Thomas Eichholtz

2.9k total citations · 2 hit papers
14 papers, 2.6k citations indexed

About

Thomas Eichholtz is a scholar working on Molecular Biology, Physiology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Thomas Eichholtz has authored 14 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Physiology and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Thomas Eichholtz's work include Protein Kinase Regulation and GTPase Signaling (5 papers), Neonatal Respiratory Health Research (3 papers) and Sphingolipid Metabolism and Signaling (3 papers). Thomas Eichholtz is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (5 papers), Neonatal Respiratory Health Research (3 papers) and Sphingolipid Metabolism and Signaling (3 papers). Thomas Eichholtz collaborates with scholars based in Netherlands, United Kingdom and Canada. Thomas Eichholtz's co-authors include Wouter H. Moolenaar, Kees Jalink, E.J. van Corven, Iris Fahrenfort, Anne J. Ridley, Beata Wójciak‐Stothard, Hidde L. Ploegh, John de Widt, Rob M. J. Liskamp and Friso R. Postma and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Biochemical Journal.

In The Last Decade

Thomas Eichholtz

14 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Lysophosphatidate-induced cell proliferation: Identification and dissection of signaling pathways mediated by G proteins

1989 760 citations E.J. van Corven, Kees Jalink et al. Cell profile →
The bioactive phospholipid lysophosphatidic acid is released from activated platelets

1993 576 citations Thomas Eichholtz, Kees Jalink et al. Biochemical Journal profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas Eichholtz Netherlands	14	1.9k	544	524	265	197	14	2.6k
Ingrid Verlaan Netherlands	19	2.2k 1.1×	616 1.1×	296 0.6×	262 1.0×	73 0.4×	25	2.8k
Shun‐ichi Nakamura Japan	26	2.3k 1.2×	825 1.5×	377 0.7×	515 1.9×	110 0.6×	74	3.0k
Stefano Fumagalli France	19	3.3k 1.7×	519 1.0×	556 1.1×	331 1.2×	141 0.7×	27	4.1k
Günter Daum United States	27	1.7k 0.9×	341 0.6×	269 0.5×	357 1.3×	136 0.7×	55	2.5k
Muriel I. Kaiser‐Kupfer United States	41	2.0k 1.0×	620 1.1×	291 0.6×	224 0.8×	404 2.1×	103	4.9k
Nadir M. Maraldi Italy	34	3.1k 1.6×	611 1.1×	344 0.7×	169 0.6×	62 0.3×	98	3.7k
Sathivel Ponniah Singapore	16	1.7k 0.9×	369 0.7×	385 0.7×	361 1.4×	41 0.2×	18	2.6k
Laurens A. van Meeteren Netherlands	27	3.0k 1.6×	1.0k 1.9×	491 0.9×	312 1.2×	166 0.8×	33	3.8k
Lesley K. Sinclair United States	12	1.6k 0.8×	313 0.6×	233 0.4×	331 1.2×	328 1.7×	13	2.3k
Yaowu Zheng China	18	1.6k 0.8×	297 0.5×	274 0.5×	432 1.6×	197 1.0×	47	3.4k

Countries citing papers authored by Thomas Eichholtz

Since Specialization

Citations

This map shows the geographic impact of Thomas Eichholtz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Eichholtz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Eichholtz more than expected).

Fields of papers citing papers by Thomas Eichholtz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Eichholtz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Eichholtz. The network helps show where Thomas Eichholtz may publish in the future.

Co-authorship network of co-authors of Thomas Eichholtz

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Eichholtz. A scholar is included among the top collaborators of Thomas Eichholtz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Eichholtz. Thomas Eichholtz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Vasavda, Nisha, Thomas Eichholtz, Akihiko Takahashi, et al.. (2006). Expression of nonmuscle cofilin-1 and steroid responsiveness in severe asthma. Journal of Allergy and Clinical Immunology. 118(5). 1090–1096. 16 indexed citations

Lagente, Vincent, et al.. (2006). Involvement of MMP-12 and phosphodiesterase type 4 in cigarette smoke-induced inflammation in mice. European Respiratory Journal. 27(6). 1102–1109. 65 indexed citations

Ogawa, Emiko, et al.. (2004). Latent adenoviral infection induces production of growth factors relevant to airway remodeling in COPD. American Journal of Physiology-Lung Cellular and Molecular Physiology. 286(1). L189–L197. 30 indexed citations

Chen, Gang, Gary R. Grotendorst, Thomas Eichholtz, & Nasreen Khalil. (2003). GM-CSF increases airway smooth muscle cell connective tissue expression by inducing TGF-β receptors. American Journal of Physiology-Lung Cellular and Molecular Physiology. 284(3). L548–L556. 38 indexed citations

Coutts, Amanda S., Gang Chen, N. L. Stephens, et al.. (2001). Release of biologically active TGF-β from airway smooth muscle cells induces autocrine synthesis of collagen. American Journal of Physiology-Lung Cellular and Molecular Physiology. 280(5). L999–L1008. 97 indexed citations

Wójciak‐Stothard, Beata, et al.. (2001). Rho and Rac but not Cdc42 regulate endothelial cell permeability. Journal of Cell Science. 114(7). 1343–1355. 406 indexed citations

Harris, Robert A., Thomas Eichholtz, Ian D. Hiles, Martin Pagé, & Michael J. O’Hare. (1999). New model of ErbB-2 over-expression in human mammary luminal epithelial cells. International Journal of Cancer. 80(3). 477–484. 53 indexed citations

Jalink, Kees, et al.. (1993). Lysophosphatidic acid induces neuronal shape changes via a novel, receptor-mediated signaling pathway: similarity to thrombin action.. PubMed. 4(4). 247–55. 182 indexed citations

Eichholtz, Thomas, Kees Jalink, Iris Fahrenfort, & Wouter H. Moolenaar. (1993). The bioactive phospholipid lysophosphatidic acid is released from activated platelets. Biochemical Journal. 291(3). 677–680. 576 indexed citations breakdown →

10.

Eichholtz, Thomas, et al.. (1993). A myristoylated pseudosubstrate peptide, a novel protein kinase C inhibitor.. Journal of Biological Chemistry. 268(3). 1982–1986. 255 indexed citations

11.

Moolenaar, Wouter H., R.L. van der Bend, E.J. van Corven, et al.. (1992). Lysophosphatidic Acid: A Novel Phospholipid with Hormone- and Growth Factor-like Activities. Cold Spring Harbor Symposia on Quantitative Biology. 57(0). 163–167. 24 indexed citations

12.

Eichholtz, Thomas, et al.. (1992). Activation of protein kinase C accelerates internalization of transferrin receptor but not of major histocompatibility complex class I, independent of their phosphorylation status.. Journal of Biological Chemistry. 267(31). 22490–22495. 30 indexed citations

13.

Eichholtz, Thomas, et al.. (1990). A pseudosubstrate peptide inhibits protein kinase C‐mediated phosphorylation in permeabilized Rat‐1 cells. FEBS Letters. 261(1). 147–150. 23 indexed citations

14.

Corven, E.J. van, et al.. (1989). Lysophosphatidate-induced cell proliferation: Identification and dissection of signaling pathways mediated by G proteins. Cell. 59(1). 45–54. 760 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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