Tomohiro Ohgomori

1.4k citations
28 papers · 1.1k indexed · 1 hit paper · h-index 14

Tomohiro Ohgomori

26 papers receiving 1.1k citations

Hit Papers

Minocycline selectively inhibits M1 polarization of micro...5942013202620172021100200300400500

Peers

Tomohiro Ohgomori
Comparison fields: 5 of 96
  • Neurology 558
  • Biological Psychiatry 115
  • Developmental Neuroscience 128
  • Behavioral Neuroscience 78
  • Cellular and Molecular Neuroscience 302
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Jana Vukovic Australia
Flavia Trettel Italy
María Santos‐Galindo Spain
Katrin Färber Germany
Thomas C. Brionne Switzerland
Blanca Díaz‐Castro United States
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Citations per field
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Citations per year

Countries citing papers authored by Tomohiro Ohgomori

Since Specialization
Citations

This map shows the geographic impact of Tomohiro Ohgomori's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomohiro Ohgomori with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomohiro Ohgomori more than expected).

Fields of papers citing papers by Tomohiro Ohgomori

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomohiro Ohgomori. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomohiro Ohgomori. The network helps show where Tomohiro Ohgomori may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Tomohiro Ohgomori, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tomohiro Ohgomori Line = papers co-authored together Tomohiro Ohgomori links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20250
2 20244
3 20222
4 20214
5 202116
6 20217
7 202013
8 20194
9 201822
10 20173
11 201517
12
Minocycline selectively inhibits M1 polarization of microgliabreakdown →
2013594
13 201321
14 201336
15 201248
16 201220
17 201277
18 201175
19 20113
20 20097

About Tomohiro Ohgomori

Tomohiro Ohgomori is a scholar working on Neurology, Developmental Neuroscience and Physical Therapy, Sports Therapy and Rehabilitation, having authored 28 papers that have together received 1.1k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (8 papers), Amyotrophic Lateral Sclerosis Research (7 papers), Nerve injury and regeneration (6 papers), Proteoglycans and glycosaminoglycans research (6 papers), Neuroscience and Neuropharmacology Research (4 papers), Parkinson's Disease Mechanisms and Treatments (4 papers), Cell Adhesion Molecules Research (4 papers) and Balance, Gait, and Falls Prevention (3 papers). The work is most often cited by research in Neurology (558 citations), Biological Psychiatry (115 citations) and Developmental Neuroscience (128 citations). Tomohiro Ohgomori has collaborated with scholars based in Japan, United States and Russia. Frequent co-authors include Kenji Kadomatsu, Naoki Ishiguro, Shiro Imagama, Shozo Jinno, Hideyuki Takeuchi, Kazuma Sakamoto, Kazuyoshi Kobayashi, Ken‐ichi Hirano, Kenji Uchimura and Akio Suzumura. Their work appears in journals such as Journal of Neuroscience, PLoS ONE and The Journal of Comparative Neurology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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