnest, et al.. (2023). Modulating p38 MAPK signaling by proteostasis mechanisms supports tissue integrity during growth and aging. Nature Communications. 14(1). 4543–4543. 20 indexed citations

Weaver, Benjamin P., Yi M. Weaver, Shizue Omi, et al.. (2020). Non-Canonical Caspase Activity Antagonizes p38 MAPK Stress-Priming Function to Support Development. Developmental Cell. 53(3). 358–369.e6. 25 indexed citations

Weaver, Benjamin P. & Min Han. (2017). Tag team: Roles of miRNAs and Proteolytic Regulators in Ensuring Robust Gene Expression Dynamics. Trends in Genetics. 34(1). 21–29. 3 indexed citations

Morley, Steven K., et al.. (2017). Perturbed-input-data ensemble modeling of magnetospheric dynamics. AGUFM. 2017. 1 indexed citations

Weaver, Benjamin P., Yi M. Weaver, Shohei Mitani, & Min Han. (2017). Coupled Caspase and N-End Rule Ligase Activities Allow Recognition and Degradation of Pluripotency Factor LIN-28 during Non-Apoptotic Development. Developmental Cell. 41(6). 665–673.e6. 37 indexed citations

Weaver, Benjamin P., Rebecca Zabinsky, Yi M. Weaver, et al.. (2014). CED-3 caspase acts with miRNAs to regulate non-apoptotic gene expression dynamics for robust development in C. elegans. eLife. 3. e04265–e04265. 48 indexed citations

10.

Weaver, Benjamin P. & Glen K. Andrews. (2011). Regulation of zinc-responsive Slc39a5 (Zip5) translation is mediated by conserved elements in the 3′-untranslated region. BioMetals. 25(2). 319–335. 28 indexed citations

11.

Weaver, Benjamin P., Yuxia Zhang, Stephen Hiscox, et al.. (2010). Zip4 (Slc39a4) Expression is Activated in Hepatocellular Carcinomas and Functions to Repress Apoptosis, Enhance Cell Cycle and Increase Migration. PLoS ONE. 5(10). e13158–e13158. 66 indexed citations

12.

Kambe, Taiho, Benjamin P. Weaver, & Glen K. Andrews. (2008). The genetics of essential metal homeostasis during development. genesis. 46(4). 214–228. 102 indexed citations

13.

Kambe, Taiho, Benjamin P. Weaver, & Glen K. Andrews. (2008). The genetics of essential metal homeostasis during development. genesis. 46(4). 5 indexed citations

14.

Dufner‐Beattie, Jodi, Benjamin P. Weaver, Jim Geiser, et al.. (2007). The mouse acrodermatitis enteropathica gene Slc39a4 ( Zip4 ) is essential for early development and heterozygosity causes hypersensitivity to zinc deficiency. Human Molecular Genetics. 16(12). 1391–1399. 96 indexed citations

15.

Weaver, Benjamin P., Jodi Dufner‐Beattie, Taiho Kambe, & Glen K. Andrews. (2007). Novel zinc-responsive post-transcriptional mechanisms reciprocally regulate expression of the mouse Slc39a4 and Slc39a5 zinc transporters (Zip4 and Zip5). Biological Chemistry. 388(12). 1301–1312. 125 indexed citations

16.

Hendry, William J., et al.. (2006). Differential progression of neonatal diethylstilbestrol-induced disruption of the hamster testis and seminal vesicle. Reproductive Toxicology. 21(3). 225–240. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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