V. Poulaki
- Molecular Biology top 10%
- Oncology top 10%
- Cancer Research top 10%
- Ophthalmology top 2%
- Immunology
- Co-authors
- Nicholas MitsiadesMaria TsokosIvan StamenkovicWei‐Hsuan YuAnthony P. AdamisC MitsiadesDemetrios A. KoutrasYuichi Kaji
- Topics
- Cell death mechanisms and regulation (7 papers)Retinal Diseases and Treatments (6 papers)Cancer-related Molecular Pathways (6 papers)
- Partner nations
- United StatesGermanyGreece
In The Last Decade
V. Poulaki
34 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 91
- Molecular Biology 735
- Oncology 300
- Cancer Research 291
- Ophthalmology 277
- Immunology 197
Countries citing papers authored by V. Poulaki
This map shows the geographic impact of V. Poulaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by V. Poulaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites V. Poulaki more than expected).
Fields of papers citing papers by V. Poulaki
This network shows the impact of papers produced by V. Poulaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by V. Poulaki. The network helps show where V. Poulaki may publish in the future.
Co-authorship network of co-authors of V. Poulaki
This figure shows the co-authorship network connecting the top 25 collaborators of V. Poulaki. A scholar is included among the top collaborators of V. Poulaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with V. Poulaki. V. Poulaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 2 | |
| 3 | 8 | |
| 4 | 9 | |
| 5 | 10 | |
| 6 | TIMP3 Inhibits Vascular Leakage and Leukostasis in an Animal Model of Streptozotocin-Induced Diabetes | 1 |
| 7 | 1 | |
| 8 | Intensive insulin therapy–induced vascular leakage and leukostasis in diabetes are decreased by anti–IGF | 1 |
| 9 | Reduced Histopathological Alterations in Long-Term Diabetic TNF-R Deficient Mice | 3 |
| 10 | 36 | |
| 11 | Suppression of Diabetic Retinopathy with Angiopoietin-1 | 2 |
| 12 | Non-steroid Alanti-inflammatory Drugs Prevent Early Diabetic Retinopathy: Aspirinand COX-2 Inhibition Prevent Blood-retinal Barrier Breakdown and Leukocyte Adhesion Via TNF-asuppression | 2 |
| 13 | A Modulatory Role for Advanced Glycation End-Products (AGEs) in Retinal Microvascular Leukostasis | 3 |
| 14 | The TNF-Inhibitor Enbrel prevents early diabetic retinal changes in vivo | 1 |
| 15 | 48 | |
| 16 | Matrix metalloproteinase-7-mediated cleavage of Fas ligand protects tumor cells from chemotherapeutic drug cytotoxicity. | 302 |
| 17 | 38 | |
| 18 | Inhibition of inflammatory corneal angiogenesis by TNP-470. | 40 |
| 19 | 20 | |
| 20 | 45 |
About V. Poulaki
V. Poulaki is a scholar working on Ophthalmology, Cancer Research and Clinical Biochemistry, having authored 34 papers that have together received 1.3k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (7 papers), Retinal Diseases and Treatments (6 papers) and Cancer-related Molecular Pathways (6 papers). The work is most often cited by research in Ophthalmology (277 citations), Cancer Research (291 citations) and Clinical Biochemistry (121 citations). V. Poulaki has collaborated with scholars based in United States, Germany and Greece. Frequent co-authors include Nicholas Mitsiades, Maria Tsokos, Ivan Stamenkovic, Wei‐Hsuan Yu, Anthony P. Adamis, C Mitsiades, Demetrios A. Koutras, Yuichi Kaji, Jonathan E. Moore and Tomohiko Usui. Their work appears in journals such as JNCI Journal of the National Cancer Institute, Oncogene and The FASEB Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.