William C. Dunty

870 total citations
10 papers, 676 citations indexed

About

William C. Dunty is a scholar working on Molecular Biology, Pediatrics, Perinatology and Child Health and Rheumatology. According to data from OpenAlex, William C. Dunty has authored 10 papers receiving a total of 676 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Pediatrics, Perinatology and Child Health and 2 papers in Rheumatology. Recurrent topics in William C. Dunty's work include Developmental Biology and Gene Regulation (6 papers), Epigenetics and DNA Methylation (3 papers) and Wnt/β-catenin signaling in development and cancer (3 papers). William C. Dunty is often cited by papers focused on Developmental Biology and Gene Regulation (6 papers), Epigenetics and DNA Methylation (3 papers) and Wnt/β-catenin signaling in development and cancer (3 papers). William C. Dunty collaborates with scholars based in United States and Italy. William C. Dunty's co-authors include Terry P. Yamaguchi, Kathleen K. Sulik, Ravindra B. Chalamalasetty, Kristin K. Biris, Deborah B. Dehart, Shao‐yu Chen, Mark Lewandoski, Makoto M. Taketo, Robert M. Zucker and Han Si and has published in prestigious journals such as Nature Communications, PLoS ONE and Development.

In The Last Decade

William C. Dunty

10 papers receiving 665 citations

Peers

Countries citing papers authored by William C. Dunty

Since Specialization

Citations

This map shows the geographic impact of William C. Dunty's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William C. Dunty with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William C. Dunty more than expected).

Fields of papers citing papers by William C. Dunty

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William C. Dunty. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William C. Dunty. The network helps show where William C. Dunty may publish in the future.

Co-authorship network of co-authors of William C. Dunty

This figure shows the co-authorship network connecting the top 25 collaborators of William C. Dunty. A scholar is included among the top collaborators of William C. Dunty based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William C. Dunty. William C. Dunty is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	Mesogenin 1 is a master regulator of paraxial presomitic mesoderm differentiation 2014 ·Development ·Ravindra B. Chalamalasetty, Robert J. Garriock, William C. Dunty, (unknown), Parthav Jailwala, Han Si, Terry P. Yamaguchi	85
2	Transcriptional Profiling of Wnt3a Mutants Identifies Sp Transcription Factors as Essential Effectors of the Wnt/β-catenin Pathway in Neuromesodermal Stem Cells 2014 ·PLoS ONE ·William C. Dunty, (unknown), Ravindra B. Chalamalasetty, Kenneth Campbell, Terry P. Yamaguchi	43
3	The Wnt3a/β-catenin target gene Mesogenin1 controls the segmentation clock by activating a Notch signalling program 2011 ·Nature Communications ·Ravindra B. Chalamalasetty, William C. Dunty, Kristin K. Biris, Rieko Ajima, Michelina Iacovino, Arica Beisaw, Lionel Feigenbaum, Deborah L. Chapman, Jeong Kyo Yoon, Michael Kyba, Terry P. Yamaguchi	52
4	Mouse Ripply2 is downstream of Wnt3a and is dynamically expressed during somitogenesis 2007 ·Developmental Dynamics ·Kristin K. Biris, William C. Dunty, Terry P. Yamaguchi	27
5	Wnt3a/β-catenin signaling controls posterior body development by coordinating mesoderm formation and segmentation 2007 ·Development ·William C. Dunty, Kristin K. Biris, Ravindra B. Chalamalasetty, Makoto M. Taketo, Mark Lewandoski, Terry P. Yamaguchi	164
6	Immunohistochemical and Microarray Analyses of a Mouse Model for the Smith-Lemli-Opitz Syndrome 2005 ·Developmental Neuroscience ·Heather Waage-Baudet, William C. Dunty, Deborah B. Dehart, Sylvia Hiller, Kathleen K. Sulik	21
7	Identification and comparative expression analyses of Daam genes in mouse and Xenopus 2004 ·Gene Expression Patterns ·Masa‐aki Nakaya, Raymond Habas, Kristin K. Biris, William C. Dunty, Yoichi Kato, Xi He, Terry P. Yamaguchi	41
8	Hindbrain and Cranial Nerve Dysmorphogenesis Result from Acute Maternal Ethanol Administration 2002 ·Developmental Neuroscience ·William C. Dunty, Robert M. Zucker, Kathleen K. Sulik	56
9	Selective Vulnerability of Embryonic Cell Populations to Ethanol‐Induced Apoptosis: Implications for Alcohol‐Related Birth Defects and Neurodevelopmental Disorder 2001 ·Alcoholism Clinical and Experimental Research ·William C. Dunty, Shao‐yu Chen, (unknown), Deborah B. Dehart, Kathleen K. Sulik	177
10	Selective Vulnerability of Embryonic Cell Populations to Ethanol-Induced Apoptosis: Implications for Alcohol-Related Birth Defects and Neurodevelopmental Disorder 2001 ·Alcoholism Clinical and Experimental Research ·William C. Dunty, Shaoyu Chen, Robert M. Zucker, Deborah B. Dehart, Kathleen K. Sulik	10

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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