Thomas R. Hundley
- Immunology
- Molecular Biology
- Pharmacology top 10%
- Physiology
- Biochemistry top 5%
- Co-authors
- Michael A. BeavenBasil RigasDean D. MetcalfeAlasdair M. GilfillanMarcus Vinícius Melo de AndradeChristine TkaczykKhosrow KashfiJennie L. Williams
- Topics
- Inflammatory mediators and NSAID effects (6 papers)Estrogen and related hormone effects (4 papers)Eicosanoids and Hypertension Pharmacology (4 papers)
- Cited by
- BiochemistryImmunologyPharmacology
- Partner nations
- United StatesNetherlands
In The Last Decade
Thomas R. Hundley
14 papers receiving 463 citations
Peers
Comparison fields: 5 of 69
- Immunology 214
- Molecular Biology 142
- Pharmacology 136
- Physiology 111
- Biochemistry 91
Countries citing papers authored by Thomas R. Hundley
This map shows the geographic impact of Thomas R. Hundley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas R. Hundley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas R. Hundley more than expected).
Fields of papers citing papers by Thomas R. Hundley
This network shows the impact of papers produced by Thomas R. Hundley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas R. Hundley. The network helps show where Thomas R. Hundley may publish in the future.
Co-authorship network of co-authors of Thomas R. Hundley
This figure shows the co-authorship network connecting the top 25 collaborators of Thomas R. Hundley. A scholar is included among the top collaborators of Thomas R. Hundley based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas R. Hundley. Thomas R. Hundley is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 19 | |
| 2 | 38 | |
| 3 | 54 | |
| 4 | 131 | |
| 5 | 1 | |
| 6 | Growth inhibition of human colon cancer cells by nitric oxide (NO)-donating aspirin is associated with cyclooxygenase-2 induction and beta-catenin/T-cell factor signaling, nuclear factor-kappaB, and NO synthase 2 inhibition: implications for chemoprevention. | 92 |
| 7 | 41 | |
| 8 | 24 | |
| 9 | 20 | |
| 10 | 3 | |
| 11 | 14 | |
| 12 | 11 | |
| 13 | 12 | |
| 14 | 17 |
About Thomas R. Hundley
Thomas R. Hundley is a scholar working on Biochemistry, Pharmacology and Immunology, having authored 14 papers that have together received 477 indexed citations. Recurring topics across this work include Inflammatory mediators and NSAID effects (6 papers), Estrogen and related hormone effects (4 papers) and Eicosanoids and Hypertension Pharmacology (4 papers). The work is most often cited by research in Biochemistry (91 citations), Immunology (214 citations) and Pharmacology (136 citations). Thomas R. Hundley has collaborated with scholars based in United States and Netherlands. Frequent co-authors include Michael A. Beaven, Basil Rigas, Dean D. Metcalfe, Alasdair M. Gilfillan, Marcus Vinícius Melo de Andrade, Christine Tkaczyk, Khosrow Kashfi, Jennie L. Williams, Jie Chen and Jianjun Gao. Their work appears in journals such as Blood, The Journal of Immunology and Gastroenterology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.