Tilo Moede

31 papers receiving 2.0k citations

Peers

Tilo Moede
Comparison fields: 5 of 84
  • Endocrinology, Diabetes and Metabolism 585
  • Surgery 1.2k
  • Genetics 610
  • Molecular Biology 990
  • Cell Biology 235
Replace Kathy D. Shelton with:
Kathy D. Shelton United States
Greg Poffenberger United States
Cristina Alarcón United States
Eric L. Ford United States
Mathieu Armanet France
Amelia K. Linnemann United States
Xiao-Qing Dai Canada
Dana Avrahami Israel
Hiroko Kadowaki Japan
Matteo Levisetti United States
Tilo Moede relative to Kathy D. Shelton United States Kathy D. Shelton's profile →
Citations per field
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Kathy D. Shelton · 1×
Citations per year

Countries citing papers authored by Tilo Moede

Since Specialization
Citations

This map shows the geographic impact of Tilo Moede's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tilo Moede with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tilo Moede more than expected).

Fields of papers citing papers by Tilo Moede

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tilo Moede. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tilo Moede. The network helps show where Tilo Moede may publish in the future.

Co-authors

The 25 scholars most cited alongside Tilo Moede, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Tilo Moede Line = papers co-authored together Tilo Moede links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 33 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2014299
2 1998231
3 2008208
4 2001202
5 1999118
6 201495
7 201094
8 199887
9 199984
10 202079
11 200373
12 201563
13 200242
14 201238
15 200735
16 201529
17 201827
18 201226
19 202022
20 200221

About Tilo Moede

Tilo Moede is a scholar working on Genetics, Surgery, Endocrinology, Diabetes and Metabolism, Toxicology and Molecular Biology, having authored 33 papers that have together received 2.0k indexed citations. Recurring topics across this work include Pancreatic function and diabetes (30 papers), Diabetes and associated disorders (18 papers), Metabolism, Diabetes, and Cancer (14 papers), Diabetes Management and Research (7 papers), Adipose Tissue and Metabolism (4 papers), Angiogenesis and VEGF in Cancer (2 papers), Diabetes Treatment and Management (2 papers) and Cellular transport and secretion (2 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (585 citations), Surgery (1.2k citations), Genetics (610 citations), Molecular Biology (990 citations) and Cell Biology (235 citations). Tilo Moede has collaborated with scholars based in Sweden, United States and Singapore. Frequent co-authors include Per‐Olof Berggren, Ingo B. Leibiger, Barbara Leibiger, Martin Köhler, Sabine Uhles, Rohit Kulkarni, C. Ronald Kahn, Lina Moitoso de Vargas, Melin J. Khandekar and Incoronata Murano. Their work appears in journals such as Proceedings of the National Academy of Sciences, The FASEB Journal, Scientific Reports, Molecular Cell and Diabetes.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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