Markus S. Schwab

2.5k total citations
20 papers, 1.2k citations indexed

About

Markus S. Schwab is a scholar working on Molecular Biology, Cell Biology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Markus S. Schwab has authored 20 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Cell Biology and 7 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Markus S. Schwab's work include Reproductive Biology and Fertility (7 papers), Microtubule and mitosis dynamics (7 papers) and Genetics, Aging, and Longevity in Model Organisms (3 papers). Markus S. Schwab is often cited by papers focused on Reproductive Biology and Fertility (7 papers), Microtubule and mitosis dynamics (7 papers) and Genetics, Aging, and Longevity in Model Organisms (3 papers). Markus S. Schwab collaborates with scholars based in Germany, United States and Switzerland. Markus S. Schwab's co-authors include James L. Maller, Stefan Groß, Andrea L. Lewellyn, Frédéric Taïeb, Jürgen M. Stein, Veerle Reynders, Oliver Schröder, Stefan Loitsch, Brian Tunquist and Dieter Steinhilber and has published in prestigious journals such as Science, Molecular and Cellular Biology and Current Biology.

In The Last Decade

Markus S. Schwab

20 papers receiving 1.2k citations

Peers

Markus S. Schwab
K. Sadhu United States
Ian Blench United Kingdom
Nelson Hsia United States
Mustafa Ünlü United States
A K Erickson United States
James Flynn United States
K. Sadhu United States
Markus S. Schwab
Citations per year, relative to Markus S. Schwab Markus S. Schwab (= 1×) peers K. Sadhu

Countries citing papers authored by Markus S. Schwab

Since Specialization
Citations

This map shows the geographic impact of Markus S. Schwab's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Markus S. Schwab with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Markus S. Schwab more than expected).

Fields of papers citing papers by Markus S. Schwab

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Markus S. Schwab. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Markus S. Schwab. The network helps show where Markus S. Schwab may publish in the future.

Co-authorship network of co-authors of Markus S. Schwab

This figure shows the co-authorship network connecting the top 25 collaborators of Markus S. Schwab. A scholar is included among the top collaborators of Markus S. Schwab based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Markus S. Schwab. Markus S. Schwab is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Weston, Anna, et al.. (2016). Yeast-based assays for screening 11β-HSD1 inhibitors. Microbial Cell Factories. 15(1). 52–52. 1 indexed citations
2.
Schwab, Markus S., Veerle Reynders, Dieter Steinhilber, & Jürgen M. Stein. (2008). Combined treatment of Caco-2 cells with butyrate and mesalazine inhibits cell proliferation and reduces Survivin protein level. Cancer Letters. 273(1). 98–106. 10 indexed citations
3.
Schwab, Markus S., Veerle Reynders, Stefan Loitsch, et al.. (2008). The dietary histone deacetylase inhibitor sulforaphane induces human β‐defensin‐2 in intestinal epithelial cells. Immunology. 125(2). 241–251. 60 indexed citations
4.
Schwab, Markus S., Veerle Reynders, Stefan Loitsch, et al.. (2008). PPAR  is involved in mesalazine-mediated induction of apoptosis and inhibition of cell growth in colon cancer cells. Carcinogenesis. 29(7). 1407–1414. 52 indexed citations
5.
Schwab, Markus S., Veerle Reynders, Stefan Loitsch, et al.. (2007). Involvement of different nuclear hormone receptors in butyrate-mediated inhibition of inducible NFκB signalling. Molecular Immunology. 44(15). 3625–3632. 116 indexed citations
6.
Schwab, Markus S., et al.. (2007). A Concise Synthesis of the Pennsylvania Green Fluorophore and Labeling of Intracellular Targets with O6-Benzylguanine Derivatives. Organic Letters. 9(19). 3741–3744. 34 indexed citations
7.
Schwab, Markus S., Veerle Reynders, Sandra Ulrich, et al.. (2006). PPARγ is a key target of butyrate-induced caspase-3 activation in the colorectal cancer cell line Caco-2. APOPTOSIS. 11(10). 1801–1811. 50 indexed citations
8.
Schwab, Markus S., et al.. (2006). Role of nuclear hormone receptors in butyrate-mediated up-regulation of the antimicrobial peptide cathelicidin in epithelial colorectal cells. Molecular Immunology. 44(8). 2107–2114. 52 indexed citations
9.
Dinarina, Ana, et al.. (2005). Characterization of a new family of cyclin-dependent kinase activators. Biochemical Journal. 386(2). 349–355. 46 indexed citations
10.
Reina, José, Emmanuel Lacroix, Gregorio Fernández‐Ballester, et al.. (2002). Computer-aided design of a PDZ domain to recognize new target sequences. Nature Structural Biology. 9(8). 621–7. 132 indexed citations
11.
Maller, James L., Markus S. Schwab, Stefan Groß, et al.. (2002). The mechanism of CSF arrest in vertebrate oocytes. Molecular and Cellular Endocrinology. 187(1-2). 173–178. 31 indexed citations
12.
Tunquist, Brian, et al.. (2002). The Spindle Checkpoint Kinase Bub1 and Cyclin E/Cdk2 Both Contribute to the Establishment of Meiotic Metaphase Arrest by Cytostatic Factor. Current Biology. 12(12). 1027–1033. 76 indexed citations
13.
Maller, James L., Markus S. Schwab, B.Tibor Roberts, et al.. (2001). The pathway of MAP kinase mediation of CSF arrest in Xenopus oocytes. Biology of the Cell. 93(1-2). 27–33. 22 indexed citations
14.
Maller, James L., et al.. (2001). Cell Cycle Transitions in Early Xenopus Development. Novartis Foundation symposium. 237. 58–78. 17 indexed citations
15.
Schwab, Markus S., B.Tibor Roberts, Stefan Groß, et al.. (2001). Bub1 is activated by the protein kinase p90 Rsk during Xenopus oocyte maturation. Current Biology. 11(3). 141–150. 87 indexed citations
16.
Groß, Stefan, Markus S. Schwab, Frédéric Taïeb, et al.. (2000). The critical role of the MAP kinase pathway in meiosis II in Xenopus oocytes is mediated by p90Rsk. Current Biology. 10(8). 430–438. 199 indexed citations
17.
Schwab, Markus S., Sang Ho Kim, Naohiro Terada, et al.. (1999). p70 S6K Controls Selective mRNA Translation during Oocyte Maturation and Early Embryogenesis in Xenopus laevis. Molecular and Cellular Biology. 19(4). 2485–2494. 51 indexed citations
18.
Groß, Stefan, Markus S. Schwab, Andrea L. Lewellyn, & James L. Maller. (1999). Induction of Metaphase Arrest in Cleaving Xenopus Embryos by the Protein Kinase p90 Rsk. Science. 286(5443). 1365–1367. 127 indexed citations
19.
Schwab, Markus S., et al.. (1998). Subcellular Distribution of Distinct Nucleolin Subfractions Recognized by Two Monoclonal Antibodies. Experimental Cell Research. 239(2). 226–234. 12 indexed citations
20.
Schwab, Markus S. & Chantal Dreyer. (1997). Protein phosphorylation sites regulate the function of the bipartite NLS of nucleolin.. PubMed. 73(4). 287–97. 64 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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