Nina D. Ullrich

1.7k total citations
47 papers, 1.3k citations indexed

About

Nina D. Ullrich is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Nina D. Ullrich has authored 47 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Molecular Biology, 24 papers in Cardiology and Cardiovascular Medicine and 17 papers in Cellular and Molecular Neuroscience. Recurrent topics in Nina D. Ullrich's work include Cardiac electrophysiology and arrhythmias (17 papers), Neuroscience and Neural Engineering (16 papers) and Ion channel regulation and function (14 papers). Nina D. Ullrich is often cited by papers focused on Cardiac electrophysiology and arrhythmias (17 papers), Neuroscience and Neural Engineering (16 papers) and Ion channel regulation and function (14 papers). Nina D. Ullrich collaborates with scholars based in Germany, Switzerland and United States. Nina D. Ullrich's co-authors include Ernst Niggli, Natalia Shirokova, Bernd Nilius, Jean Prenen, Guy Droogmans, Rudi Vennekens, Thomas Voets, Karel Talavera, Kenneth T. MacLeod and Markus Hecker and has published in prestigious journals such as The Journal of Immunology, PLoS ONE and Biomaterials.

In The Last Decade

Nina D. Ullrich

46 papers receiving 1.3k citations

Peers

Nina D. Ullrich

CCM

CMN

SURGE

John J. Kelly Canada

Randolph Corteling United Kingdom

Hitoshi Onoue Japan

Yukinao Shibukawa Japan

Séverine Groh United States

Kevin M. Tharp United States

Deqiang Li United States

Núria Comes Spain

Xin‐Ming Shen United States

John J. Kelly Canada

Nina D. Ullrich

1.0k ×1.4

83 ×0.2

CCM

103 ×0.5

CMN

281 ×1.5

92 ×0.5

SURGE

Citations per year, relative to Nina D. Ullrich Nina D. Ullrich (= 1×) peers John J. Kelly

Countries citing papers authored by Nina D. Ullrich

Since Specialization

Citations

This map shows the geographic impact of Nina D. Ullrich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nina D. Ullrich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nina D. Ullrich more than expected).

Fields of papers citing papers by Nina D. Ullrich

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nina D. Ullrich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nina D. Ullrich. The network helps show where Nina D. Ullrich may publish in the future.

Co-authorship network of co-authors of Nina D. Ullrich

This figure shows the co-authorship network connecting the top 25 collaborators of Nina D. Ullrich. A scholar is included among the top collaborators of Nina D. Ullrich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nina D. Ullrich. Nina D. Ullrich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Rahm, Ann‐Kathrin, Daniël A. Pijnappels, Antoine A.F. de Vries, et al.. (2024). Cardiac stereotactic body radiotherapy to treat malignant ventricular arrhythmias directly affects the cardiomyocyte electrophysiology. Heart Rhythm. 22(1). 90–99. 5 indexed citations

Mosqueira, Matías, Enrico Domenico Lemma, Kleopatra Rapti, et al.. (2023). Membrane remodelling triggers maturation of excitation–contraction coupling in 3D-shaped human-induced pluripotent stem cell-derived cardiomyocytes. Basic Research in Cardiology. 118(1). 13–13. 6 indexed citations

Frisk, Michael, Cathrine R. Carlson, Andreas Brech, et al.. (2023). BIN1, Myotubularin, and Dynamin-2 Coordinate T-Tubule Growth in Cardiomyocytes. Circulation Research. 132(11). e188–e205. 15 indexed citations

Schmidt, Constanze, et al.. (2022). Distress-Mediated Remodeling of Cardiac Connexin-43 in a Novel Cell Model for Arrhythmogenic Heart Diseases. International Journal of Molecular Sciences. 23(17). 10174–10174. 14 indexed citations

Rahm, Ann‐Kathrin, Patrick Most, Tanja Weis, et al.. (2021). Trigger-Specific Remodeling of KCa2 Potassium Channels in Models of Atrial Fibrillation. Pharmacogenomics and Personalized Medicine. Volume 14. 579–590. 8 indexed citations

