W. King Engel

12.2k total citations · 2 hit papers
195 papers, 9.1k citations indexed

About

W. King Engel is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, W. King Engel has authored 195 papers receiving a total of 9.1k indexed citations (citations by other indexed papers that have themselves been cited), including 98 papers in Molecular Biology, 54 papers in Cellular and Molecular Neuroscience and 48 papers in Neurology. Recurrent topics in W. King Engel's work include Muscle Physiology and Disorders (48 papers), Genetic Neurodegenerative Diseases (31 papers) and Ion channel regulation and function (26 papers). W. King Engel is often cited by papers focused on Muscle Physiology and Disorders (48 papers), Genetic Neurodegenerative Diseases (31 papers) and Ion channel regulation and function (26 papers). W. King Engel collaborates with scholars based in United States, Italy and Netherlands. W. King Engel's co-authors include Michael H. Brooke, Valerie Askanas, George Karpati, Felix E. Zajac, Robert E. Burke, David N. Levine, Peter Tsairis, Marinos C. Dalakas, David S. Zee and John R. Warmolts and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

W. King Engel

194 papers receiving 8.1k citations

Hit Papers

Mammalian Motor Units: Physiological-Histochemical Correl... 1963 2026 1984 2005 1971 1963 200 400 600

Peers

W. King Engel
Hannu Kalimo Finland
Arthur P. Hays United States
Michael H. Brooke United States
Daniel B. Drachman United States
Gerta Vrbovà United Kingdom
W. King Engel
Citations per year, relative to W. King Engel W. King Engel (= 1×) peers Stirling Carpenter

Countries citing papers authored by W. King Engel

Since Specialization
Citations

This map shows the geographic impact of W. King Engel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. King Engel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. King Engel more than expected).

Fields of papers citing papers by W. King Engel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. King Engel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. King Engel. The network helps show where W. King Engel may publish in the future.

Co-authorship network of co-authors of W. King Engel

This figure shows the co-authorship network connecting the top 25 collaborators of W. King Engel. A scholar is included among the top collaborators of W. King Engel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. King Engel. W. King Engel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nogalska, Anna, Carla D’Agostino, W. King Engel, & Valerie Askanas. (2014). Sodium phenylbutyrate reverses lysosomal dysfunction and decreases amyloid-β42 in an in vitro-model of inclusion-body myositis. Neurobiology of Disease. 65. 93–101. 10 indexed citations
2.
Askanas, Valerie, W. King Engel, & Anna Nogalska. (2014). Sporadic inclusion-body myositis: A degenerative muscle disease associated with aging, impaired muscle protein homeostasis and abnormal mitophagy. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1852(4). 633–643. 78 indexed citations
3.
Wójcik, Sławomir, Anna Nogalska, W. King Engel, & Valerie Askanas. (2007). G.P.3.16 In skeletal muscle of patients with type-II fiber atrophy, myostatin (MSTN) and myostatin precursor protein (MSTN-PP) are both increased. Neuromuscular Disorders. 17(9-10). 787–787. 1 indexed citations
4.
Fratta, Pietro, et al.. (2003). Novel expression of dysferlin in recently denervated and apoptoid human muscle fibers. UCL Discovery (University College London). 1 indexed citations
5.
Broccolini, Aldobrando, et al.. (2000). Paired Helical Filaments of Inclusion-Body Myositis Muscle Contain RNA and Survival Motor Neuron Protein. American Journal Of Pathology. 156(4). 1151–1155. 22 indexed citations
6.
Askanas, Valerie, Renate B. Alvarez, & W. King Engel. (1993). β‐Amyloid precursor epitopes in muscle fibers of inclusion body myositis. Annals of Neurology. 34(4). 551–560. 121 indexed citations
7.
Braun, Serge, Valerie Askanas, W. King Engel, & Emad Ibrahim. (1993). Long‐Term Treatment with Glucocorticoids Increases Synthesis and Stability of Junctional Acetylcholine Receptors on Innervated Cultured Human Muscle. Journal of Neurochemistry. 60(5). 1929–1935. 24 indexed citations
8.
Martinuzzi, Andrea, Lodovica Vergani, Rosalba Carrozzo, et al.. (1993). Expression of muscle-type phosphorylase in innervated and aneural cultured muscle of patients with myophosphorylase deficiency.. Journal of Clinical Investigation. 92(4). 1774–1780. 13 indexed citations
9.
Askanas, Valerie, et al.. (1993). Prion protein is abnormally accumulated in inclusion-body myositis. Neuroreport. 5(1). 25–28. 49 indexed citations
10.
Askanas, Valerie, et al.. (1992). Strong immunoreactivity of β-amyloid precursor protein, including the β-amyloid protein sequence, at human neuromuscular junctions. Neuroscience Letters. 143(1-2). 96–100. 51 indexed citations
11.
Vita, Giuseppe, et al.. (1991). Down-regulation of thyrotropin-releasing hormone (TRH) receptors in spinal cord after transection as revealed by quantitative autoradiography. Experimental Brain Research. 83(2). 381–4. 3 indexed citations
12.
Martinuzzi, Andrea, Valerie Askanas, & W. King Engel. (1990). Paralysis of Innervated Cultured Human Muscle Fibers Affects Enzymes Differentially. Journal of Neurochemistry. 54(1). 223–229. 8 indexed citations
13.
Vita, Giuseppe, et al.. (1990). Effects of experimental spinal cord transection on substance P receptors: A quantitative autoradiography study. Neuropeptides. 17(3). 147–153. 17 indexed citations
14.
Tomé, F.M.S., et al.. (1989). Nuclear inclusions in innervated cultured muscle fibers from patients with oculopharyngeal muscular dystrophy. Neurology. 39(7). 926–926. 21 indexed citations
15.
Nau, H.-E., et al.. (1988). Zum Wert evozierter Potentiale auf der Neurochirurgischen Intensivstation. min - Minimally Invasive Neurosurgery. 31(S 1). 170–174. 6 indexed citations
16.
Vita, Giuseppe, Valerie Askanas, Andrea Martinuzzi, & W. King Engel. (1988). Histoenzymatic profile of human muscle cultured in monolayer and innervated de novo by fetal rat spinal cord. Muscle & Nerve. 11(1). 1–9. 24 indexed citations
17.
Martinuzzi, Andrea, Valerie Askanas, Takayoshi Kobayashi, & W. King Engel. (1988). Asynchronous regulation of muscle specific isozymes of creatine kinase, glycogen phosphorylase, lactic dehydrogenase and phosphoglycerate mutase in innervated and non-innervated cultured human muscle. Neuroscience Letters. 89(2). 216–222. 31 indexed citations
18.
Hawkins, Edward F. & W. King Engel. (1987). Kinetic analysis of thyrotropin-releasing hormone binding in the central nervous system: evidence for receptor desensitization. Neuroscience Letters. 79(1-2). 157–162. 9 indexed citations
19.
Engel, W. King, et al.. (1978). Ageing of the female gamete. 2 Lactate dehydrogenase activity in fragmented oocytes and ova of the rat.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 18(71-72). 151–6. 6 indexed citations
20.
Engel, W. King, et al.. (1966). Reversal of the ATPase Reaction in Muscle Fibres By EDTA. Nature. 212(5070). 1551–1553. 47 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026