Wei‐Li Kuan

1.9k total citations
31 papers, 1.1k citations indexed

About

Wei‐Li Kuan is a scholar working on Neurology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Wei‐Li Kuan has authored 31 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Neurology, 13 papers in Cellular and Molecular Neuroscience and 12 papers in Molecular Biology. Recurrent topics in Wei‐Li Kuan's work include Parkinson's Disease Mechanisms and Treatments (10 papers), Nerve injury and regeneration (8 papers) and Neurological disorders and treatments (6 papers). Wei‐Li Kuan is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (10 papers), Nerve injury and regeneration (8 papers) and Neurological disorders and treatments (6 papers). Wei‐Li Kuan collaborates with scholars based in United Kingdom, Australia and United States. Wei‐Li Kuan's co-authors include Roger A. Barker, Anna Huefner, Sumeet Mahajan, Sarah Mason, Jeremy N. Skepper, Isabelle St‐Amour, Penny Gowland, Giulia Cisbani, Susan Francis and Steve Lacroix and has published in prestigious journals such as Nature Communications, The Journal of Experimental Medicine and Nano Letters.

In The Last Decade

Wei‐Li Kuan

31 papers receiving 1.1k citations

Peers

Wei‐Li Kuan
Jennifer McDonough United States
Chaejeong Heo South Korea
Scott R. Burks United States
Marco Morsch Australia
Charles Ducrot Canada
Cláudia Saraiva Portugal
Devrim Kilinc Ireland
Chiara Cordiglieri Italy
Ferdinanda Annesi Italy
Markus Aswendt Germany
Jennifer McDonough United States
Wei‐Li Kuan
Citations per year, relative to Wei‐Li Kuan Wei‐Li Kuan (= 1×) peers Jennifer McDonough

Countries citing papers authored by Wei‐Li Kuan

Since Specialization
Citations

This map shows the geographic impact of Wei‐Li Kuan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei‐Li Kuan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei‐Li Kuan more than expected).

Fields of papers citing papers by Wei‐Li Kuan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei‐Li Kuan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei‐Li Kuan. The network helps show where Wei‐Li Kuan may publish in the future.

Co-authorship network of co-authors of Wei‐Li Kuan

This figure shows the co-authorship network connecting the top 25 collaborators of Wei‐Li Kuan. A scholar is included among the top collaborators of Wei‐Li Kuan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei‐Li Kuan. Wei‐Li Kuan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rahman, S.M. Jamshedur, et al.. (2025). Connexin 43 is downregulated in advanced Parkinson’s disease in multiple brain regions which correlates with symptoms. Scientific Reports. 15(1). 10250–10250. 1 indexed citations
2.
Barker, Roger A., et al.. (2024). An update on immune-based alpha-synuclein trials in Parkinson’s disease. Journal of Neurology. 272(1). 21–21. 14 indexed citations
3.
Baranes, Koby, S.M. Jamshedur Rahman, Wei‐Li Kuan, et al.. (2023). Transcription factor combinations that define human astrocyte identity encode significant variation of maturity and function. Glia. 71(8). 1870–1889. 8 indexed citations
4.
Kuan, Wei‐Li, et al.. (2022). Therapeutic Potential of Astrocyte Transplantation. Cell Transplantation. 31. 4221163355–4221163355. 25 indexed citations
5.
Bråthen, Geir, et al.. (2021). Early functional changes associated with alpha-synuclein proteinopathy in engineered human neural networks. American Journal of Physiology-Cell Physiology. 320(6). C1141–C1152. 9 indexed citations
6.
Dar, Ghulam Hassan, Cláudia C. Mendes, Wei‐Li Kuan, et al.. (2021). GAPDH controls extracellular vesicle biogenesis and enhances the therapeutic potential of EV mediated siRNA delivery to the brain. Nature Communications. 12(1). 6666–6666. 65 indexed citations
7.
Zhao, Yanyan, Ole Tietz, Wei‐Li Kuan, et al.. (2020). A fluorescent molecular imaging probe with selectivity for soluble tau aggregated protein. Chemical Science. 11(18). 4773–4778. 26 indexed citations
8.
Armstrong, James P. K., Timothy J. Keane, P. Stephen Patrick, et al.. (2020). A blueprint for translational regenerative medicine. Science Translational Medicine. 12(572). 33 indexed citations
9.
Harris, Kate, Wei‐Li Kuan, Sarah Mason, & Roger A. Barker. (2020). Antidopaminergic treatment is associated with reduced chorea and irritability but impaired cognition in Huntington’s disease (Enroll-HD). Journal of Neurology Neurosurgery & Psychiatry. 91(6). 622–630. 23 indexed citations
10.
Wijeyekoon, Ruwani, Deborah Kronenberg‐Versteeg, Kirsten M. Scott, et al.. (2020). Peripheral innate immune and bacterial signals relate to clinical heterogeneity in Parkinson’s disease. Brain Behavior and Immunity. 87. 473–488. 72 indexed citations
11.
Kuan, Wei‐Li, Judith Mihály, Zoltán Varga, et al.. (2019). DJ-1 can form β-sheet structured aggregates that co-localize with pathological amyloid deposits. Neurobiology of Disease. 134. 104629–104629. 8 indexed citations
12.
Kuan, Wei‐Li, Katherine Stott, Xiaoling He, et al.. (2019). Systemic α-synuclein injection triggers selective neuronal pathology as seen in patients with Parkinson’s disease. Molecular Psychiatry. 26(2). 556–567. 22 indexed citations
13.
Kuan, Wei‐Li, Neal K. Bennett, Xiaoling He, et al.. (2016). α-Synuclein pre-formed fibrils impair tight junction protein expression without affecting cerebral endothelial cell function. Experimental Neurology. 285(Pt A). 72–81. 60 indexed citations
14.
Poole, Emma, Wei‐Li Kuan, Roger A. Barker, & John Sinclair. (2015). The human cytomegalovirus non-coding Beta2.7 RNA as a novel therapeutic for Parkinson's disease – Translational research with no translation. Virus Research. 212. 64–69. 12 indexed citations
15.
Huefner, Anna, Wei‐Li Kuan, Karin H. Müller, et al.. (2015). Characterization and Visualization of Vesicles in the Endo-Lysosomal Pathway with Surface-Enhanced Raman Spectroscopy and Chemometrics. ACS Nano. 10(1). 307–316. 86 indexed citations
16.
Kuan, Wei‐Li, Emma Poole, Michael Fletcher, et al.. (2011). A novel neuroprotective therapy for Parkinson’s disease using a viral noncoding RNA that protects mitochondrial Complex I activity. The Journal of Experimental Medicine. 209(1). 1–10. 32 indexed citations
17.
Barker, Roger A. & Wei‐Li Kuan. (2010). Graft-Induced Dyskinesias in Parkinson's Disease: What Is It All About?. Cell stem cell. 7(2). 148–149. 26 indexed citations
18.
19.
Kuan, Wei‐Li, Carrie B. Hurelbrink, & Roger A. Barker. (2006). Increased capacity for axonal outgrowth using xenogenic tissue in vitro and in a rodent model of Parkinson's disease. Xenotransplantation. 13(3). 233–247. 12 indexed citations
20.
Kuan, Wei‐Li, Rachel Lin, Pam Tyers, & Roger A. Barker. (2006). The importance of A9 dopaminergic neurons in mediating the functional benefits of fetal ventral mesencephalon transplants and levodopa-induced dyskinesias. Neurobiology of Disease. 25(3). 594–608. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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