Thomas Meyer

5.1k total citations
144 papers, 3.5k citations indexed

About

Thomas Meyer is a scholar working on Cardiology and Cardiovascular Medicine, Oncology and Immunology. According to data from OpenAlex, Thomas Meyer has authored 144 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 57 papers in Cardiology and Cardiovascular Medicine, 33 papers in Oncology and 29 papers in Immunology. Recurrent topics in Thomas Meyer's work include Cytokine Signaling Pathways and Interactions (30 papers), Cardiac Health and Mental Health (19 papers) and Acute Myocardial Infarction Research (13 papers). Thomas Meyer is often cited by papers focused on Cytokine Signaling Pathways and Interactions (30 papers), Cardiac Health and Mental Health (19 papers) and Acute Myocardial Infarction Research (13 papers). Thomas Meyer collaborates with scholars based in Germany, United States and United Kingdom. Thomas Meyer's co-authors include Uwe Vinkemeier, Christoph Herrmann‐Lingen, Andreas Begitt, Arnd B. Buchwald, Lutz Binder, Andreas Marg, Hilmar Luthe, Ulrich Meyer, Inga Lödige and Aribert Rothenberger and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Experimental Medicine.

In The Last Decade

Thomas Meyer

141 papers receiving 3.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Meyer Germany 30 960 920 820 702 457 144 3.5k
Benôıt Marin France 35 630 0.7× 476 0.5× 490 0.6× 301 0.4× 442 1.0× 156 5.5k
W. J. Stevens Belgium 45 283 0.3× 503 0.5× 318 0.4× 1.1k 1.6× 387 0.8× 166 5.9k
Abhijit Chaudhuri United Kingdom 26 251 0.3× 368 0.4× 280 0.3× 386 0.5× 928 2.0× 63 3.9k
Torleiv O. Rognum Norway 45 887 0.9× 2.2k 2.4× 1.5k 1.8× 720 1.0× 653 1.4× 170 7.1k
Jürgen Westermann Germany 42 154 0.2× 691 0.8× 422 0.5× 2.1k 3.0× 306 0.7× 151 4.8k
Richard L. Kradin United States 34 197 0.2× 412 0.4× 465 0.6× 911 1.3× 541 1.2× 165 3.8k
Jeroen Aerssens Belgium 39 430 0.4× 1.4k 1.6× 479 0.6× 125 0.2× 840 1.8× 134 4.9k
Dimitrios Vassilopoulos Greece 42 385 0.4× 1.0k 1.1× 224 0.3× 947 1.3× 1.7k 3.8× 273 6.2k
S. Fredrikson Sweden 45 259 0.3× 738 0.8× 903 1.1× 1.9k 2.8× 457 1.0× 198 7.5k
Marc Tardieu France 49 251 0.3× 1.3k 1.5× 593 0.7× 1.1k 1.6× 1.2k 2.6× 179 8.5k

Countries citing papers authored by Thomas Meyer

Since Specialization
Citations

This map shows the geographic impact of Thomas Meyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Meyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Meyer more than expected).

Fields of papers citing papers by Thomas Meyer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Meyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Meyer. The network helps show where Thomas Meyer may publish in the future.

Co-authorship network of co-authors of Thomas Meyer

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Meyer. A scholar is included among the top collaborators of Thomas Meyer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Meyer. Thomas Meyer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zampar, Silvia, et al.. (2023). A Combination of Caffeine Supplementation and Enriched Environment in an Alzheimer’s Disease Mouse Model. International Journal of Molecular Sciences. 24(3). 2155–2155. 4 indexed citations
2.
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Lühder, Fred, et al.. (2023). Lack of STAT1 co-operative DNA binding protects against adverse cardiac remodelling in acute myocardial infarction. Frontiers in Cardiovascular Medicine. 10. 975012–975012. 1 indexed citations
4.
Wirths, Oliver, et al.. (2023). A novel interface between the N-terminal and coiled-coil domain of STAT1 functions in an auto-inhibitory manner. Cell Communication and Signaling. 21(1). 170–170. 1 indexed citations
5.
Klafki, Hans‐Wolfgang, et al.. (2023). Brain Region-Specific Differences in Amyloid-β Plaque Composition in 5XFAD Mice. Life. 13(4). 1053–1053. 11 indexed citations
6.
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Zampar, Silvia, et al.. (2022). Combined long-term enriched environment and caffeine supplementation improve memory function in C57Bl6 mice. European Archives of Psychiatry and Clinical Neuroscience. 273(1). 269–281. 5 indexed citations
8.
Czesnik, Dirk, et al.. (2021). Men Show Reduced Cardiac Baroreceptor Sensitivity during Modestly Painful Electrical Stimulation of the Forearm: Exploratory Results from a Sham-Controlled Crossover Vagus Nerve Stimulation Study. International Journal of Environmental Research and Public Health. 18(21). 11193–11193. 2 indexed citations
9.
Lawrence, Steven J., Thomas Meyer, Darja Schmidt, et al.. (2020). Broad Antibody and Cellular Immune Response From a Phase 2 Clinical Trial With a Novel Multivalent Poxvirus-Based Respiratory Syncytial Virus Vaccine. The Journal of Infectious Diseases. 223(6). 1062–1072. 46 indexed citations
10.
11.
Meyer, Thomas, et al.. (2016). Posttraumatic Stress Disorder (PTSD) Patients Exhibit a Blunted Parasympathetic Response to an Emotional Stressor. Applied Psychophysiology and Biofeedback. 41(4). 395–404. 16 indexed citations
12.
Meyer, Thomas, et al.. (2016). Cardiovascular reactivity is independently associated with better mental health. Blood Pressure Monitoring. 21(4). 215–223. 2 indexed citations
13.
14.
Herrmann‐Lingen, Christoph, et al.. (2013). A rapid conformational rearrangement of STAT1 dimers is required for termination rather than for amplification of interferon-γ signaling. PubMed. 2(1). e23576–e23576. 4 indexed citations
15.
Herrmann‐Lingen, Christoph, et al.. (2013). Clinically Relevant Dimer Interface Mutants of STAT1 Transcription Factor Exhibit Differential Gene Expression. PLoS ONE. 8(7). e69903–e69903. 16 indexed citations
16.
Pfau, Bastian, et al.. (2013). Stimulation of primary osteoblasts with ATP induces transient vinculin clustering at sites of high intracellular traction force. Journal of Molecular Histology. 45(1). 81–89. 5 indexed citations
17.
Barth, Peter, et al.. (2012). Cell-Type-Specific Expression of STAT Transcription Factors in Tissue Samples from Patients with Lymphocytic Thyroiditis. Endocrine Pathology. 23(3). 141–150. 12 indexed citations
18.
Meyer, Thomas, Karsten Gavénis, & Uwe Vinkemeier. (2002). Cell Type-Specific and Tyrosine Phosphorylation-Independent Nuclear Presence of STAT1 and STAT3. Experimental Cell Research. 272(1). 45–55. 78 indexed citations
19.
Meyer, Thomas, et al.. (2000). Cardiac troponin I elevation in acute pulmonary embolism is associated with right ventricular dysfunction. Journal of the American College of Cardiology. 36(5). 1632–1636. 224 indexed citations
20.
Meyer, Thomas, et al.. (1998). Expression of porcine major histocompatibility antigens in cardiac tissue. Apmis. 106(7-12). 935–940. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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