Rahm, Ann‐Kathrin, Patrick Lugenbiel, Rasmus Rivinius, et al.. (2020). Inhibition of cardiac Kv4.3 (Ito) channel isoforms by class I antiarrhythmic drugs lidocaine and mexiletine. European Journal of Pharmacology. 880. 173159–173159. 5 indexed citations

Schweizer, Patrick A., Fabrice F. Darche, Nina D. Ullrich, et al.. (2017). Subtype-specific differentiation of cardiac pacemaker cell clusters from human induced pluripotent stem cells. Stem Cell Research & Therapy. 8(1). 229–229. 56 indexed citations

Richter, Benjamin, et al.. (2017). Novel Microarchitecture Induces Functional Remodeling of the Calcium Signaling Mechanisms in Restructured IPSC-Cardiomyocytes. Biophysical Journal. 112(3). 537a–537a. 1 indexed citations

Dias, Priyanthi, Thomas Desplantez, Majd El‐Harasis, et al.. (2014). Characterisation of Connexin Expression and Electrophysiological Properties in Stable Clones of the HL-1 Myocyte Cell Line. PLoS ONE. 9(2). e90266–e90266. 38 indexed citations

10.

Ullrich, Nina D., et al.. (2013). ‘Eventless’ InsP₃‐dependent SR‐Ca²⁺ release affecting atrial Ca²⁺ sparks. The Journal of Physiology. 591(8). 2103–2111. 13 indexed citations

11.

Kyrychenko, Sergii, Eva Polaková, Nina D. Ullrich, Ernst Niggli, & Natalia Shirokova. (2012). Insights into RyRs Dysfunctions via Studies of Intracellular Calcium Signals. Biophysical Journal. 102(3). 213a–213a. 1 indexed citations

12.

Niggli, Ernst, et al.. (2012). Posttranslational modifications of cardiac ryanodine receptors: Ca2+ signaling and EC-coupling. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1833(4). 866–875. 74 indexed citations

13.

Ullrich, Nina D., Héctor H. Valdivia, & Ernst Niggli. (2012). PKA phosphorylation of cardiac ryanodine receptor modulates SR luminal Ca2+ sensitivity. Journal of Molecular and Cellular Cardiology. 53(1). 33–42. 46 indexed citations

14.

Gonzales, Christine R., Nina D. Ullrich, Stefan Gerber, et al.. (2011). Isolation of Cardiovascular Precursor Cells from the Human Fetal Heart. Tissue Engineering Part A. 18(1-2). 198–207. 16 indexed citations

15.

Ullrich, Nina D. & Ernst Niggli. (2011). Cardiac Ryanodine Receptor Phosphorylation at Ser2808 is Involved in Intra-SR Calcium Sensing. Biophysical Journal. 100(3). 353a–353a. 1 indexed citations

16.

Ullrich, Nina D. & Ernst Niggli. (2010). Impaired Ca2+ Release Synchronization in RyR2-S2808a Mouse Cardiomyocytes During β-Adrenergic Stimulation. Biophysical Journal. 98(3). 550a–550a. 1 indexed citations

17.

Stahl, Heiko, Tanja Fauti, Nina D. Ullrich, et al.. (2009). miR-155 Inhibition Sensitizes CD4+ Th Cells for TREG Mediated Suppression. PLoS ONE. 4(9). e7158–e7158. 72 indexed citations

18.

Shkryl, Vyacheslav M., Adriano S. Martins, Nina D. Ullrich, et al.. (2009). Reciprocal amplification of ROS and Ca2+ signals in stressed mdx dystrophic skeletal muscle fibers. Pflügers Archiv - European Journal of Physiology. 458(5). 915–928. 89 indexed citations

19.

Peters, Björn, Nina D. Ullrich, Oliver Carroll, et al.. (2008). Characterizing the N-Terminal Processing Motif of MHC Class I Ligands. The Journal of Immunology. 180(5). 3210–3217. 33 indexed citations

20.

Ullrich, Nina D., Thomas Voets, Jean Prenen, et al.. (2005). Comparison of functional properties of the Ca2+-activated cation channels TRPM4 and TRPM5 from mice. Cell Calcium. 37(3). 267–278. 202 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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