Toshihiro Ichiki

9.0k total citations · 1 hit paper
111 papers, 7.4k citations indexed

About

Toshihiro Ichiki is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Toshihiro Ichiki has authored 111 papers receiving a total of 7.4k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Molecular Biology, 43 papers in Cardiology and Cardiovascular Medicine and 22 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Toshihiro Ichiki's work include Renin-Angiotensin System Studies (34 papers), Receptor Mechanisms and Signaling (29 papers) and Hormonal Regulation and Hypertension (20 papers). Toshihiro Ichiki is often cited by papers focused on Renin-Angiotensin System Studies (34 papers), Receptor Mechanisms and Signaling (29 papers) and Hormonal Regulation and Hypertension (20 papers). Toshihiro Ichiki collaborates with scholars based in Japan, United States and China. Toshihiro Ichiki's co-authors include Akira Takeshita, Tadashi Inagami, Hiroaki Shimokawa, Kensuke Egashira, Kotaro Takeda, Tomotake Tokunou, Yuko Funakoshi, Kenji Sunagawa, Naoko Iino and Iekuni Ichikawa and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

Toshihiro Ichiki

109 papers receiving 7.3k citations

Hit Papers

Effects on blood pressure and exploratory behaviour of mi... 1995 2026 2005 2015 1995 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Effects on blood pressure and exploratory behaviour of mice lacking angiotensin II type-2 receptor
    1995 730 citations Toshihiro Ichiki, Patricia A. Labosky et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Toshihiro Ichiki Japan 46 3.5k 3.4k 2.1k 1.1k 842 111 7.4k
Hiroshi Iwao Japan 48 3.2k 0.9× 3.7k 1.1× 1.2k 0.6× 1.1k 1.1× 857 1.0× 249 8.2k
Wolfgang Linz Germany 48 4.6k 1.3× 2.7k 0.8× 1.7k 0.8× 2.0k 1.9× 998 1.2× 151 9.1k
Masaru Iwai Japan 47 2.7k 0.8× 2.2k 0.6× 1.8k 0.8× 931 0.9× 806 1.0× 137 6.1k
Shokei Kim Japan 44 2.6k 0.7× 2.6k 0.8× 1.1k 0.5× 915 0.9× 737 0.9× 119 5.9k
Sven Waßmann Germany 39 2.5k 0.7× 2.0k 0.6× 1.3k 0.6× 1.1k 1.0× 1.6k 1.9× 101 7.0k
Christophe Heymes France 40 2.9k 0.8× 2.6k 0.8× 1.3k 0.6× 1.6k 1.5× 985 1.2× 86 6.2k
Klaus Lindpaintner United States 46 4.1k 1.2× 2.0k 0.6× 2.3k 1.1× 617 0.6× 982 1.2× 138 8.2k
George W. Booz United States 41 2.6k 0.8× 2.5k 0.7× 878 0.4× 613 0.6× 597 0.7× 148 6.1k
Matsuhiko Hayashi Japan 46 1.8k 0.5× 2.5k 0.7× 1.7k 0.8× 582 0.6× 788 0.9× 203 6.1k
Masakazu Kohno Japan 51 4.0k 1.2× 2.8k 0.8× 2.1k 1.0× 2.4k 2.3× 1.4k 1.6× 273 9.1k

Countries citing papers authored by Toshihiro Ichiki

Since Specialization
Citations

This map shows the geographic impact of Toshihiro Ichiki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Toshihiro Ichiki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Toshihiro Ichiki more than expected).

Fields of papers citing papers by Toshihiro Ichiki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Toshihiro Ichiki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Toshihiro Ichiki. The network helps show where Toshihiro Ichiki may publish in the future.

