Tzong‐Hae Lee

5.0k total citations · 1 hit paper
74 papers, 3.6k citations indexed

About

Tzong‐Hae Lee is a scholar working on Infectious Diseases, Hematology and Immunology. According to data from OpenAlex, Tzong‐Hae Lee has authored 74 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Infectious Diseases, 18 papers in Hematology and 16 papers in Immunology. Recurrent topics in Tzong‐Hae Lee's work include Blood groups and transfusion (14 papers), Prenatal Screening and Diagnostics (13 papers) and HIV Research and Treatment (10 papers). Tzong‐Hae Lee is often cited by papers focused on Blood groups and transfusion (14 papers), Prenatal Screening and Diagnostics (13 papers) and HIV Research and Treatment (10 papers). Tzong‐Hae Lee collaborates with scholars based in United States, Canada and Brazil. Tzong‐Hae Lee's co-authors include Michael P. Busch, Joseph M. McCune, Leilani Montalvo, Hitoshi Ohto, Marcus O. Muench, Karen Beckerman, Jeff E. Mold, Douglas F. Nixon, Jakob Michaëlsson and Trevor D. Burt and has published in prestigious journals such as Science, Blood and PLoS ONE.

In The Last Decade

Tzong‐Hae Lee

71 papers receiving 3.5k citations

Hit Papers

Maternal Alloantigens Promote the Development of Toleroge... 2008 2026 2014 2020 2008 200 400 600

Peers

Tzong‐Hae Lee
Comparison fields: 5 of 107
  • Infectious Diseases 1.1k
  • Immunology 1.1k
  • Virology 680
  • Pediatrics, Perinatology and Child Health 660
  • Public Health, Environmental and Occupational Health 655
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Citations per field, relative to Tzong‐Hae Lee
Tzong‐Hae Lee · 1×
Citations per year, relative to Tzong‐Hae Lee
Tzong‐Hae Lee · 1×

Countries citing papers authored by Tzong‐Hae Lee

Since Specialization
Citations

This map shows the geographic impact of Tzong‐Hae Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tzong‐Hae Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tzong‐Hae Lee more than expected).

Fields of papers citing papers by Tzong‐Hae Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tzong‐Hae Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tzong‐Hae Lee. The network helps show where Tzong‐Hae Lee may publish in the future.

Co-authorship network of co-authors of Tzong‐Hae Lee

This figure shows the co-authorship network connecting the top 25 collaborators of Tzong‐Hae Lee. A scholar is included among the top collaborators of Tzong‐Hae Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tzong‐Hae Lee. Tzong‐Hae Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 3
2 8
3 30
4 95
5 48
6 36
7 30
8 195
9
Maternal Alloantigens Promote the Development of Tolerogenic Fetal Regulatory T Cells in Utero breakdown →
625
10 27
11 59
12 12
13 7
14 38
15 5
16 6
17 16
18 241
19 24
20 63

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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