Tomiichiro Oda

1.8k total citations
31 papers, 1.5k citations indexed

About

Tomiichiro Oda is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Tomiichiro Oda has authored 31 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 15 papers in Physiology and 9 papers in Cellular and Molecular Neuroscience. Recurrent topics in Tomiichiro Oda's work include Alzheimer's disease research and treatments (14 papers), Neuroscience and Neuropharmacology Research (6 papers) and Cholinesterase and Neurodegenerative Diseases (4 papers). Tomiichiro Oda is often cited by papers focused on Alzheimer's disease research and treatments (14 papers), Neuroscience and Neuropharmacology Research (6 papers) and Cholinesterase and Neurodegenerative Diseases (4 papers). Tomiichiro Oda collaborates with scholars based in Japan, Germany and United States. Tomiichiro Oda's co-authors include Giulio Maria Pasinetti, Caleb E. Finch, Mark S. Shearman, Yasutomi Nishizuka, Irina Rozovsky, Steve A. Johnson, Yoshiko Onozawa, Tomoaki Komai, Judes Poirier and Philìppe Bertrand and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Comparative Neurology and Brain Research.

In The Last Decade

Tomiichiro Oda

31 papers receiving 1.5k citations

Peers

Tomiichiro Oda
Tomohiro Chiba Japan
Robert P. Strosznajder Poland
E. Matsubara Japan
Richard C. Dodel Germany
Tinyi Chu United States
Andrew McShea United States
Brandy Wilkinson United States
Shih-Chu Kao United States
Binhui Ni United States
Ken-ichiro Fukuchi United States
Tomohiro Chiba Japan
Tomiichiro Oda
Citations per year, relative to Tomiichiro Oda Tomiichiro Oda (= 1×) peers Tomohiro Chiba

Countries citing papers authored by Tomiichiro Oda

Since Specialization
Citations

This map shows the geographic impact of Tomiichiro Oda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomiichiro Oda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomiichiro Oda more than expected).

Fields of papers citing papers by Tomiichiro Oda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomiichiro Oda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomiichiro Oda. The network helps show where Tomiichiro Oda may publish in the future.

