Uwe Vinkemeier
- Oncology top 1%
- Molecular Biology top 5%
- Immunology top 1%
- Cancer Research top 5%
- Pathology and Forensic Medicine top 5%
- Co-authors
- James DarnellThomas MeyerJohn KuriyanAndreas BegittXiaohong ChenIsmail MoarefiYanxiang ZhaoDavid Jeruzalmi
- Topics
- Cytokine Signaling Pathways and Interactions (33 papers)RNA Research and Splicing (18 papers)interferon and immune responses (16 papers)
- Cited by
- OncologyImmunologyCancer Research
- Partner nations
- GermanyUnited KingdomUnited States
In The Last Decade
Uwe Vinkemeier
44 papers receiving 3.6k citations
Hit Papers
Peers
Comparison fields: 5 of 98
- Oncology 2.2k
- Molecular Biology 1.9k
- Immunology 1.7k
- Cancer Research 471
- Pathology and Forensic Medicine 269
Countries citing papers authored by Uwe Vinkemeier
This map shows the geographic impact of Uwe Vinkemeier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Uwe Vinkemeier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Uwe Vinkemeier more than expected).
Fields of papers citing papers by Uwe Vinkemeier
This network shows the impact of papers produced by Uwe Vinkemeier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Uwe Vinkemeier. The network helps show where Uwe Vinkemeier may publish in the future.
Co-authorship network of co-authors of Uwe Vinkemeier
This figure shows the co-authorship network connecting the top 25 collaborators of Uwe Vinkemeier. A scholar is included among the top collaborators of Uwe Vinkemeier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Uwe Vinkemeier. Uwe Vinkemeier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 4 | |
| 3 | 9 | |
| 4 | 16 | |
| 5 | 1 | |
| 6 | 42 | |
| 7 | 34 | |
| 8 | 66 | |
| 9 | 7 | |
| 10 | 26 | |
| 11 | 23 | |
| 12 | 49 | |
| 13 | 70 | |
| 14 | 64 | |
| 15 | 13 | |
| 16 | 99 | |
| 17 | 126 | |
| 18 | 78 | |
| 19 | Crystal Structure of a Tyrosine Phosphorylated STAT-1 Dimer Bound to DNAbreakdown → | 571 |
| 20 | Titin associated proteins intracellular members of the immunoglobulin superfamily | 1 |
About Uwe Vinkemeier
Uwe Vinkemeier is a scholar working on Oncology, Immunology and Aging, having authored 44 papers that have together received 3.7k indexed citations. Recurring topics across this work include Cytokine Signaling Pathways and Interactions (33 papers), RNA Research and Splicing (18 papers) and interferon and immune responses (16 papers). The work is most often cited by research in Oncology (2.2k citations), Immunology (1.7k citations) and Cancer Research (471 citations). Uwe Vinkemeier has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include James Darnell, Thomas Meyer, John Kuriyan, Andreas Begitt, Xiaohong Chen, Ismail Moarefi, Yanxiang Zhao, David Jeruzalmi, Andreas Marg and Dieter O. Fürst. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.