Tristan J. Iseli

1.6k total citations
16 papers, 1.4k citations indexed

About

Tristan J. Iseli is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Surgery. According to data from OpenAlex, Tristan J. Iseli has authored 16 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Endocrinology, Diabetes and Metabolism and 6 papers in Surgery. Recurrent topics in Tristan J. Iseli's work include Metabolism, Diabetes, and Cancer (8 papers), Liver Disease Diagnosis and Treatment (6 papers) and Diabetes Treatment and Management (3 papers). Tristan J. Iseli is often cited by papers focused on Metabolism, Diabetes, and Cancer (8 papers), Liver Disease Diagnosis and Treatment (6 papers) and Diabetes Treatment and Management (3 papers). Tristan J. Iseli collaborates with scholars based in Australia, China and Hong Kong. Tristan J. Iseli's co-authors include Bruce E. Kemp, David Stapleton, Lee A. Witters, Belinda J. Michell, Mark Walter, Frosa Katsis, Bryce J. W. van Denderen, Julian J. Adams, Sid Murthy and Ian G. Jennings and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Diabetologia.

In The Last Decade

Tristan J. Iseli

16 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tristan J. Iseli Australia 15 880 400 389 294 270 16 1.4k
Xiwen Xiong China 18 722 0.8× 231 0.6× 713 1.8× 184 0.6× 248 0.9× 38 1.6k
Sung‐E Choi South Korea 22 694 0.8× 322 0.8× 450 1.2× 247 0.8× 352 1.3× 53 1.6k
Pierre‐Damien Denechaud France 17 1.2k 1.3× 552 1.4× 609 1.6× 384 1.3× 428 1.6× 27 2.0k
Stephen L. Pinkosky United States 15 754 0.9× 617 1.5× 363 0.9× 434 1.5× 240 0.9× 19 1.6k
Masumi Hara Japan 22 747 0.8× 330 0.8× 226 0.6× 237 0.8× 283 1.0× 47 1.4k
Elaine Xu Canada 17 762 0.9× 565 1.4× 399 1.0× 536 1.8× 372 1.4× 22 1.7k
Mara T. Mashek United States 15 653 0.7× 213 0.5× 478 1.2× 135 0.5× 536 2.0× 21 1.4k
Virginia Pardo Spain 15 499 0.6× 156 0.4× 630 1.6× 159 0.5× 183 0.7× 28 1.3k
Jean‐Paul Pégorier France 15 815 0.9× 382 1.0× 277 0.7× 262 0.9× 344 1.3× 31 1.3k
Wenbin Chen China 20 798 0.9× 109 0.3× 402 1.0× 278 0.9× 164 0.6× 57 1.5k

Countries citing papers authored by Tristan J. Iseli

Since Specialization
Citations

This map shows the geographic impact of Tristan J. Iseli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tristan J. Iseli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tristan J. Iseli more than expected).

Fields of papers citing papers by Tristan J. Iseli

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tristan J. Iseli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tristan J. Iseli. The network helps show where Tristan J. Iseli may publish in the future.

