Vicente Andrés
- Molecular Biology top 1%
- Immunology top 1%
- Physiology top 1%
- Oncology top 2%
- Cardiology and Cardiovascular Medicine top 2%
- Co-authors
- Kenneth WalshJosé J. FusterJosé María GonzálezMagda R. HamczykAntonio Díez‐JuanKun GuoValentı́n FusterVijak Mahdavi
- Topics
- Nuclear Structure and Function (41 papers)RNA Research and Splicing (37 papers)Atherosclerosis and Cardiovascular Diseases (28 papers)
- Cited by
- AgingImmunologyMolecular Biology
- Partner nations
- SpainUnited StatesUnited Kingdom
In The Last Decade
Vicente Andrés
183 papers receiving 9.6k citations
Hit Papers
Peers
Comparison fields: 5 of 160
- Molecular Biology 5.5k
- Immunology 1.7k
- Physiology 1.3k
- Oncology 1.1k
- Cardiology and Cardiovascular Medicine 1.1k
Countries citing papers authored by Vicente Andrés
This map shows the geographic impact of Vicente Andrés's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vicente Andrés with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vicente Andrés more than expected).
Fields of papers citing papers by Vicente Andrés
This network shows the impact of papers produced by Vicente Andrés. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vicente Andrés. The network helps show where Vicente Andrés may publish in the future.
Co-authorship network of co-authors of Vicente Andrés
This figure shows the co-authorship network connecting the top 25 collaborators of Vicente Andrés. A scholar is included among the top collaborators of Vicente Andrés based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vicente Andrés. Vicente Andrés is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 8 | |
| 3 | 8 | |
| 4 | 12 | |
| 5 | 1 | |
| 6 | 48 | |
| 7 | 11 | |
| 8 | 4 | |
| 9 | 35 | |
| 10 | 29 | |
| 11 | 25 | |
| 12 | 16 | |
| 13 | 27 | |
| 14 | 5 | |
| 15 | Clonal hematopoiesis associated with TET2 deficiency accelerates atherosclerosis development in micebreakdown → | 978 |
| 16 | 8 | |
| 17 | 55 | |
| 18 | 0 | |
| 19 | 43 | |
| 20 | 66 |
About Vicente Andrés
Vicente Andrés is a scholar working on Aging, Immunology and Allergy and Immunology, having authored 185 papers that have together received 9.7k indexed citations. Recurring topics across this work include Nuclear Structure and Function (41 papers), RNA Research and Splicing (37 papers) and Atherosclerosis and Cardiovascular Diseases (28 papers). The work is most often cited by research in Aging (348 citations), Immunology (1.7k citations) and Molecular Biology (5.5k citations). Vicente Andrés has collaborated with scholars based in Spain, United States and United Kingdom. Frequent co-authors include Kenneth Walsh, José J. Fuster, José María González, Magda R. Hamczyk, Antonio Díez‐Juan, Kun Guo, Valentı́n Fuster, Vijak Mahdavi, Carlos López-Otı́n and Beatriz Dorado. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.