Co-authorship network of co-authors of Tomiichiro Oda

This figure shows the co-authorship network connecting the top 25 collaborators of Tomiichiro Oda. A scholar is included among the top collaborators of Tomiichiro Oda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomiichiro Oda. Tomiichiro Oda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Suzuki, M., Sachiko Takaishi, Miyuki Nagasaki, et al.. (2013). Medium-chain Fatty Acid-sensing Receptor, GPR84, Is a Proinflammatory Receptor. Journal of Biological Chemistry. 288(15). 10684–10691. 165 indexed citations
2.
Onozawa, Yoshiko, Tomoaki Komai, & Tomiichiro Oda. (2011). Activation of T cell death-associated gene 8 attenuates inflammation by negatively regulating the function of inflammatory cells. European Journal of Pharmacology. 654(3). 315–319. 39 indexed citations
3.
Nishizawa, Tomohiro, Ken Kitayama, Kenji Wakabayashi, et al.. (2006). A novel compound, R-138329, increases plasma HDL cholesterol via inhibition of scavenger receptor BI-mediated selective lipid uptake. Atherosclerosis. 194(2). 300–308. 10 indexed citations
4.
Morimoto, Kiyoshi & Tomiichiro Oda. (2003). Kainate exacerbates β-amyloid toxicity in rat hippocampus. Neuroscience Letters. 340(3). 242–244. 7 indexed citations
5.
6.
Nishimura, Satoko, Takekazu Kubo, Isao Kaneko, et al.. (2003). RS‐4252 Inhibits Amyloid β‐Induced Cytotoxicity in HeLa Cells. Pharmacology & Toxicology. 93(1). 29–32. 19 indexed citations
7.
Tokuhiro, Shinya, et al.. (2003). β-Amyloid-specific upregulation of stearoyl coenzyme A desaturase-1 in macrophages. Biochemical and Biophysical Research Communications. 303(1). 302–305. 19 indexed citations
8.
Nakagami, Yasuhiro & Tomiichiro Oda. (2002). Glutamate Exacerbates Amyloid β1 − 42-Induced Impairment of Long-Term Potentiation in Rat Hippocampal Slices. The Japanese Journal of Pharmacology. 88(2). 223–226. 19 indexed citations
9.
Harada, Jun, et al.. (2002). Troglitazone inhibits both post-glutamate neurotoxicity and low-potassium-induced apoptosis in cerebellar granule neurons. Brain Research. 924(2). 229–236. 63 indexed citations
10.
Tokuhiro, Shinya, et al.. (2002). A novel compound, RS-1178, specifically inhibits neuronal cell death mediated by β-amyloid-induced macrophage activation in vitro. Brain Research. 946(2). 298–306. 21 indexed citations
11.
Nakagami, Yasuhiro, Satoko Nishimura, Isao Kaneko, et al.. (2002). A novelβ‐sheet breaker, RS‐0406, reverses amyloidβ‐induced cytotoxicity and impairment of long‐term potentiationin vitro. British Journal of Pharmacology. 137(5). 676–682. 85 indexed citations
12.
Nakagami, Yasuhiro, Satoko Nishimura, Takekazu Kubo, et al.. (2002). A novel compound RS-0466 reverses β-amyloid-induced cytotoxicity through the Akt signaling pathway in vitro. European Journal of Pharmacology. 457(1). 11–17. 24 indexed citations
13.
Morimoto, Kiyoshi, et al.. (1998). Amyloidogenic peptides such as β-amyloid, amylin and calcitonin strongly enhance the susceptibility of rat hippocampal neurons to excitatory amino acids in vivo. Folia Pharmacologica Japonica. 112(supplement). 83–87. 5 indexed citations
14.
Sato, Aiya, et al.. (1997). N-Type Calcium Channel Blockers from a Marine Bacterium, Cytophaga sp. SANK 71996.. The Journal of Antibiotics. 50(6). 457–468. 17 indexed citations
15.
Oda, Tomiichiro, P.A. Wals, Heinz H. Osterburg, et al.. (1995). Clusterin (apoJ) Alters the Aggregation of Amyloid β-Peptide (Aβ1-42) and Forms Slowly Sedimenting Aβ Complexes That Cause Oxidative Stress. Experimental Neurology. 136(1). 22–31. 282 indexed citations
16.
Bertrand, Philìppe, Judes Poirier, Tomiichiro Oda, Caleb E. Finch, & Giulio Maria Pasinetti. (1995). Association of apolipoprotein E genotype with brain levels of apolipoprotein E and apolipoprotein J (clusterin) in Alzheimer disease. Molecular Brain Research. 33(1). 174–178. 177 indexed citations
17.
Shearman, Mark S., Tetsutaro Shinomura, Tomiichiro Oda, & Yasutomi Nishizuka. (1991). Protein kinase C subspecies in adult rat hippocampal synaptosomes Activation by diacylglycerol and arachidonic acid. FEBS Letters. 279(2). 261–264. 47 indexed citations
18.
Shearman, Mark S., Tetsutaro Shinomura, Tomiichiro Oda, & Yasutomi Nishizuka. (1991). Synaptosomal Protein Kinase C Subspecies: A. Dynamic Changes in the Hippocampus and Cerebellar Cortex Concomitant with Synaptogenesis. Journal of Neurochemistry. 56(4). 1255–1262. 63 indexed citations
19.
Oda, Tomiichiro, Mark S. Shearman, & Yasutomi Nishizuka. (1991). Synaptosomal Protein Kinase C Subspecies: B. Down‐Regulation Promoted by Phorbol Ester and Its Effect on Evoked Norepinephrine Release. Journal of Neurochemistry. 56(4). 1263–1269. 65 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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