Veerle Reynders

601 total citations
9 papers, 511 citations indexed

About

Veerle Reynders is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Veerle Reynders has authored 9 papers receiving a total of 511 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Cancer Research. Recurrent topics in Veerle Reynders's work include Drug Transport and Resistance Mechanisms (3 papers), Peroxisome Proliferator-Activated Receptors (3 papers) and NF-κB Signaling Pathways (3 papers). Veerle Reynders is often cited by papers focused on Drug Transport and Resistance Mechanisms (3 papers), Peroxisome Proliferator-Activated Receptors (3 papers) and NF-κB Signaling Pathways (3 papers). Veerle Reynders collaborates with scholars based in Germany and Belgium. Veerle Reynders's co-authors include Markus S. Schwab, Jürgen M. Stein, Oliver Schröder, Stefan Loitsch, Dieter Steinhilber, Lieven Dupont, Robin Vos, Geert M. Verleden, Dirk Van Raemdonck and Dominique Bullens and has published in prestigious journals such as European Respiratory Journal, Carcinogenesis and Cancer Letters.

In The Last Decade

Veerle Reynders

9 papers receiving 504 citations

Peers

Veerle Reynders

MB

SURGE

IMMUN

PRM

MICRO

Chengfei Wu China

Gabriele Hölzlwimmer Germany

Andrea McDowell South Korea

Natália Jaeger United States

John P. Sotolongo United States

M. Brett Everhart United States

Toshiyuki Fukutomi Japan

Kelley M. Bradley United States

F Herweck Germany

Chengfei Wu China

Veerle Reynders

287 ×1.4

MB

143 ×1.2

SURGE

74 ×0.7

IMMUN

44 ×0.5

PRM

31 ×0.4

MICRO

Citations per year, relative to Veerle Reynders Veerle Reynders (= 1×) peers Chengfei Wu

Countries citing papers authored by Veerle Reynders

Since Specialization

Citations

This map shows the geographic impact of Veerle Reynders's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Veerle Reynders with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Veerle Reynders more than expected).

Fields of papers citing papers by Veerle Reynders

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Veerle Reynders. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Veerle Reynders. The network helps show where Veerle Reynders may publish in the future.

Co-authorship network of co-authors of Veerle Reynders

This figure shows the co-authorship network connecting the top 25 collaborators of Veerle Reynders. A scholar is included among the top collaborators of Veerle Reynders based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Veerle Reynders. Veerle Reynders is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Vanaudenaerde, Bart, Stéphanie I. De Vleeschauwer, Robin Vos, et al.. (2008). The Role of the IL23/IL17 Axis in Bronchiolitis Obliterans Syndrome After Lung Transplantation. American Journal of Transplantation. 8(9). 1911–1920. 143 indexed citations

2.

Schwab, Markus S., Veerle Reynders, Dieter Steinhilber, & Jürgen M. Stein. (2008). Combined treatment of Caco-2 cells with butyrate and mesalazine inhibits cell proliferation and reduces Survivin protein level. Cancer Letters. 273(1). 98–106. 10 indexed citations

3.

Schwab, Markus S., Veerle Reynders, Stefan Loitsch, et al.. (2008). The dietary histone deacetylase inhibitor sulforaphane induces human β‐defensin‐2 in intestinal epithelial cells. Immunology. 125(2). 241–251. 60 indexed citations

4.

Schwab, Markus S., Veerle Reynders, Stefan Loitsch, et al.. (2008). PPAR is involved in mesalazine-mediated induction of apoptosis and inhibition of cell growth in colon cancer cells. Carcinogenesis. 29(7). 1407–1414. 52 indexed citations

5.

Schwab, Markus S., Veerle Reynders, Stefan Loitsch, et al.. (2007). Involvement of different nuclear hormone receptors in butyrate-mediated inhibition of inducible NFκB signalling. Molecular Immunology. 44(15). 3625–3632. 116 indexed citations

6.

Schwab, Markus S., Veerle Reynders, Sandra Ulrich, et al.. (2006). PPARγ is a key target of butyrate-induced caspase-3 activation in the colorectal cancer cell line Caco-2. APOPTOSIS. 11(10). 1801–1811. 50 indexed citations

7.

Reynders, Veerle, et al.. (2006). Peroxisome Proliferator-Activated Receptor α (PPARα) down-regulation in cystic fibrosis lymphocytes. Respiratory Research. 7(1). 104–104. 24 indexed citations

8.

Schwab, Markus S., et al.. (2006). Role of nuclear hormone receptors in butyrate-mediated up-regulation of the antimicrobial peptide cathelicidin in epithelial colorectal cells. Molecular Immunology. 44(8). 2107–2114. 52 indexed citations

9.

Reynders, Veerle, Éric Marchand, Ghislaine Gayan‐Ramirez, et al.. (2002). Alterations in gene expression in hamster diaphragm after emphysema and lung volume reduction surgery. European Respiratory Journal. 19(6). 1064–1071. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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