Co-authorship network of co-authors of Toshihiro Ichiki

This figure shows the co-authorship network connecting the top 25 collaborators of Toshihiro Ichiki. A scholar is included among the top collaborators of Toshihiro Ichiki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Toshihiro Ichiki. Toshihiro Ichiki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Watanabe, Takanori, et al.. (2016). A case of coronary subclavian vertebral steal syndrome successfully treated with stenting to the stenosis of left subclavian artery. Journal of Cardiology Cases. 14(5). 157–160. 1 indexed citations
2.
Ichiki, Toshihiro, et al.. (2015). A case of Kounis syndrome associated with transcatheter arterial chemoembolization for hepatocellular carcinoma. Journal of Cardiology Cases. 12(4). 106–109. 3 indexed citations
3.
Ikeda, Jiro, Toshihiro Ichiki, Hiroshi Kojima, et al.. (2015). PPARγ Agonists Attenuate Palmitate-Induced ER Stress through Up-Regulation of SCD-1 in Macrophages. PLoS ONE. 10(6). e0128546–e0128546. 45 indexed citations
4.
Ichiki, Toshihiro & Kenji Sunagawa. (2014). Novel roles of hypoxia response system in glucose metabolism and obesity. Trends in Cardiovascular Medicine. 24(5). 197–201. 30 indexed citations
5.
Ichiki, Toshihiro. (2013). Regulation of Angiotensin II Receptor Expression. Current Pharmaceutical Design. 19(17). 3013–3021. 9 indexed citations
6.
Takeda, Kotaro, Toshihiro Ichiki, Keita Inanaga, et al.. (2009). Inhibition of Prolyl Hydroxylase Domain-Containing Protein Suppressed Lipopolysaccharide-Induced TNF-α Expression. Arteriosclerosis Thrombosis and Vascular Biology. 29(12). 2132–2137. 59 indexed citations
7.
Ichiki, Toshihiro, Ryohei Miyazaki, Keita Inanaga, et al.. (2007). Inducible cAMP Early Repressor Inhibits Growth of Vascular Smooth Muscle Cell. Arteriosclerosis Thrombosis and Vascular Biology. 27(7). 1549–1555. 14 indexed citations
8.
Hiroki, Junko, Hiroaki Shimokawa, Midoriko Higashi, et al.. (2004). Inflammatory stimuli upregulate Rho-kinase in human coronary vascular smooth muscle cells. Journal of Molecular and Cellular Cardiology. 37(2). 537–546. 133 indexed citations
9.
Hiroki, Junko, Hiroaki Shimokawa, Midoriko Higashi, et al.. (2003). Inflammatory Stimuli Upregulate Rho-Kinase in Human Coronary Vascular Smooth Muscle Cells : Divergent Effects of Estrogen and Nicotine. Japanese Circulation Journal-english Edition. 67. 152. 1 indexed citations
10.
Ichiki, Toshihiro, et al.. (2002). 15-deoxy-Delta^ -prostaglandin J2 And Thiazolidinediones Activate MEK/ERK Pathway through Phosphatidylinositol 3-Kinase In Vascular Smooth Muscle Cells. Japanese Circulation Journal-english Edition. 66. 211. 3 indexed citations
11.
Ni, Weihua, Kensuke Egashira, Shiro Kitamoto, et al.. (2002). Anti-Monocyte Chemoattractant Protein-1 Gene Therapy Limits Progression and Destabilization of Established Atherosclerosis in Apolipoprotein E-Knockout Mice. Japanese Circulation Journal-english Edition. 66. 704. 1 indexed citations
12.
Hiroki, Junko, Hiroaki Shimokawa, Tadashi Kandabashi, et al.. (2002). Inflammatory stimuli upregulate Rho-kinase expression in human coronary vascular smooth muscle cells. Japanese Circulation Journal-english Edition. 66. 282. 1 indexed citations
13.
Takeda, Kotaro, Toshihiro Ichiki, Tomotake Tokunou, et al.. (2001). Critical Role of Rho-Kinase and MEK/ERK Pathways for Angiotensin II–Induced Plasminogen Activator Inhibitor Type-1 Gene Expression. Arteriosclerosis Thrombosis and Vascular Biology. 21(5). 868–873. 124 indexed citations
14.
Setoguchi, Soko, Masahiro Mohri, Yoshitaka Hirooka, et al.. (2000). IS089 Limited Availability of Nitric Oxide Synthase Cofactor, Tetrahydrobiopterin, is Responsible for Coronary Endothelial Dysfunction in Patients with Coronary Risk Factors. Japanese Circulation Journal-english Edition. 64. 147. 2 indexed citations
15.
Takeda, Kotaro, Toshihiro Ichiki, Tomotake Tokunou, et al.. (2000). Critical Roles of Rho Kinase and Mek/Erk Pathways for Angiotensin Ii-Induced Plasminogen Activator Inhibitor-1 Gene Expression. Hypertension. 36. 721–721. 4 indexed citations
16.
Hunley, Tracy E., Masaaki Tamura, Hideki Nishimura, et al.. (2000). The angiotensin type II receptor tonically inhibits angiotensin-converting enzyme in AT2 null mutant mice. Kidney International. 57(2). 570–577. 36 indexed citations
17.
Sakagawa, Takashi, Shigeru Okuyama, Naoya Kawashima, et al.. (2000). Pain threshold, learning and formation of brain edema in mice lacking the angiotensin II type 2 receptor. Life Sciences. 67(21). 2577–2585. 36 indexed citations
18.
Inagami, Tadashi, Satoru Eguchi, Satoshi Tsuzuki, & Toshihiro Ichiki. (1998). Angiotensin II Receptors AT1 and AT2: New Mechanisms of Signaling And Antagonistic Effects of AT1 and AT2. 129–139. 13 indexed citations
19.
Niimura, Fumio, Patricia A. Labosky, Junji Kakuchi, et al.. (1995). Gene targeting in mice reveals a requirement for angiotensin in the development and maintenance of kidney morphology and growth factor regulation.. Journal of Clinical Investigation. 96(6). 2947–2954. 275 indexed citations
20.
Ichiki, Toshihiro, Wataru Takahashi, & Takeshi Watanabe. (1992). The effect of cytokines and mitogens on the induction of Cε germline transcripts in a human Burkitt lymphoma B cell line. International Immunology. 4(7). 747–754. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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