Vanesa Santos

1.5k total citations
18 papers, 1.0k citations indexed

About

Vanesa Santos is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Vanesa Santos has authored 18 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 7 papers in Oncology and 2 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Vanesa Santos's work include Microbial metabolism and enzyme function (8 papers), Cancer Cells and Metastasis (5 papers) and Kruppel-like factors research (3 papers). Vanesa Santos is often cited by papers focused on Microbial metabolism and enzyme function (8 papers), Cancer Cells and Metastasis (5 papers) and Kruppel-like factors research (3 papers). Vanesa Santos collaborates with scholars based in Spain, United States and France. Vanesa Santos's co-authors include Amparo Cano, Francisco Portillo, Gema Moreno‐Bueno, Héctor Peinado, Eva P. Cuevas, José Palacios, David Olmeda, Saleta Morales, Eva Cubillo and David Hardisson and has published in prestigious journals such as Nature Communications, The EMBO Journal and PLoS ONE.

In The Last Decade

Vanesa Santos

18 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Vanesa Santos Spain 12 800 308 225 173 130 18 1.0k
Dai Nakashima Japan 22 832 1.0× 346 1.1× 328 1.5× 145 0.8× 178 1.4× 49 1.2k
Jessica Lo Hong Kong 11 506 0.6× 343 1.1× 373 1.7× 278 1.6× 81 0.6× 16 1.0k
Helen Sadik United States 11 410 0.5× 265 0.9× 290 1.3× 109 0.6× 44 0.3× 11 705
Aline Meulle France 9 671 0.8× 553 1.8× 607 2.7× 175 1.0× 76 0.6× 12 1.4k
Raphaël Leblanc France 14 535 0.7× 290 0.9× 175 0.8× 119 0.7× 172 1.3× 22 819
Veronica Catalano Italy 11 657 0.8× 694 2.3× 337 1.5× 239 1.4× 152 1.2× 11 1.3k
Cuiju Tang China 14 382 0.5× 340 1.1× 178 0.8× 96 0.6× 99 0.8× 47 853
Yingduan Cheng China 19 815 1.0× 171 0.6× 386 1.7× 73 0.4× 75 0.6× 33 1.1k
Tieju Liu China 20 764 1.0× 340 1.1× 451 2.0× 78 0.5× 129 1.0× 38 1.0k
Yosuke Endo‐Sakamoto Japan 17 456 0.6× 173 0.6× 203 0.9× 80 0.5× 81 0.6× 37 634

Countries citing papers authored by Vanesa Santos

Since Specialization
Citations

This map shows the geographic impact of Vanesa Santos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vanesa Santos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vanesa Santos more than expected).

Fields of papers citing papers by Vanesa Santos

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vanesa Santos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vanesa Santos. The network helps show where Vanesa Santos may publish in the future.

