Werner Hoch
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 1%
- Neurology top 1%
- Cell Biology top 2%
- Infectious Diseases top 5%
- Co-authors
- Heinrich BetzJames T. CampanelliRichard H. SchellerCord‐Michael BeckerAngela VincentJohn Newsom–DavisJohn McConvilleSigrun Helms
- Topics
- Ion channel regulation and function (15 papers)Neuroscience and Neuropharmacology Research (10 papers)Muscle Physiology and Disorders (7 papers)
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
Werner Hoch
36 papers receiving 3.3k citations
Hit Papers
Peers
Comparison fields: 5 of 92
- Molecular Biology 2.0k
- Cellular and Molecular Neuroscience 1.4k
- Neurology 1.1k
- Cell Biology 632
- Infectious Diseases 344
Countries citing papers authored by Werner Hoch
This map shows the geographic impact of Werner Hoch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Werner Hoch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Werner Hoch more than expected).
Fields of papers citing papers by Werner Hoch
This network shows the impact of papers produced by Werner Hoch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Werner Hoch. The network helps show where Werner Hoch may publish in the future.
Co-authorship network of co-authors of Werner Hoch
This figure shows the co-authorship network connecting the top 25 collaborators of Werner Hoch. A scholar is included among the top collaborators of Werner Hoch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Werner Hoch. Werner Hoch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 56 | |
| 2 | 30 | |
| 3 | 15 | |
| 4 | 15 | |
| 5 | Auto-antibodies to the receptor tyrosine kinase MuSK in patients with myasthenia gravis without acetylcholine receptor antibodiesbreakdown → | 839 |
| 6 | 53 | |
| 7 | 43 | |
| 8 | 92 | |
| 9 | 79 | |
| 10 | 38 | |
| 11 | 73 | |
| 12 | 69 | |
| 13 | 270 | |
| 14 | 11 | |
| 15 | 13 | |
| 16 | 18 | |
| 17 | 105 | |
| 18 | 25 | |
| 19 | 132 | |
| 20 | 54 |
About Werner Hoch
Werner Hoch is a scholar working on Cellular and Molecular Neuroscience, Cell Biology and Molecular Biology, having authored 36 papers that have together received 3.3k indexed citations. Recurring topics across this work include Ion channel regulation and function (15 papers), Neuroscience and Neuropharmacology Research (10 papers) and Muscle Physiology and Disorders (7 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.4k citations), Neurology (1.1k citations) and Cell Biology (632 citations). Werner Hoch has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Heinrich Betz, James T. Campanelli, Richard H. Scheller, Cord‐Michael Becker, Angela Vincent, John Newsom–Davis, John McConville, Sigrun Helms, Arthur Melms and Michael Ferns. Their work appears in journals such as Cell, Journal of Biological Chemistry and Nature Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.