Satoko Nishimura

754 total citations
17 papers, 514 citations indexed

About

Satoko Nishimura is a scholar working on Physiology, Molecular Biology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Satoko Nishimura has authored 17 papers receiving a total of 514 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Physiology, 5 papers in Molecular Biology and 4 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Satoko Nishimura's work include Alzheimer's disease research and treatments (6 papers), Computational Drug Discovery Methods (3 papers) and Cholinesterase and Neurodegenerative Diseases (3 papers). Satoko Nishimura is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Computational Drug Discovery Methods (3 papers) and Cholinesterase and Neurodegenerative Diseases (3 papers). Satoko Nishimura collaborates with scholars based in Japan, United States and Germany. Satoko Nishimura's co-authors include Isao Kaneko, Takekazu Kubo, Yoshihiro Kumagae, Tomiichiro Oda, Yutaka Tomita, Tomoko Tanaka, Hiroshi Kogen, Yasuhiro Nakagami, Shinji Marumoto and Masaki Meguro and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and British Journal of Pharmacology.

In The Last Decade

Satoko Nishimura

16 papers receiving 511 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Satoko Nishimura Japan 11 286 188 107 105 64 17 514
Carlos Morgan Chile 13 351 1.2× 235 1.3× 94 0.9× 166 1.6× 110 1.7× 26 716
Paul S. Marinec United States 9 270 0.9× 363 1.9× 204 1.9× 128 1.2× 74 1.2× 10 657
James E. Finley United States 7 277 1.0× 150 0.8× 129 1.2× 115 1.1× 74 1.2× 11 535
Hirra A. Arain United States 5 319 1.1× 175 0.9× 57 0.5× 97 0.9× 32 0.5× 5 518
Nicolle Siegart United States 6 227 0.8× 188 1.0× 41 0.4× 86 0.8× 27 0.4× 7 502
Biuse Guivernau Spain 13 265 0.9× 243 1.3× 122 1.1× 52 0.5× 20 0.3× 16 669
William Z. Suo United States 13 155 0.5× 321 1.7× 164 1.5× 92 0.9× 47 0.7× 25 689
Johanna Fälting Sweden 14 427 1.5× 306 1.6× 121 1.1× 239 2.3× 237 3.7× 25 838
Gregory D. Van Vickle United States 6 501 1.8× 161 0.9× 71 0.7× 94 0.9× 66 1.0× 6 608
Giulia Mazzoleni Italy 13 385 1.3× 275 1.5× 145 1.4× 44 0.4× 30 0.5× 15 719

Countries citing papers authored by Satoko Nishimura

Since Specialization
Citations

This map shows the geographic impact of Satoko Nishimura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satoko Nishimura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satoko Nishimura more than expected).

Fields of papers citing papers by Satoko Nishimura

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Satoko Nishimura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satoko Nishimura. The network helps show where Satoko Nishimura may publish in the future.

Co-authorship network of co-authors of Satoko Nishimura

This figure shows the co-authorship network connecting the top 25 collaborators of Satoko Nishimura. A scholar is included among the top collaborators of Satoko Nishimura based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Satoko Nishimura. Satoko Nishimura is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Visser, Lance C., et al.. (2018). Acute echocardiographic effects of sotalol on ventricular systolic function in dogs with ventricular arrhythmias. Journal of Veterinary Internal Medicine. 32(4). 1299–1307. 9 indexed citations
2.
Nishimura, Satoko, et al.. (2018). Echocardiographic evaluation of velocity ratio, velocity time integral ratio, and pulmonary valve area in dogs with pulmonary valve stenosis. Journal of Veterinary Internal Medicine. 32(5). 1570–1578. 16 indexed citations
3.
Gunther‐Harrington, Catherine T., et al.. (2018). High-pressure balloon valvuloplasty for severe pulmonary valve stenosis: a prospective observational pilot study in 25 dogs. Journal of Veterinary Cardiology. 20(2). 115–122. 14 indexed citations
4.
Nishimura, Satoko, et al.. (2013). Contralateral Axillary Node Metastasis From Recurrence After Conservative Breast Cancer Surgery. Clinical Nuclear Medicine. 39(2). 181–183. 8 indexed citations
5.
Uno, Makiko, Satoko Nishimura, Yasuyuki Kaneta, et al.. (2012). Identification of Physiologically Active Substances as Novel Ligands for MRGPRD. SHILAP Revista de lepidopterología. 2012. 1–9. 21 indexed citations
6.
Nishimura, Satoko, Makiko Uno, Yasuyuki Kaneta, et al.. (2012). MRGD, a MAS-related G-protein Coupled Receptor, Promotes Tumorigenisis and Is Highly Expressed in Lung Cancer. PLoS ONE. 7(6). e38618–e38618. 19 indexed citations
7.
Toda, Akihisa, Satoko Nishimura, Hiroaki Kuroki, et al.. (2009). The stimulatory effects of caffeine with oseltamivir (Tamiflu®) on light–dark behavior and open-field behavior in mice. Neuroscience Letters. 469(2). 184–188. 10 indexed citations
9.
Nishimura, Satoko, Takekazu Kubo, Isao Kaneko, et al.. (2003). RS‐4252 Inhibits Amyloid β‐Induced Cytotoxicity in HeLa Cells. Pharmacology & Toxicology. 93(1). 29–32. 19 indexed citations
10.
Kubo, Takekazu, Satoko Nishimura, Yoshihiro Kumagae, & Isao Kaneko. (2002). In vivo conversion of racemized β‐amyloid ([D‐Ser26]Aβ1–40) to truncated and toxic fragments ([D‐Ser26]Aβ25–35/40) and fragment presence in the brains of Alzheimer's patients. Journal of Neuroscience Research. 70(3). 474–483. 224 indexed citations
11.
Nakagami, Yasuhiro, Satoko Nishimura, Isao Kaneko, et al.. (2002). A novelβ‐sheet breaker, RS‐0406, reverses amyloidβ‐induced cytotoxicity and impairment of long‐term potentiationin vitro. British Journal of Pharmacology. 137(5). 676–682. 85 indexed citations
12.
Nakagami, Yasuhiro, Satoko Nishimura, Takekazu Kubo, et al.. (2002). A novel compound RS-0466 reverses β-amyloid-induced cytotoxicity through the Akt signaling pathway in vitro. European Journal of Pharmacology. 457(1). 11–17. 24 indexed citations
13.
Kubo, Takekazu, Satoko Nishimura, & Tomiichiro Oda. (2002). Amyloid β-peptide alters the distribution of early endosomes and inhibits phosphorylation of Akt in the presence of 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT). Molecular Brain Research. 106(1-2). 94–100. 13 indexed citations
15.
Tanaka, Tomoko, Yutaka Tomita, & Satoko Nishimura. (1987). Prognostic Significance of the Electrically Elicited Blink Reflex in Neonates. Journal of Child Neurology. 2(4). 287–292. 8 indexed citations
16.
Tomita, Yutaka, Satoko Nishimura, & Tomoko Tanaka. (1986). Short latency SEPs in infants and children: Developmental changes and maturational index of SEPs. Electroencephalography and Clinical Neurophysiology/Evoked Potentials Section. 65(5). 335–343. 34 indexed citations
17.
Nishimura, Satoko, et al.. (1986). Developmental change of short latency somatosensory evoked potential waves between P3 and N1 components in children. Brain and Development. 8(1). 6–9. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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