Co-authorship network of co-authors of Tristan J. Iseli

This figure shows the co-authorship network connecting the top 25 collaborators of Tristan J. Iseli. A scholar is included among the top collaborators of Tristan J. Iseli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tristan J. Iseli. Tristan J. Iseli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Alzahrani, Badr, Tristan J. Iseli, Mehdi Ramezani–Moghadam, et al.. (2017). The role of AdipoR1 and AdipoR2 in liver fibrosis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864(3). 700–708. 32 indexed citations
2.
Poorten, David van der, et al.. (2016). Midkine Increases Diagnostic Yield in AFP Negative and NASH-Related Hepatocellular Carcinoma. PLoS ONE. 11(5). e0155800–e0155800. 54 indexed citations
3.
Dong, Zhixia, Lin Su, Saeed Esmaili, et al.. (2015). Adiponectin attenuates liver fibrosis by inducing nitric oxide production of hepatic stellate cells. Journal of Molecular Medicine. 93(12). 1327–1339. 48 indexed citations
4.
Ramezani–Moghadam, Mehdi, Jianhua Wang, Tristan J. Iseli, et al.. (2015). Adiponectin Reduces Hepatic Stellate Cell Migration by Promoting Tissue Inhibitor of Metalloproteinase-1 (TIMP-1) Secretion. Journal of Biological Chemistry. 290(9). 5533–5542. 55 indexed citations
5.
Alzahrani, Badr, Tristan J. Iseli, & Lionel Hebbard. (2013). Non-viral causes of liver cancer: Does obesity led inflammation play a role?. Cancer Letters. 345(2). 223–229. 47 indexed citations
6.
Iseli, Tristan J., Nigel Turner, Xiao‐Yi Zeng, et al.. (2013). Activation of AMPK by Bitter Melon Triterpenoids Involves CaMKKβ. PLoS ONE. 8(4). e62309–e62309. 51 indexed citations
7.
Tonks, Katherine, Yi Zhen Ng, Adelle C.F. Coster, et al.. (2013). Impaired Akt phosphorylation in insulin-resistant human muscle is accompanied by selective and heterogeneous downstream defects. Diabetologia. 56(4). 875–885. 87 indexed citations
8.
Lee, Kyoungtae, Tristan J. Iseli, Andrew J. Hoy, et al.. (2013). Compound K modulates fatty acid‐induced lipid droplet formation and expression of proteins involved in lipid metabolism in hepatocytes. Liver International. 33(10). 1583–1593. 25 indexed citations
9.
Ren, Luping, Stanley M. H. Chan, Xiao‐Yi Zeng, et al.. (2012). Differing Endoplasmic Reticulum Stress Response to Excess Lipogenesis versus Lipid Oversupply in Relation to Hepatic Steatosis and Insulin Resistance. PLoS ONE. 7(2). e30816–e30816. 126 indexed citations
10.
Zeng, Xiao‐Yi, Yi‐Ping Wang, James Cantley, et al.. (2012). Oleanolic Acid Reduces Hyperglycemia beyond Treatment Period with Akt/FoxO1-Induced Suppression of Hepatic Gluconeogenesis in Type-2 Diabetic Mice. PLoS ONE. 7(7). e42115–e42115. 59 indexed citations
11.
Brandon, Amanda E., Andrew J. Hoy, Lauren E. Wright, et al.. (2011). The evolution of insulin resistance in muscle of the glucose infused rat. Archives of Biochemistry and Biophysics. 509(2). 133–141. 16 indexed citations
12.
Lü, Qiang, Tristan J. Iseli, Fuzai Yin, et al.. (2010). The relationship between the waist-to-height ratio and glucose and lipid metabolism in Han adolescents. The Indian Journal of Pediatrics. 77(5). 547–550. 4 indexed citations
13.
Scott, John W., Bryce J. W. van Denderen, Sebastian B. Jørgensen, et al.. (2008). Thienopyridone Drugs Are Selective Activators of AMP-Activated Protein Kinase β1-Containing Complexes. Chemistry & Biology. 15(11). 1220–1230. 209 indexed citations
14.
Iseli, Tristan J., Jonathan S. Oakhill, Michael F. Bailey, et al.. (2007). AMP-activated Protein Kinase Subunit Interactions. Journal of Biological Chemistry. 283(8). 4799–4807. 25 indexed citations
15.
Iseli, Tristan J., Mark Walter, Bryce J. W. van Denderen, et al.. (2005). AMP-activated Protein Kinase β Subunit Tethers α and γ Subunits via Its C-terminal Sequence (186–270). Journal of Biological Chemistry. 280(14). 13395–13400. 111 indexed citations
16.
Kemp, Bruce E., David Stapleton, Duncan J. Campbell, et al.. (2003). AMP-activated protein kinase, super metabolic regulator. Biochemical Society Transactions. 31(1). 162–168. 403 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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