Co-authorship network of co-authors of Vanesa Santos

This figure shows the co-authorship network connecting the top 25 collaborators of Vanesa Santos. A scholar is included among the top collaborators of Vanesa Santos based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vanesa Santos. Vanesa Santos is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Hergueta‐Redondo, Marta, Sara Sánchez‐Redondo, Begoña Hurtado, et al.. (2025). The impact of a high fat diet and platelet activation on pre-metastatic niche formation. Nature Communications. 16(1). 2897–2897. 3 indexed citations
2.
Lucas‐Ruiz, Fernando, Felipe Alconchel, L. Martínez‐Alarcón, et al.. (2024). Cell-specific Extracellular Vesicles and Their miRNA Cargo Released Into the Organ Preservation Solution During Cold Ischemia Storage as Biomarkers for Liver Transplant Outcomes. Transplantation. 108(10). e301–e312. 2 indexed citations
3.
Santos, Vanesa, Marina S. Mazariegos, Eduardo Caleiras, et al.. (2024). NGFR regulates stromal cell activation in germinal centers. Cell Reports. 43(2). 113705–113705. 3 indexed citations
4.
López‐Menéndez, Celia, Vanesa Santos, Pierre Dubus, et al.. (2021). E2A Modulates Stemness, Metastasis, and Therapeutic Resistance of Breast Cancer. Cancer Research. 81(17). 4529–4544. 20 indexed citations
5.
Cano, Amparo, Cyrill Géraud, Kalle Sipilä, et al.. (2018). Loxl2 is dispensable for dermal development, homeostasis and tumour stroma formation. PLoS ONE. 13(6). e0199679–e0199679. 8 indexed citations
6.
Salvador, Fernando, Alberto Martín, Celia López‐Menéndez, et al.. (2017). Lysyl Oxidase–like Protein LOXL2 Promotes Lung Metastasis of Breast Cancer. Cancer Research. 77(21). 5846–5859. 116 indexed citations
7.
Cuevas, Eva P., Pilar Eraso, Marı́a J. Mazón, et al.. (2017). LOXL2 drives epithelial-mesenchymal transition via activation of IRE1-XBP1 signalling pathway. Scientific Reports. 7(1). 44988–44988. 87 indexed citations
8.
Santamarı́a, Patricia G., Alfredo Floristán, Bárbara Fontanals-Cirera, et al.. (2017). Lysyl oxidase-like 3 is required for melanoma cell survival by maintaining genomic stability. Cell Death and Differentiation. 25(5). 935–950. 42 indexed citations
9.
Martín, Alberto, Fernando Salvador, Gema Moreno‐Bueno, et al.. (2015). Lysyl oxidase‐like 2 represses Notch1 expression in the skin to promote squamous cell carcinoma progression. The EMBO Journal. 34(8). 1090–1109. 74 indexed citations
10.
Moreno‐Bueno, Gema, Saleta Morales, Vanesa Santos, et al.. (2015). Loss of Snail2 favors skin tumor progression by promoting the recruitment of myeloid progenitors. Carcinogenesis. 36(5). 585–597. 4 indexed citations
11.
Díaz‐López, Antonio, Juan Díaz‐Martín, Gema Moreno‐Bueno, et al.. (2014). Zeb1 and Snail1 engage miR‐200f transcriptional and epigenetic regulation during EMT. International Journal of Cancer. 136(4). E62–73. 54 indexed citations
12.
Cuevas, Eva P., Gema Moreno‐Bueno, Giacomo Canesin, et al.. (2014). LOXL2 catalytically inactive mutants mediate epithelial-to-mesenchymal transition. Biology Open. 3(2). 129–137. 53 indexed citations
13.
Cubillo, Eva, Antonio Díaz‐López, Eva P. Cuevas, et al.. (2013). E47 and Id1 Interplay in Epithelial-Mesenchymal Transition. PLoS ONE. 8(3). e59948–e59948. 45 indexed citations
15.
Moreno‐Bueno, Gema, Fernando Salvador, Alberto Martín, et al.. (2011). Lysyl oxidase‐like 2 (LOXL2), a new regulator of cell polarity required for metastatic dissemination of basal‐like breast carcinomas. EMBO Molecular Medicine. 3(9). 528–544. 136 indexed citations
16.
Moreno‐Bueno, Gema, Héctor Peinado, Patricia Molina, et al.. (2009). The morphological and molecular features of the epithelial-to-mesenchymal transition. Nature Protocols. 4(11). 1591–1613. 179 indexed citations
17.
Peinado, Héctor, Gema Moreno‐Bueno, David Hardisson, et al.. (2008). Lysyl Oxidase–Like 2 as a New Poor Prognosis Marker of Squamous Cell Carcinomas. Cancer Research. 68(12). 4541–4550. 167 indexed citations
18.
Souza, Gustavo A. de, et al.. (1998). Proteolytic effets of Loxoceles intermedia (brown spider) venom proteins on EHS-basemant membrane structures. Acta Biológica Paranaense. 27. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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