Tina Notter

1.9k total citations
23 papers, 1.4k citations indexed

About

Tina Notter is a scholar working on Biological Psychiatry, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Tina Notter has authored 23 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Biological Psychiatry, 12 papers in Neurology and 7 papers in Cellular and Molecular Neuroscience. Recurrent topics in Tina Notter's work include Tryptophan and brain disorders (12 papers), Neuroinflammation and Neurodegeneration Mechanisms (12 papers) and Neuroscience and Neuropharmacology Research (6 papers). Tina Notter is often cited by papers focused on Tryptophan and brain disorders (12 papers), Neuroinflammation and Neurodegeneration Mechanisms (12 papers) and Neuroscience and Neuropharmacology Research (6 papers). Tina Notter collaborates with scholars based in Switzerland, United Kingdom and United States. Tina Notter's co-authors include Urs Meyer, Hanspeter Naegeli, Hans Christian Winkler, Jennifer M. Coughlin, Akira Sawa, Irène Knuesel, Sandra L. Pfister, Dimitrije Krstic, Daniele Mattei and Juliet Richetto and has published in prestigious journals such as Neuroscience, Science Advances and Molecular Psychiatry.

In The Last Decade

Tina Notter

23 papers receiving 1.3k citations

Peers

Tina Notter
Bingjin Li China
Stephanie Matt United States
Xingming Wang China
Gabriela K. Ferreira Brazil
Shanshan Zhang China
Wen‐Ning Wu China
Julian Hellmann‐Regen Germany
William A. Banks United States
Arya Haj‐Mirzaian Iran
Jelena Djordjevic Serbia
Bingjin Li China
Tina Notter
Citations per year, relative to Tina Notter Tina Notter (= 1×) peers Bingjin Li

Countries citing papers authored by Tina Notter

Since Specialization
Citations

This map shows the geographic impact of Tina Notter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tina Notter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tina Notter more than expected).

Fields of papers citing papers by Tina Notter

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tina Notter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tina Notter. The network helps show where Tina Notter may publish in the future.

Co-authorship network of co-authors of Tina Notter

This figure shows the co-authorship network connecting the top 25 collaborators of Tina Notter. A scholar is included among the top collaborators of Tina Notter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tina Notter. Tina Notter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Notter, Tina, et al.. (2025). Ontogeny and plasticity of resilience and susceptibility in a mouse model of maternal immune activation. Brain Behavior and Immunity. 129. 666–680. 1 indexed citations
2.
Mueller, Flavia S., Sina M. Schalbetter, Joel Gruchot, et al.. (2025). Recapitulation and reversal of neuropsychiatric phenotypes in a mouse model of human endogenous retrovirus type W expression. Molecular Psychiatry. 30(7). 3325–3337. 1 indexed citations
3.
Mueller, Flavia S., et al.. (2024). Intrauterine position effects in a mouse model of maternal immune activation. Brain Behavior and Immunity. 120. 391–402. 6 indexed citations
4.
Mattei, Daniele, et al.. (2023). Prefrontal microglia deficiency during adolescence disrupts adult cognitive functions and synaptic structures: A follow-up study in female mice. Brain Behavior and Immunity. 111. 230–246. 7 indexed citations
5.
Mueller, Flavia S., Tina Notter, Sina M. Schalbetter, et al.. (2022). Deficient DNA base-excision repair in the forebrain leads to a sex-specific anxiety-like phenotype in mice. BMC Biology. 20(1). 170–170. 9 indexed citations
6.
Schalbetter, Sina M., Flavia S. Mueller, Juliet Richetto, et al.. (2021). Oral application of clozapine-N-oxide using the micropipette-guided drug administration (MDA) method in mouse DREADD systems. Lab Animal. 50(3). 69–75. 16 indexed citations
7.
Notter, Tina. (2021). Astrocytes in schizophrenia. PubMed. 5. 1492754044–1492754044. 48 indexed citations
8.
Notter, Tina, Sina M. Schalbetter, Nicholas E. Clifton, et al.. (2020). Neuronal activity increases translocator protein (TSPO) levels. Molecular Psychiatry. 26(6). 2025–2037. 90 indexed citations
9.
Mattei, Daniele & Tina Notter. (2019). Basic Concept of Microglia Biology and Neuroinflammation in Relation to Psychiatry. Current topics in behavioral neurosciences. 44. 9–34. 29 indexed citations
10.
Winkler, Hans Christian, Tina Notter, Urs Meyer, & Hanspeter Naegeli. (2018). Critical review of the safety assessment of titanium dioxide additives in food. Journal of Nanobiotechnology. 16(1). 51–51. 170 indexed citations
11.
Notter, Tina, et al.. (2018). Controversies and prospects about microglia in maternal immune activation models for neurodevelopmental disorders. Brain Behavior and Immunity. 73. 51–65. 75 indexed citations
12.
Notter, Tina, Leonie Aengenheister, Ulrike Weber‐Stadlbauer, et al.. (2018). Prenatal exposure to TiO2 nanoparticles in mice causes behavioral deficits with relevance to autism spectrum disorder and beyond. Translational Psychiatry. 8(1). 193–193. 37 indexed citations
13.
Notter, Tina. (2018). Immunological Processes in Schizophrenia Pathology: Potential Biomarkers?. Current topics in behavioral neurosciences. 40. 389–410. 6 indexed citations
14.
Notter, Tina, Jennifer M. Coughlin, Tilo Gschwind, et al.. (2017). Translational evaluation of translocator protein as a marker of neuroinflammation in schizophrenia. Molecular Psychiatry. 23(2). 323–334. 152 indexed citations
15.
Notter, Tina, Jennifer M. Coughlin, Akira Sawa, & Urs Meyer. (2017). Reconceptualization of translocator protein as a biomarker of neuroinflammation in psychiatry. Molecular Psychiatry. 23(1). 36–47. 110 indexed citations
16.
Labouesse, Marie A., Olivier Lassalle, Juliet Richetto, et al.. (2016). Hypervulnerability of the adolescent prefrontal cortex to nutritional stress via reelin deficiency. Molecular Psychiatry. 22(7). 961–971. 50 indexed citations
17.
Giovanoli, Sandra, Tina Notter, Juliet Richetto, et al.. (2015). Late prenatal immune activation causes hippocampal deficits in the absence of persistent inflammation across aging. Journal of Neuroinflammation. 12(1). 221–221. 87 indexed citations
18.
Notter, Tina & Irène Knuesel. (2013). Reelin immunoreactivity in neuritic varicosities in the human hippocampal formation of non-demented subjects and Alzheimer’s disease patients. Acta Neuropathologica Communications. 1(1). 27–27. 17 indexed citations
19.
Krstic, Dimitrije, Sandra L. Pfister, Tina Notter, & Irène Knuesel. (2013). Decisive role of Reelin signaling during early stages of Alzheimer’s disease. Neuroscience. 246. 108–116. 27 indexed citations
20.
Krstic, Dimitrije, Jana Doehner, Tina Notter, et al.. (2012). Systemic immune challenges trigger and drive Alzheimer-like neuropathology in mice. Journal of Neuroinflammation. 9(1). 151–151. 310 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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